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SP8 specifically identifies dorsal lateral ganglionic eminence and dorsal caudal ganglionic eminence derived cortical interneurons

SP8 specifically identifies dorsal lateral ganglionic eminen...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Tong MA,Qiang-Qiang ZHANG,Yu-Qun CAI,Chi-Fei SUN,Yan YOU,Fang LIU,Zhen-Gang YANG~* Institutes of Brain Science and State Key Laboratory of Medical Neurobiology,Fudan University,Shanghai 200032, China

The origin and the diversity of cortical interneurons have been extensively *** studies, using genetic fate mapping,reveal the lateral/caudal ganglionic eminence(LGE/CGE) contribute large population of interneurons to the upper layers of the ***,less is known about this population of cortical interneurons compared with the well characterized MGE derived ones,because of no specific markers available except the recently found CGE lineage marker serotonin 5-hydroxytryptamine 3A(5-HT3A ) *** we show that the zinc finger transcription factor SP8 specifically labels cohorts of mice cortical interneurons originating in the dorsal LGE (dLGE) and dorsal CGE(dCGE).SP8-expressing interneurons initially migrate from rostral dLGE to invade the cortex at embryonic 12.5(E12.5),then from both dLGE and dCGE since *** tangentially migrate through and accumulate in the subventricular/intermediate zone(SVZ/IZ),and only at late embryonic stage and first postnatal week radially migrate into the cortical plate and the marginal ***8-expressing dLGE/dCGE derived interneurons preferentially occupy superficial cortical layers and account for one fifth of all cortical interneurons in adult brain. These results indicate that cortical intemeurons have more heterogeneous origins and subtypes.

关键词： transcription factor SP8 intemeurons lateral ganglionic eminence subventricular zone

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Slit-2 repels the migration of olfactory ensheathing cells by triggering Ca2+-dependent cofilin activation and RhoA inhibition

Slit-2 repels the migration of olfactory ensheathing cells b...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Zhi-Hui HUANG~(1,2,4),Ying WANG~(1,2,4),Zhi-Da SU~1,Jian-Guo GENG~5,Yi-Zhang CHEN~(1,4),Xiao-Bing YUAN~(3,*), Cheng HE~(1,4,*) 1 Institute of Neuroscience and Key Laboratory of Molecular Neurobiology of Ministry of Education,Neuroscience Research Center of Changzheng Hospital,Second Military Medical University,Shanghai 200433,China 2 Institute of Hypoxia Medicine,Wenzhou Medical College,Wenzhou,Zhejiang 325035,China 3 Institute of Neuroscience and State Key Laboratory of Neuroscience,Chinese Academy of Sciences,Shanghai 200031, China 4 Institute of Neuroscience,Zhejiang University School of Medicine,Hangzhou,Zhejiang 310058,China 5 Institute of Biochemistry and Cell Biology,Shanghai Institutes for Biological Sciences,Chinese Academy of Sciences, Shanghai 200031,China

Olfactory ensheathing cells（OECs） migrate from the olfactory epithelium towards the olfactory bulb during ***,the guidance mechanism for OEC migration remains a *** we show that migrating OECs expressed the receptor of the repulsive guidance factor Slit-2.A gradient of Slit-2 in front of cultured OECs first caused the collapse of the leading front,then the reversal of cell *** Slit-2 effects depended on the Ca release from internal stores through IP3 receptor ***,in response to Slit-2 stimulation,collapse of the leading front required the activation of the F-actin severing protein cofilin in a Ca-dependent manner,whereas the subsequent reversal of the soma migration depended on the reversal of RhoA activity across the ***,the Slit- 2-induced repulsion of cell migration was fully mimicked by co-application of inhibitors of F-actin polymerization and RhoA *** findings revealed Slit-2 as a repulsive guidance factor for OEC migration and an unexpected link between Ca and cofilin signaling during Slit-2-triggered repulsion.

关键词： olfactory ensheathing cells Slit-2 migration cofilin Ca2+ RhoA

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The effect of Calmodulin kinase Ⅱ on long-term memory reconsolidation

The effect of Calmodulin kinase Ⅱ on long-term memory recon...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Hong-Yan GUO~(1,2),Min-Qing JIANG~(1,2),Xing-Xia CHEN~(1,2),Hui-Min WANG~(1,2,*) 1 Key Laboratory of Brain Functional Genomics,Ministry of Education,Institute of Cognitive Neuroscience,Shanghai 200062,China 2 East China Normal University,Shanghai 200062,China

Objective Memories are dynamic in *** retrieval,ample evidence has suggested that memories can enter into a labile,protein-synthesis dependent process,referred to as reconsolidation,from which memories can be modified or *** the molecular mechanisms underlying memory reconsolidation could provide crucial insight into the dynamic aspects of memory formation and psychological *** investigate the molecular basis underlying this labile state,we have employed an inducible protein activity-manipulation technique and examined the molecular dynamics of aCaMKIl during long-term memory reactivation in the mouse *** show that an upward-shift of CaMKII activity within the initial 10 minute time-window of synaptic activation is capable of shifting the established long-term potentiation to the higher *** critical time window parallels the initial labile phase of post-reactive long-term memory since post-reactive upward-shift of CaMKII activity during this period also disrupts post-reactive long-term memory ***,post-reactive short-term memory is still intact upon the same ***,we conclude that CaMKII plays a crucial role in numerical and dynamical control and maintenance of synaptic representation of long-term memory traces during reconsolidation process. Methods Using hippocampus-dependent memory(contextual fear memory),or amygdala-dependent memory (auditory fear memory) to test the memory reconsolidation function of *** stimulus(conditioned stimulus,CS) is 85 dB,2800 HZ sound stimulation,non-conditioned stimulus(unconditioned stimulus,US) is the intensity of 0.5 mA for 30 seconds foot electrical *** / CS paired training,the mice continued to stay at the training ground for 30 *** the test process,the mice were put back the original training ground to record 3 minutes freezing response(contextual freezing).Then,mice were placed in a new place,and record CS re

关键词： memories CaMKⅡ transgenic reconsolidation

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Forced notch signaling inhibits commissural axon outgrowth in the developing chick central nerve system

Forced notch signaling inhibits commissural axon outgrowth i...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Ming SHI~(1,*),Min-Hua ZHENG~2,Gang ZHAO~1,Ying HUANG~3,Hua HAN~2,Yu-Qiang DING~(3,*) 1 Department of Neurology,Xijing Hospital,Fourth Military Medical University,Xi'an,China 2 Department of Medical Genetics and Developmental Biology,Fourth Military Medical University,Xi 'an,China 3 Department of Anatomy and Neurobiology,Tongji University School of Medicine,Shanghai,China

Objective A collection of in vitro evidence has demonstrated that Notch signaling plays a key role in the growth of neurites in differentiated ***,the effects of Notch signaling on axon outgrowth in an in vivo condition remain largely *** In this study,the neural tubes of HH10-11 chick embryos were in ovo electroporated with various Notch transgenes of activating or inhibiting Notch signaling,and then their effects on commissural axon outgrowth across the floor plate midline in the chick developing central nerve system were ***(1) Forced expression of Notch intracellular domain,constitutively active form of RBPJ, or full-length Hesl in the rostral hindbrain,diencephalon and spinal cord at stage HH10-11 significantly inhibited commissural axon outgrowth;(2) Inhibition of Notch signaling by ectopically expressing a dominant-negative form of RBPJ promoted commissural axonal growth along the circumferential axis;(3) These Notch signaling-mediated axon outgrowth defects may be not due to the alteration of axon guidance since commissural axon marker TAG1 was present in the axons in floor plate midline,and also not result from the changes in cell fate determination of commissural neurons since the expression of postmitotic neuron marker Tuj 1 and specific commissural markers TAG1 and Pax7 was *** We first used an in vivo system to provide evidence that forced Notch signaling negatively regulates commissural axon outgrowth.

关键词： Notch commissural neurons axon outgrowth axon guidance in ovo electroporation chick embryo

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A circuit model for working memory with analog features

A circuit model for working memory with analog features

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Yan MENG,Hong-Zhi YOU,Da-Hui WANG~* Department of Systems Science,Beijing Normal University,Beijing,100875

Working memory with analogous stimulus plays an important role in the implementation of higher order cognitive *** indicate that the brain apply the distributed representation algorithm to encode and actively maintain the analogous stimulus during the delay period,such as working memory with spatial orientation and *** demonstrate the spatial working memory,Amari type line attractor model has been proposed,in which the bump attractors represent orientation or *** on Amari type line attractor model,characteristics of spatial working memory,such as drift,accuracy,and capacity,have been elaborately ***,Amari type line attractor model was challenged by the recent experiments in which the population firing rate encodes the analogous stimuli such as the value of ***,to actively maintain the value signal,one population of neurons should keep the persistent activity with a firing rate monotonically dependent on the value of *** this study,we simulated the working memory of the value of options using a randomly connected spiking neuron network. The simulations reproduce the observed monotonic persistent activity encoding the value of options during the delay *** spiking neuron model consists of 2,000 neurons,including 1,600 pyramidal cells and 400 inhibitory cells, and its connection probability is 20%.Both pyramidal cells and interneurons are leaky integrate-and-fire（LIF） model. The excitatory postsynaptic currents（EPSCs） are mediated by fast AMPA receptors and slow NMDA receptors,and the inhibitory postsynaptic currents（IPSCs） are mediated by GABA *** to electrophysiological properties of cortical neurons,we introduce two basic mechanisms to encode value of options in the working memory. First,we introduce a voltage-depend cubic nonlinear current into the LIF model of pyramidal cells and interneurons. The membrane potential is bistable and can stay at the depolarized up state

关键词： spiking neuron network line attractor value

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Chromatic processing in mouse retinal ganglion cells

Chromatic processing in mouse retinal ganglion cells

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Tobias Breuninger Thomas Euler Centre for Integrative Neuroscience（CIN） /Institute for Ophthalmic Research University of Tbingen

＜正＞Objective Color processing has been intensively studied in ganglion cells of the primate retina,but in mouse correspondent knowledge is still *** expression in mouse photoreceptor has distinct features such as opsin gradient and co-expression,but it is quite unknown how these features contribute to color processing. The main purpose of our study is to explore this *** We used dichromatic light stimulation（in the photopic range）of the short（S）and the medium（M）wavelength-sensitive mouse cone photoreceptors by two LED light *** relative contribution of S-and M-opsin to ganglion cell input was estimated by modulating the intensity of the two chromatic stimulus components sinusoidally or in *** 2-photon imaging,we targeted GFP-positive ganglion cells in transgenic mice in whole-mounted retina for extracellular *** mouse line we used（GFP-O;Feng et ai,2000）features sparse labeling of various morphological types of retinal ganglion cells. The morphology of the targeted cell was classified using image stacks taken after the ***（1）Our data show that ON alpha ganglion cells（soma diameter＞20μm,dendritic field size＞200μm）received strong M-opsin but weak S-opsin input in dorsal retina,whereas in the ventral retina they received significantly stronger S-opsin input. This is consistent with the presence of a gradient in S-opsin co-expression in M-cones along dorsal-ventral axis in mouse retina.（2）Ratios of M-opsin and S-opsin input to a single alpha ganglion cell estimated by different sizes of stimuli vary in a systematic way,which could be explained by the cooperation of opsin gradient and center-surround *** ganglion cells close to the transition zone of opsin gradient show clear color opponency when tested with large stimuli.（3）Opsin gradient could be observed in the receptive field of a single retinal ganglion cell. Conclusion Large scale opsin inhomogeneity like opsin gradient could lead to differ

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A functional role of TRPMl-L retinal bipolar transduction channel

A functional role of TRPMl-L retinal bipolar transduction ch...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Takahisa Furukawa Osaka Bioscience Institute PRESTO JSTJapan

An essential step in intricate visual processing is the segregation of visual signals into ON and OFF pathways by retinal bipolar *** found that TRPM1-L is a component of the ON BC transduction channel. We identified a mouse trpm1 homologue of human ***1 is alternatively spliced,resulting in the production of a long form（trpm1-L） and a short N-terminal form（trpm1 -S）.Although mouse trpm1-S was previously identified as melastatin,mouse trpm1-L was not *** distinction between trpm1-L or -S,trpm1 has been reported to be detected in human primary melanocytes,poorly metastatic melanoma cell lines,mouse retinal pigment epithelium （RPE）,and subsets of ON and OFF *** found that TRPM 1 -L localization is developmentally restricted to the dendritic tips of ON BCs in co-localization with mouse GRM6（mGluR6）.Trpm1-null mutant mice completely lose the photoresponse of ON ***1-L channel activity is negatively regulated by activated G in the mGluR6 *** examined if TRPM1-L is regulated by Goa or *** performed inside-out patch clamp in the presence of purified brain Goαor Gβγusing TRPM1-L expressing CHO *** then investigated a possible involvement of TRPM1 abnormality in pathogenesis of human retinal *** identified human TRPM1 gene mutations in congenital stationary night blindness（CSNB） *** retinopathy（PR）,including cancer-associated retinopathy（CAR） and melanoma-associated retinopathy（MAR）,is a progressive retinal disease caused by antibodies generated against neoplasms not associated with the *** several autoantibodies against retinal antigens have been identified,there has been no known autoantibody reacting specifically against bipolar cell antigens in the sera of patients with *** recently found that TRPM1 is one of the autoantigens targeted by autoantibodies in patients with CAR or MAR associated with retinal ON bipolar cell ***,our results suggest that TRPM1 plays an

关键词： TRPMl channel bipolar cell night blindness cancer-associated retinopathy

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NMDA-receptor antagonist MK-801 attenuates MMN-like responses in both awake and anesthetized rats

NMDA-receptor antagonist MK-801 attenuates MMN-like response...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Ying-Ying LIU~(1,2),Ning WANG~1,Jin-Yan WANG~1,Fei LUO~(1,*) 1 Key Laboratory of Mental Health,Institute of Psychology,Chinese Academy of Sciences,Beijing 100101,China 2 Graduate University of Chinese Academy of Sciences,Beijing 100101,China

Objective Schizophrenia patients always have significant attenuated mismatch negativity(MMN) response,which reveals a deficient cognitive process precision on pre-attentive sensory *** in NMDA receptor system are supposed to mediate the MMN attenuation on schizophrenia ***,there are few studies on MMN response in animal schizophrenia model,especially on awake ***,this study is aimed to explore the proper condition to evoke MMN responses in both awake and anesthetized rats,compare MMN responses under different consciousness states,and estimate whether the NMDA receptor antagonist MK-801 could block the MMN *** Auditory evoked potentials were recorded during odd ball paradigm,in different states(awake,anesthetized),drugs(saline,low dose MK-801,high dose MK-801) and inter-stimulus intervals(ISI) (1000 ms,2000 ms,3000 ms).Results(1) MMN response in awake rats presented two negative components which peaked at 50 ms and 120 ms ***,in the anesthetized rats,the MMN response was one positive slow wave happened during 150-350 ms.(2) Compared with the saline injection,both the low-dose and high-dose MK-801 injection could significantly attenuate the MMN responses in the two states.(3) MMN responses had a significantly lager amplitude at the 3000 ms ISI both during the awake and anesthetized states,compared with the 1000 ms and 2000 ms *** MMN response could be estimated in both awake and anesthetized rats. The NMDA receptor antagonist MK-801,which suggested a temporary schizophrenia simulation,could attenuate the MMN response.

关键词： schizophrenia MMN NMDA receptor MK-801

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Activation of CDK5 by noncanonical Wnt/Ca2+ signaling during neurogenesis and neuronal differentiation

Activation of CDK5 by noncanonical Wnt/Ca2+ signaling during...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Zheng WEN,Yang SHU,Bo TIAN~* Department of Neurobiology,Tongji Medical School,Huazhong University of Science and Technology,Wuhan 430030, China

Objective Canonical Wnt/β-catenin signals play important roles during neuronal differentiation and neurogenesis,but less is known about noncanonical Wnt signals in this ***,increasing evidence demonstrates that noncanonical Wnt/Ca signaling may participate in the neuronal *** kinase 5（Cdk5） has been previously implicated in the maturation and survival of neurons born during embryonic *** the present study,the role of noncanonical Wnt signals and its relationship with Cdk5 activation in neurogenesis and neuronal differentiation is to be *** The expression and secretion of noncanonical Wnt ligands（Wnt-4,Wnt-5a and Wnt-11） in cortex of different embryonic stages,and RA/BDNF-induced differentiated SH-SY5Y cells were measured by quantitative Real-time PCR（qRT-PCR） and ELISA assays. The Cdk5 activation by Wnt ligands was tested by in vitro kinase *** effects of Canonical Wnt/β-catenin/TCF signaling were measured by Top/Fop-Flash reporter ***（1） The expression and secretion of Wnt-4,Wnt- 5a and Wnt-11 were all increased in embryonic cortex of different stages and differentiated SH-SY5Y cells.（2） Wnt- 5a,but not Wnt-11,can directly activate Cdk5 kinase activity in neuron and is essential in neuronal differentiation.（3） The activation of Cdk5 by Wnt-5a is and not dependent onβ-catenin *** Noncanonical Wnt/Ca signaling may participate in the neurogenesis and neuronal differentiation by activation of Cdk5.

关键词： Wnt/Ca2+ signaling CDK5 calpain neurogenesis

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Microsphere embolism-induced microglia activation via P2X7-FasL signaling

Microsphere embolism-induced microglia activation via P2X7-F...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Geng-Sheng ZHANG,Rong-Rong TAO,Ying-Mei LU,Ji-Yun HUANG,Yue-Long JI,Mei-Hua LIAO, Jun-Feng ZHU,Xue-Jing SHAO,Huan-Wei WANG,Shen-Quan YAN,Feng HAN~* Institute of Pharmacology,Toxicology and Biochemical Pharmaceutics,Zhejiang University,Hangzhou,China.

Objective Emerging epidemiological and clinical evidence indicates that microglia-induced neurotoxicity may be a factor contributing to the development of ischemia or neurodegeneration in response to pathological signals. The current study focused on the temporal,morphological and biochemical aspects of FasL and its expression during the pathological process of cerebral microemboli in *** characterized a mechanism whereby activated microglia mediate neuronal cell death via P2X purinergic receptors and FasL/Fas signaling in response to *** The intracellular events following microsphere embolism were identified by immunohistochemistry study and Western blot *** role of P2X-FasL signaling was addressed by pharmacological approaches and FasL-deficient（gld/ gld） *** Based on our observations,we predict that the early activation of P2X purinoceptors in microglia results in gradual accumulation of FasL and the subsequent activation of downstream ***,in turn,further exaggerates P2X-mediated microglial activation and triggers a vicious circle of neuronal cell *** findings show that FasL is a necessary component of microglia-induced cell death in vitro and in vivo and introduce a novel apoptotic stimulus that works through a P2X-dependent *** These results highlight the potential of a novel therapeutic neuroprotective approach using pharmacological blockade of sustained microglial activation and the death-inducing ligand.

关键词： microsphere embolism ischemia microglia Fas ligand P2X7 receptor neuronal cell death

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