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Effect of long-term synaptic plasticity on spatial cognition of Wistar rats

Effect of long-term synaptic plasticity on spatial cognition...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Zhuo YANG Da-Dong HAN Medical School of Nankai University Tianjin 300071 China

Objective Long-term synaptic plasticity, including long term potentiation (LTP) and long term depression (LTD), was an eventful pattern to modulate efficiency of synaptic transmission in nervous system, and LTP was regarded as the underlying mechanism in learning and *** and LTD switched enhancement or attenuation of neural signal via adjustment the ratio of NR2A/*** study focused on the relationship between the alteration of long-term synaptic plasticity and the performance of Morris water maze (MWM), attempted to interpret the mechanism, which electrophysiological effects on the *** Rats were randomly divided into 3 groups, MK-801 (n=16), AP-V (n=16) and control group (n=16).MK-801 (10 μg, 1 μg/μL), AP-V (10 μg, 1 μg/μL) and artificial cerebrospinal fluid (ACSF, 10 μL) were administered in dentate gyrus (DG) of *** was carried, and LTP and LTD were recorded *** LTP was significantly attenuated (P<0.01), while LTD was barely influenced (P>0.05) in DG region of MK-801 group compared with *** the contrary, LTP was barely influenced (P>0.05), while LTD was significantly attenuated (P<0.01) in DG region of AP-V group compared with *** acquisition and retention tests, the escape latency and path length were prolonged (P<0.05; P<0.01) in MK-801 group, and were cut down (P<0.05; P<0.01) in AP-V group compared with control *** the contrary, in reacquisition phase, the escape latency (P<0.05) was cut down in MK-801 group, and was prolonged (P<0.05) in AP-Ⅴ compared with control *** The unbalance of long-term synaptic plasticity results in the spatial cognitive impairment in rats.

关键词： long-term synaptic plasticity LTP LTD spatial learning and memory

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The effects of embedding catgut in acupoints on behaviors of WKY rat,a putative animal model of depression

The effects of embedding catgut in acupoints on behaviors of...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Ting WANG Chun-Lei DING Bu-Yi WANG Yang GUO Shou-Shan MA Sheng-Tian LI School of Life Sciences & Biotechnology Shanghai Jiao Tong University Shanghai 200240 China School of Life Sciences & Biotechnology Shanghai Jiao Tong University Shanghai 200240 China Kao (China) Research & Development Center No.623 Ziri Rd. Minhang District Shanghai 200241 China

Objective In China, acupuncture has been used for treatment of depressive disorders, while the evaluations of therapeutic effects have been lack of experimental *** catgut in acupoints (ECP) is an alternative method of acupuncture that continuously stimulates the acupoints via producing physiological and biochemical *** to hand-acupuncture or electro-acupuncture, ECP is more easily to perform in animals because which can be conducted under *** the current study we investigated the effect of ECP on Wistar Kyoto (WKY) rats, a genetic animal model for depressive *** WKY rats were treated with ECP once per week at GV20 (Bai Hui), bilateral BL15 (Xin Shu), BL23 (Shen Shu), PC6 (Nei Guan), ST36 (Zu San Li) and SP6 (San Yin Jiao) under *** depressive, anxiety-like and spatial learning behaviors were examined by behavior test battery including open field test, forced swim test, Morris water maze test, elevated plus maze test and light/dark transition *** Compared with sham ECP treatment, ECP treatment significantly increased total distance traveled and time stayed in the central area in open field test and the time spent in the target quadrant in Morris water maze *** These findings indicate that ECP treatment can improve depressive, anxiety-like and spatial learning behaviors of WKY rats.

关键词： acupuncture depression anxiety spacial learning and memory nomifensine

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Targeting A-type K+ channels in primary sensory neurons for bone cancer pain in a rat model

Targeting A-type K+ channels in primary sensory neurons for ...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Kai-Zheng DUAN Qian XU Xiao-Meng ZHANG Zhi-Qi ZHAO Yan-Ai MEI Yu-Qiu ZHANG Institute of Neurobiology Institutes of Brain Science and State Key Laboratory of Medical Neurobiology Fudan University Shanghai 200032 China School of Life Sciences Institutes of Brain Science and State Key Laboratory of Medical Neurobiology Fudan University Shanghai 200032 China

The treatment of bone cancer pain remains a clinically challenging ***, we report that A-type K+ channels in primary sensory neurons are involved in the neuropathy of rat bone cancer pain and are a new target for diclofenac, a non-steroidal anti-inflammatory drug that can be used for therapy for this distinct *** injection of diclofenac dose-dependently attenuated bone cancer-induced spontaneous pain, mechanical allodynia and thermal hyperalgesia, all of which were reversed or prevented by administration of the A-type K+ channel blockers 4-AP or pandinotoxin-Kα or with siRNA targeted against Kv1.4 or ***, diclofenac significantly inhibited action potential firing and rapidly increased A-type K+ currents in IB4-positive small dorsal root ganglion (DRG) neurons in both normal and bone cancer ***, in bone cancer rats the A-type K+ currents were significantly increased on post-tumor day 14 (PTD 14); they then faded, but they still remained at a higher level on PTD ***, the expression levels of the A-type K+ channels Kv1.4, Kv3.4, and Kv4.3 showed time-dependent changes during bone cancer *** diclofenac administration not only decreased soft tissue swelling adjacent to the tumor but also attenuated bone *** results indicate that A-type K+ channel modulators may provide a novel mechanism-based therapeutic strategy for bone cancer pain.

关键词： A-type K+currents bone cancer pain diclofenac dorsal root ganglion rat

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APPL1 mediates synaptic NMDA receptor-dependent neuroprotection

APPL1 mediates synaptic NMDA receptor-dependent neuroprotect...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Yu-Bin WANG Shuang QIU Jie-Jie WANG Shao-Hua WANG Jian-Hong LUO Department of Neurobiology Zhejiang University School of Medicine Hangzhou 310058 China

N-methyl-D-aspartate (NMDA) receptors are glutamate gated ion channels and play important roles in both physiology and pathophysiology of central *** activation of NMDA receptor can cause neuronal loss in acute trauma such as ischemia, as well as certain neurodegenerative diseases such as Huntington's ***, physiological levels of synaptic NMDA receptor activity can promote neuronal survival through different signaling pathways under different *** them, PI3K (phosphoinositide-3-kinase)/Akt kinase cascade is a key signaling pathway responsible for neuroprotective effects of NMDA receptor *** is still unknown how NMDA receptors are coupled with PI3K-Akt ***1 (adaptor protein containing pH domain, PTB domain, and Leucine zipper motif), is an endosomal adaptor protein which are associated with various transmembrane ***, APPL1 has been reported to interact with, and regulate the activity of, the kinase *** the present study, using biochemical analysis and immunostaining, we found that APPL1 associates with NMDA receptor complex through its C-terminal PDZ binding *** importantly, APPL1 is invoIved in synaptic NMDA receptor mediated neuroprotective *** of APPL1-NMDA receptor complex interaction by peptide against APPL 1 C-terminal PDZ binding motif blocks activation of Akt by synaptic NMDA receptor, and also blocks NMDA receptor-dependent neuroprotection from starve-induced apoptosis, It suggests that in central nervous system APPL 1, as an adaptor protein, connects synaptic NMDA receptor with intracellular PI3K/Akt cascade and down-stream prosurvival signaling pathway.

关键词： APPL1 NMDA receptor Akt neuroprotection

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Forced notch signaling inhibits commissural axon outgrowth in the developing chick central nerve system

Forced notch signaling inhibits commissural axon outgrowth i...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Ming SHI Min-Hua ZHENG Gang ZHAO Ying HUANG Hua HAN Yu-Qiang DING Department of Neurology Xijing Hospital Fourth Military Medical University Xi''an China Department of Medical Genetics and Developmental Biology Fourth Military Medical University Xi ''an China Department of Anatomy and Neurobiology Tongji University School of Medicine Shanghai China

Objective A collection of in vitro evidence has demonstrated that Notch signaling plays a key role in the growth of neurites in differentiated ***, the effects of Notch signaling on axon outgrowth in an in vivo condition remain largely *** In this study, the neural tubes of HH10-11 chick embryos were in ovo electroporated with various Notch transgenes of activating or inhibiting Notch signaling, and then their effects on commissural axon outgrowth across the floor plate midline in the chick developing central nerve system were *** (1) Forced expression of Notch intracellular domain, constitutively active form of RBPJ, or full-length Hes 1 in the rostral hindbrain, diencephalon and spinal cord at stage HH10-11 significantly inhibited commissural axon outgrowth; (2) Inhibition of Notch signaling by ectopically expressing a dominant-negative form of RBPJ promoted commissural axonal growth along the circumferential axis; (3) These Notch signaling-mediated axon outgrowth defects may be not due to the alteration of axon guidance since commissural axon marker TAG1 was present in the axons in floor plate midline, and also not result from the changes in cell fate determination of commissural neurons since the expression of postmitotic neuron marker Tuj 1 and specific commissural markers TAG1 and Pax7 was *** We first used an in vivo system to provide evidence that forced Notch signaling negatively regulates commissural axon outgrowth.

关键词： Notch commissural neurons axon outgrowth axon guidance in ovo electroporation chick embryo

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Facilitating synaptic transmission of (-)clausenamide in CA1 region of hippocampal slices by activating CaMKⅡα

Facilitating synaptic transmission of (-)clausenamide in CA1...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Jin-Feng HU Na Ning Yu-He YUAN Jun-Tian ZHANG Nai-Hong CHEN Key Laboratory of Bioactive Substances and Resources Utilization Ministry of Education Department of Pharmacology Institute of Materia Medica Chinese Academy of Medical Sciences and Peking Union Medical College Beijing 100050 China

Objective Effect of (-)clausenamide on potentiating field excitatory postsynaptic potential (f-EPSP) in CA1 region of rats' hippocampal slice and its related mechanism were investigated in the *** 8 × 8 multi-electrode array probe was used to measure *** phosphorylation and expression of CaMKⅡα, ERK1/2, CREB were detected by Western *** (-)Clausenamide facilitated synaptic plasticity at dose-dependent *** (-)clausenamide also activated CaMKⅡα and ERK1/*** with KN93 (CaMKⅡα inhibitor, 10 μM) for 20 min before (-)clausenamide addition completely blocked the enhancement of synaptic transimisson induced by (-)***98059 (MEK inhibitor, 30 μM) partially blocked the effect of (-)*** KN93 and PD98059 attenuated the activation of CREB induced by (-)***, the results suggested that CaMKⅡα-ERK1/2-CREB involved in the process of (-)clausenamide facilitating synaptic transmission.U73122, an inhibitor of PLCγ, attenuated the slop of f-EPSP before or after the treating of (-)*** blocked partially the effect of (-)clausenamide by pre-incubation, but showing no difference with basic value when treated after the perfusion of (-)*** data indicated that external calcium influx induced by (-)clausenamide was necessary for initiating synaptic transmission and calcium influx induced by (-)clausenamide may promote the release ofintracellular calcium responsible for synaptic *** (-)clausenamide promoted calcium influx to initiating synaptic transmission and triggering intracellular calcium release, and subsequently activated CaMKⅡα-ERK1/2-CREB signal pathway.

关键词： (-)clausenamide synaptic transmission CaMKⅡα calcium

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Effect of neonatal hypoxia-ischemia brain injury on the function of CA1 pyramidal cells

Effect of neonatal hypoxia-ischemia brain injury on the func...

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中国神经科学学会第九届全国学术会议暨第五届会员代表大会

作者： Yan-Dong ZHAO Wen-Juan HE Lu DU Huai-Zhen RUAN Department of Neurobiology College of Basic Medical Sciences Chongqing Key Laboratory of Neurobiology Third Military Medical University Chongqing 400038 China

Hypoxic-ischemic (HI) brain injury in the perinatal period is one of the most important factors of neonatal mortality and subsequent *** combined effects of cellular energy failure, acidosis, glutamate release, intracellular Ca2+ accumulation, lipid peroxidation, and nitric-oxide neurotoxicity can lead to neuronal apoptosis and necrosis, and then result in the loss of *** the same time, the changes of neural functions should be earlier than the morphological changes; well there is still no report about *** this study, animals were divided into control group and hypoxia-ischemia *** HI brain injury rat model was obtained by unilateral occlusion of left carotid artery, followed by exposure to hypoxia (92% N2, 8% O2) for 1.5 h at 7-day-old *** patch clamp, immunohistochemistry and western bloting technologies were used to test the normal electrophysiological properties, the excitatory postsynaptic currents (EPSCs), the expression and disposition of synaptophysin, VGluT1, NMDAR, and AMPAR of hippocampal CA1 area pyramidal cells immediately after HI injury and *** with the naive control group, CA1 pyramidal neurons of HI models showed that the excitabilities of HI injury hippocampal CA1 pyramidal cells were decreased and the amplitude and duration of EPSCs were *** expression of synaptophysin, NMDAR, and AMPAR was not changed, while the expression of VGluT1 was lower than the control *** of these results indicated that the decrease of neural excitability of HI injury models was the protective responses of neurons, but the damage of HI brain injury was beyond the ability of ***, giving the proper treatment after injury was very necessary.

关键词： hypoxia-ischemia brain injury patch clamp hippocampus

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Role of different subunit-containing NMDA-receptors in retinal spreading depression

Role of different subunit-containing NMDA-receptors in retin...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Paul L Chazot Sura Ali Stevens F Duckett Tihomir P Obrenovitch Department of Biological Sciences University of DurhamDurham DHl 3LEUK School of Pharmacy and Biomedical Sciences University of Central LancashirePreston PR12HEUK School of Pharmacy University of BradfordBradford BD7 1DPUK

Spreading depression(SD) is a temporary disruption of local ionic homeostasis that propagates slowly across the *** depolarisation associated with SD occurs in different types of stroke and may contribute to the initiation of migraine *** receptor(NMDAR) antagonists are known to be the most effective drμgs to suppress *** far,NMDAR antagonist are still perceived as unlikely candidate as anti-SD drμg for migraine aura treatment because of their unacceptable side effects,but the NMDAR antagonist,memantine is now used chronically in elderly for Alzheimer's and epilepsy patients and some subtype selective receptor antagonists may well be effective with reduced side *** an efficient in vitro chick retinal SD model with imaging for NMDAR pharmacology, we have determined whether NMDAR antagonists,with different type NR2 subunit selectivity,can suppress spreading depolarization associated with SD in the chicken retina *** results sμggested NR2A plays a key role in mediating SD,indicating potential therapeutic benefit for NR2A-preferring antagonist to treat SD-related neurological diseases,*** with alleviated side effect.

关键词： Role of different subunit-containing NMDA-receptors in retinal spreading depression

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Peroxynitrite as modulator effects on synaptic transmission in rat hippocampal CA1 neurons

Peroxynitrite as modulator effects on synaptic transmission ...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Yong-Ling XIE,Zhao-Wei LIU,Zhuo YANG~* School of Medicine,Nankai University,Tianjin 300071,China

Objective Peroxynitrite(ONOO),which formed in vivo under the oxidative stress in a diffusioncontrolled reaction between nitric oxide(NO)and superoxide anion,is a strong oxidizing and nitrating agent to mediate much of the toxicity of *** has been suggested that this kind of deleterious reactive nitrogen species(RNS) was involved in the development of a variety of pathological conditions such as the neurodegenerative disorders with disrupted memory and impaired *** aim of this study was to investigate actions of ONOCT on synaptic transmission in hippocampal CA1 neurons of Wistar *** Using patch clamp methods,we made the recording of NMDA-mediated miniature glutamatergic excitatory postsynaptic currents(mEPSC)and AMPA-mediated mEPSC in the hippocampal neurons in CA1 area of Wistar *** Our results demonstrated that SIN-1 (3-morpholino-sydnonimine),used as the donor of ONOO,induced a bidirectional modulation of glutamatergic transmission under different concentrations.A high dose(1 mM)of SIN-1 decreased amplitudes of both NMDA-and AMPA-mediated mEPSC,mainly through altering the *** lower dose(100 uM)of SIN-1 caused a consistent increase in amplitudes of AMPA-and NMDA-mediated mEPSC,but depressed the frequencies. Additionally,co-treatment with SIN-1 and uric acid,a perxynitite scavenger,had no effects on *** The results suggested that perxynitite could induce neurotoxicity through modulating excitatory transmitter release in both presynaptic and postsynaptic ways and play a role in the modification of synaptic function in hippocampal CA1 neurons.

关键词： peroxynitrite synaptic transmission hippocampus slice

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The neurovascular imaging in cerebral ischemia by laser speckle contrast analysis

The neurovascular imaging in cerebral ischemia by laser spec...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Shan-Bao TONG~*,Peng MIAO,Min-Heng LI,Hong-Yang LU,Qi LIU,Yao LI School of Biomedical Engineering,Shanghai Jiao Tong University,Shanghai 2000240,China

Objective Laser speckle contrast analysis has been used in imaging the cerebral vasculature and cerebral blood flow(CBF).However,in practice,the estimation of the CBF may be biased by the ambient background field, dark current and other anomalies in the circuits of CCD camera,non-uniform distributed laser illumination,cortical curvature,breathing/heart beating of the subjects,and the nonstationary change of CBF under functional stimulation, and the motion artifacts if applied to the behavioring *** In order to obtain a stable and high spatiotemporal resolution CBF imaging in the study of the ischemic brain injury,we developed several techniques to improve the current techniques of laser speckle contrast analysis,including(1) enhanced laser speckle contrast analysis (eLASCA);(2) model Based LSI reconstruction;(3) registered laser speckle contrast analysis(rLASCA);(4) Random process estimator for laser speckle *** finally we integrate these techniques and designed a miniature headstage imaging system which can be used to obtain the CBF imaging when the animal(***) is moving freely. Results By using the above techniques,high resolution CBF information can be obtained in real time for either anesthetized or freely moving *** We developed high-resolution optical imaging technique for CBF monitoring,which can be used for real time monitoring both structural and functional neurovascular changing in ischemic brain injury.

关键词： neurovascular imaging cerebral blood flow laser speckle imaging laser speckle contrast analysis

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