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Forced notch signaling inhibits commissural axon outgrowth in the developing chick central nerve system

Forced notch signaling inhibits commissural axon outgrowth i...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Ming SHI~(1,*),Min-Hua ZHENG~2,Gang ZHAO~1,Ying HUANG~3,Hua HAN~2,Yu-Qiang DING~(3,*) 1 Department of Neurology,Xijing Hospital,Fourth Military Medical University,Xi'an,China 2 Department of Medical Genetics and Developmental Biology,Fourth Military Medical University,Xi 'an,China 3 Department of Anatomy and Neurobiology,Tongji University School of Medicine,Shanghai,China

Objective A collection of in vitro evidence has demonstrated that Notch signaling plays a key role in the growth of neurites in differentiated ***,the effects of Notch signaling on axon outgrowth in an in vivo condition remain largely *** In this study,the neural tubes of HH10-11 chick embryos were in ovo electroporated with various Notch transgenes of activating or inhibiting Notch signaling,and then their effects on commissural axon outgrowth across the floor plate midline in the chick developing central nerve system were ***(1) Forced expression of Notch intracellular domain,constitutively active form of RBPJ, or full-length Hesl in the rostral hindbrain,diencephalon and spinal cord at stage HH10-11 significantly inhibited commissural axon outgrowth;(2) Inhibition of Notch signaling by ectopically expressing a dominant-negative form of RBPJ promoted commissural axonal growth along the circumferential axis;(3) These Notch signaling-mediated axon outgrowth defects may be not due to the alteration of axon guidance since commissural axon marker TAG1 was present in the axons in floor plate midline,and also not result from the changes in cell fate determination of commissural neurons since the expression of postmitotic neuron marker Tuj 1 and specific commissural markers TAG1 and Pax7 was *** We first used an in vivo system to provide evidence that forced Notch signaling negatively regulates commissural axon outgrowth.

关键词： Notch commissural neurons axon outgrowth axon guidance in ovo electroporation chick embryo

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Effect of chronic unpredictable stress on conditioning fear behavior and somatosensory evoked potential of rats

Effect of chronic unpredictable stress on conditioning fear ...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Hong-Ping HUANG~(1,*),Qing-Chun ZHU~1,Feng WANG~2,Bang-An WANG~1,Huan-Huan ZHANG~1,Meng-Ya WANG~1 1 Department of Humanity and Manage,Cell Electrophysiology Lab 2 Department of The Third Medicine,Wannan Medical College,Wuhu 241002,China

Objective Chronic unpredictable stress was commonly used to evoke depression-like behavior in *** present study was undertaken to investigate the effect of chronic unpredictable stress on conditioning fear behavior and somatosensory evoked potential(SEP) of *** correlation of the conditioning fear behavior and SEP was also *** Twenty-seven male SD rats(weight 190-200 g) were random divided into control group and model *** in model group were subjected to chronic unpredictable *** fear behavior of rats was observed with Freeze monitor system and the latency of somatosensory evoked potential was evaluated with Powerlab *** Compared with control group,rats in model group had decreased freezing behavior responded to the sound stimulation during fear memory stage(56.64%±13.78%,n = 13,vs 69.72%±18.10%,n = 14, P<0.05,mean±SD).The latency of SEP decreased in the rats treated with chronic unpredictable stress(70.54±10.13 ms,n = 14 vs 78.46±7.80 ms,n = 14,P<0.05),which had the significant difference between the two group rats. The correlation coefficient of the conditioning fear behavior and SEP was positive(r = 0.507,P<0.05).Conclusion Chronic unpredictable stress inhibited conditioning fear behavior of rats,decreased latency of SEP,which suggests chronic unpredictable stress stimulations active the neurotransmitter network system involved with both conditioning fear behavior and SEP.

关键词： chronic unpredictable stress conditioning fear behavior somatosensory evoked potential rat

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The effect of Calmodulin kinase Ⅱ on long-term memory reconsolidation

The effect of Calmodulin kinase Ⅱ on long-term memory recon...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Hong-Yan GUO~(1,2),Min-Qing JIANG~(1,2),Xing-Xia CHEN~(1,2),Hui-Min WANG~(1,2,*) 1 Key Laboratory of Brain Functional Genomics,Ministry of Education,Institute of Cognitive Neuroscience,Shanghai 200062,China 2 East China Normal University,Shanghai 200062,China

Objective Memories are dynamic in *** retrieval,ample evidence has suggested that memories can enter into a labile,protein-synthesis dependent process,referred to as reconsolidation,from which memories can be modified or *** the molecular mechanisms underlying memory reconsolidation could provide crucial insight into the dynamic aspects of memory formation and psychological *** investigate the molecular basis underlying this labile state,we have employed an inducible protein activity-manipulation technique and examined the molecular dynamics of aCaMKIl during long-term memory reactivation in the mouse *** show that an upward-shift of CaMKII activity within the initial 10 minute time-window of synaptic activation is capable of shifting the established long-term potentiation to the higher *** critical time window parallels the initial labile phase of post-reactive long-term memory since post-reactive upward-shift of CaMKII activity during this period also disrupts post-reactive long-term memory ***,post-reactive short-term memory is still intact upon the same ***,we conclude that CaMKII plays a crucial role in numerical and dynamical control and maintenance of synaptic representation of long-term memory traces during reconsolidation process. Methods Using hippocampus-dependent memory(contextual fear memory),or amygdala-dependent memory (auditory fear memory) to test the memory reconsolidation function of *** stimulus(conditioned stimulus,CS) is 85 dB,2800 HZ sound stimulation,non-conditioned stimulus(unconditioned stimulus,US) is the intensity of 0.5 mA for 30 seconds foot electrical *** / CS paired training,the mice continued to stay at the training ground for 30 *** the test process,the mice were put back the original training ground to record 3 minutes freezing response(contextual freezing).Then,mice were placed in a new place,and record CS re

关键词： memories CaMKⅡ transgenic reconsolidation

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Chromatic processing in mouse retinal ganglion cells

Chromatic processing in mouse retinal ganglion cells

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Tobias Breuninger Thomas Euler Centre for Integrative Neuroscience（CIN） /Institute for Ophthalmic Research University of Tbingen

＜正＞Objective Color processing has been intensively studied in ganglion cells of the primate retina,but in mouse correspondent knowledge is still *** expression in mouse photoreceptor has distinct features such as opsin gradient and co-expression,but it is quite unknown how these features contribute to color processing. The main purpose of our study is to explore this *** We used dichromatic light stimulation（in the photopic range）of the short（S）and the medium（M）wavelength-sensitive mouse cone photoreceptors by two LED light *** relative contribution of S-and M-opsin to ganglion cell input was estimated by modulating the intensity of the two chromatic stimulus components sinusoidally or in *** 2-photon imaging,we targeted GFP-positive ganglion cells in transgenic mice in whole-mounted retina for extracellular *** mouse line we used（GFP-O;Feng et ai,2000）features sparse labeling of various morphological types of retinal ganglion cells. The morphology of the targeted cell was classified using image stacks taken after the ***（1）Our data show that ON alpha ganglion cells（soma diameter＞20μm,dendritic field size＞200μm）received strong M-opsin but weak S-opsin input in dorsal retina,whereas in the ventral retina they received significantly stronger S-opsin input. This is consistent with the presence of a gradient in S-opsin co-expression in M-cones along dorsal-ventral axis in mouse retina.（2）Ratios of M-opsin and S-opsin input to a single alpha ganglion cell estimated by different sizes of stimuli vary in a systematic way,which could be explained by the cooperation of opsin gradient and center-surround *** ganglion cells close to the transition zone of opsin gradient show clear color opponency when tested with large stimuli.（3）Opsin gradient could be observed in the receptive field of a single retinal ganglion cell. Conclusion Large scale opsin inhomogeneity like opsin gradient could lead to differ

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A functional role of TRPMl-L retinal bipolar transduction channel

A functional role of TRPMl-L retinal bipolar transduction ch...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Takahisa Furukawa Osaka Bioscience Institute PRESTO JSTJapan

An essential step in intricate visual processing is the segregation of visual signals into ON and OFF pathways by retinal bipolar *** found that TRPM1-L is a component of the ON BC transduction channel. We identified a mouse trpm1 homologue of human ***1 is alternatively spliced,resulting in the production of a long form（trpm1-L） and a short N-terminal form（trpm1 -S）.Although mouse trpm1-S was previously identified as melastatin,mouse trpm1-L was not *** distinction between trpm1-L or -S,trpm1 has been reported to be detected in human primary melanocytes,poorly metastatic melanoma cell lines,mouse retinal pigment epithelium （RPE）,and subsets of ON and OFF *** found that TRPM 1 -L localization is developmentally restricted to the dendritic tips of ON BCs in co-localization with mouse GRM6（mGluR6）.Trpm1-null mutant mice completely lose the photoresponse of ON ***1-L channel activity is negatively regulated by activated G in the mGluR6 *** examined if TRPM1-L is regulated by Goa or *** performed inside-out patch clamp in the presence of purified brain Goαor Gβγusing TRPM1-L expressing CHO *** then investigated a possible involvement of TRPM1 abnormality in pathogenesis of human retinal *** identified human TRPM1 gene mutations in congenital stationary night blindness（CSNB） *** retinopathy（PR）,including cancer-associated retinopathy（CAR） and melanoma-associated retinopathy（MAR）,is a progressive retinal disease caused by antibodies generated against neoplasms not associated with the *** several autoantibodies against retinal antigens have been identified,there has been no known autoantibody reacting specifically against bipolar cell antigens in the sera of patients with *** recently found that TRPM1 is one of the autoantigens targeted by autoantibodies in patients with CAR or MAR associated with retinal ON bipolar cell ***,our results suggest that TRPM1 plays an

关键词： TRPMl channel bipolar cell night blindness cancer-associated retinopathy

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NMDA-receptor antagonist MK-801 attenuates MMN-like responses in both awake and anesthetized rats

NMDA-receptor antagonist MK-801 attenuates MMN-like response...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Ying-Ying LIU~(1,2),Ning WANG~1,Jin-Yan WANG~1,Fei LUO~(1,*) 1 Key Laboratory of Mental Health,Institute of Psychology,Chinese Academy of Sciences,Beijing 100101,China 2 Graduate University of Chinese Academy of Sciences,Beijing 100101,China

Objective Schizophrenia patients always have significant attenuated mismatch negativity(MMN) response,which reveals a deficient cognitive process precision on pre-attentive sensory *** in NMDA receptor system are supposed to mediate the MMN attenuation on schizophrenia ***,there are few studies on MMN response in animal schizophrenia model,especially on awake ***,this study is aimed to explore the proper condition to evoke MMN responses in both awake and anesthetized rats,compare MMN responses under different consciousness states,and estimate whether the NMDA receptor antagonist MK-801 could block the MMN *** Auditory evoked potentials were recorded during odd ball paradigm,in different states(awake,anesthetized),drugs(saline,low dose MK-801,high dose MK-801) and inter-stimulus intervals(ISI) (1000 ms,2000 ms,3000 ms).Results(1) MMN response in awake rats presented two negative components which peaked at 50 ms and 120 ms ***,in the anesthetized rats,the MMN response was one positive slow wave happened during 150-350 ms.(2) Compared with the saline injection,both the low-dose and high-dose MK-801 injection could significantly attenuate the MMN responses in the two states.(3) MMN responses had a significantly lager amplitude at the 3000 ms ISI both during the awake and anesthetized states,compared with the 1000 ms and 2000 ms *** MMN response could be estimated in both awake and anesthetized rats. The NMDA receptor antagonist MK-801,which suggested a temporary schizophrenia simulation,could attenuate the MMN response.

关键词： schizophrenia MMN NMDA receptor MK-801

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CXCLl in spinal cord astrocytes contributes to neuropathic pain

CXCLl in spinal cord astrocytes contributes to neuropathic p...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Zhi-Jun ZHANG~1,De-Li CAO~1,Ru-Rong JI~2,Yong-Jing GAO~1 1 Institute of Nautical Medicine,Nantong University,Jiangsu,China 2 Sensory Plasticity Laboratory,Pain Research Center,Department of Anesthesiology,Brigham and Women's Hospital and Harvard Medical School,Boston, Massachusetts 02115

Neuropathic pain is one of the most widespread and intractable of human *** is recognized to play key roles in the development and maintenance of neuropathic *** cytokines and chemokines have been found to be involved in the neuroinflammation in both peripheral and central nervous system. CXCL1[Growth-Related Oncogene(GRO) or keratinocyte-derived chemokines(KC)]is one chemokine in CXC family and was found to be expressed in dorsal root ganglion neurons and increased after spinal nerve *** the expression of CXCL1 in spinal cord and the role in neuropathic pain have not been *** previous study showed that TNF-αincreased CXCL1 expression in cultured *** the present study,we found that intrathecal injection of TNF-αinduced pain hypersensitivity as well as CXCL1 upregulation in the spinal *** addition,spinal nerve ligation(SNL) induced persistent CXCL1 upregulation in the spinal cord,which started from day 3,peaked at day 10 and maintained for more than 21 ***,CXCL1 was colocalized with the astrocytic maker GFAP, but not with microglial marker OX42 or neuronal marker NeuN,indicating the expression of CXCL1 in astrocytes. Furthermore,spinal application of CXCL1 induced pain sensitization and Fos expression in the spinal cord neurons. Spinal administration of CXCR2 antagonist attenuated neuropathic pain ***,our results suggest that CXCL1-CXCR2 may be involved in the maintenance of neuropathic pain by regulating the astrocytic-neuronal interaction in the spinal cord following spinal nerve *** of the CXCL1-CXCR2 may provide a new therapy for neuropathic pain management.

关键词： CXCLl CXCR2 chemokines astrocytes neuropathic pain

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Ethanol blocks long-term potentiation of GABAergic synapses in the ventral tegmental area involving mu-opioid receptors

Ethanol blocks long-term potentiation of GABAergic synapses ...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Yan-Zhong GUAN~(1,2),Jiang-Hong YE~1 1 Department of Anesthesiology,University of Medicine and Dentistry of New Jersey,New Jersey Medical School, Newark,NJ,07103,USA 2 Department of Physiology,Mudanjiang Medical University,Mudanjiang 157011,China

Objective It is well documented that ethanol exposure alters GABA（Y-aminobutyric acid）-releasing synapses,and ethanol addiction is associated with endogenous opioid *** evidence indicates that opioids block long-term potentiation in the fast inhibitory GABA receptor synapses(LTP) onto dopamine-containing neurons in the ventral tegmental area（VTA）,a brain region essential for reward-seeking ***,how ethanol affects LTP is not *** present study is to explore the effects of ethanol on long-term potentiation of GABAergic synapses in the ventral tegmental *** Using whole-cell patch clamp methods we recorded evoked IPSCs from DA neurons in acute coronal midbrain slices of young Sprague-Dawley *** We report here that in acute midbrain slices from rats,clinically relevant concentrations of ethanol applied both in vitro and in vivo prevents LTP,which is reversed,respectively,by in vitro and in vivo administration of naloxone,a mu-opioid receptor（MOR） ***,the blockade of LTP induced by a brief in vitro ethanol treatment is mimicked by DAMGO（[D-Ala2,N-MePhe4,Gly-ol]-enkephalin）,a MOR ***-pulse ratios are similar in slices,24 h after in vivo injection with either saline or ***-cAMPS,a stable cAMP analog,and pCPT-cGMP, a cGMP analog,potentiate GABA-mediated inhibitory postsynaptic currents in slices from ethanol-treated rats, indicating that a single in vivo ethanol exposure does not maximally increase GABA ***,ethanol produces a long-lasting inability to generate *** Ethanol blocks long-term potentiation of GABAergic synapses in the ventral tegmental area involving *** neuroadaptations to ethanol might contribute to early stage of addiction.

关键词： ethanol long-term potentiation GABAergic synapses ventral tegmental area

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Microsphere embolism-induced microglia activation via P2X7-FasL signaling

Microsphere embolism-induced microglia activation via P2X7-F...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Geng-Sheng ZHANG,Rong-Rong TAO,Ying-Mei LU,Ji-Yun HUANG,Yue-Long JI,Mei-Hua LIAO, Jun-Feng ZHU,Xue-Jing SHAO,Huan-Wei WANG,Shen-Quan YAN,Feng HAN~* Institute of Pharmacology,Toxicology and Biochemical Pharmaceutics,Zhejiang University,Hangzhou,China.

Objective Emerging epidemiological and clinical evidence indicates that microglia-induced neurotoxicity may be a factor contributing to the development of ischemia or neurodegeneration in response to pathological signals. The current study focused on the temporal,morphological and biochemical aspects of FasL and its expression during the pathological process of cerebral microemboli in *** characterized a mechanism whereby activated microglia mediate neuronal cell death via P2X purinergic receptors and FasL/Fas signaling in response to *** The intracellular events following microsphere embolism were identified by immunohistochemistry study and Western blot *** role of P2X-FasL signaling was addressed by pharmacological approaches and FasL-deficient（gld/ gld） *** Based on our observations,we predict that the early activation of P2X purinoceptors in microglia results in gradual accumulation of FasL and the subsequent activation of downstream ***,in turn,further exaggerates P2X-mediated microglial activation and triggers a vicious circle of neuronal cell *** findings show that FasL is a necessary component of microglia-induced cell death in vitro and in vivo and introduce a novel apoptotic stimulus that works through a P2X-dependent *** These results highlight the potential of a novel therapeutic neuroprotective approach using pharmacological blockade of sustained microglial activation and the death-inducing ligand.

关键词： microsphere embolism ischemia microglia Fas ligand P2X7 receptor neuronal cell death

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The effects of positive emotion on working memory:researches from ERPS

The effects of positive emotion on working memory:researches...

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中国神经科学学会第九届全国学术会议暨第五次会员代表大会

作者： Yun-Ying DONG~1,Xin GAO~1,Ren-Lai ZHOU~(1,2,3,*),Pan LIU~2 1 Key Laboratory of Child Development and Learning Science(Southeast University),Ministry of Education,Nanjing 210096,China 2 State Key Laboratory of Cognitive Neuroscience and Learning,Beijing Normal University,Beijing 100875,China 3 Beijing Key Lab of Applied Experimental Psychology,Beijing Normal University,Beijing 100875,China

Objective Working memory is divided into separable subsystems,spatial working memory and verbal working memory involving manipulation and retention of visuospatial and verbal information *** present study is to assess the infuences of positive emotion on spatial and verbal working *** We applied the modified DMS paradigm("picture - target - delay - probe" 4 stages for an experimental unit) with the positive and neutral picture from the International Affective Picture System(IAPS;Lang,Bradley & Cuthbert,1999),using ERP technology in healthy ***(a) Positive emotion impairs low load spatial working memory task, not high load,which is revealed by the lower P3 and slow wave in the maintaining phase,and the higher of early LPP, late LPP,and slow wave in the matching phase in the low load spatial working memory task,(b) Positive emotion in a certain extent facilitates both high and low load verbal working memory,which is revealed by the shorter task reaction time,and the higher mean amplitude of P2 in the decoding phase,the higher P3 and slow wave in the maintaining phase,and the lower LPP in the matching *** ERP components respectively reflected attention allocation, attention maintenance,and executive control processes of working *** This research finds the selective effects of positive emotion on working memory,and the interaction with working memory task load,which could primarily demonstrate the potential mechanism of how positive emotion affects working memory.

关键词： positive emotion spatial working memory verbal working memory task load

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