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检索条件"任意字段=第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议"

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Study on the effect of Neuropeptide (Nesfatin-1) on epileptic discharges in rat insular cortex neurons

Study on the effect of Neuropeptide (Nesfatin-1) on epilepti...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Shuang-lai Ren Zheng Liu Tao Sun NingXia key Laboratory of Cerebrocranial Disease Ningxia Medical University NingXia Institute of Medical Sciences Depatment of Neurosurgery Affiliated Hospital of Ningxia Medical University

Nesfatin-1 is the discovered peptide hormones involved in the control of appetite in 2006. Besides its main appetite-control function, it causes depolarization in the paraventricular nucleus(PVN). However, the effect of nesfatin-1 on epilepsy is still not clear. Our research found that serum and hypothalamus nesfatin-1 levels from epilepsy rat kindled by kainic acid was significantly higher(P<0.05)than control group rat's level. The expression of Nesfatin-1 was decreased significant(P<0.05)after anti-epilepsy with valproate(VPA) treatment, while higher than the blank control group. Tissue samples were analyzed for nesfatin-1 expression by immunochemistry, serum nesfatin-1 levels by ELISA. Epilepsy is one of the most common neurological disorders. In order to further investigate the antiepileptic mechanism of nesfatin-1, the patch clamp technique is used to observe the effects of nesfatin-1 on 4-aminopyridine(4-AP) induced epileptiform events in neurons of rat insular slices. We used voltage clamp-whole cell recording mode to acquire electric currents by Gap Free protocol, 10μM nesfatin-1 was added through ACSF perfusion in brain slices for more than 20 minutes, we found no significant change of epileptiform induced by 4-AP(n=6), such as, the generation frequency of epilepsy, and generation mode of epilepsy. So the exactitude detection of expression level of Nesfatin-1 in the serum could reflect the neuropeptide variance level in hypothalamus. This method could be the simpler, sensitive and noninvasive way on epilepsy diagnose, but is not good potent target for epilepsy therapy.

关键词： epilepsy Nesfatin-1 insular cortex brain slices patch clamp

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Exogenous relaxin-3 in dentate gyrus facilitates paired-pulse inhibitory responses as a measure of short-term synaptic plasticity

Exogenous relaxin-3 in dentate gyrus facilitates paired-puls...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Mohsen Nategh Fereshteh Motamedi Neuroscience Research Center Shahid Beheshti University of Medical Sciences

The pontine nucleus incertus has been well characterized as a main source of relaxin-3 in rodent brain which projects to different areas including the hippocampus. In this study we examined effects of exogenous relaxin-3 on short-term and long-term synaptic plasticity of dentate gyrus(DG) which expresses the relaxin-3 cognate receptor, RXFP3. Male Wistar rats were implanted with stimulating electrode in perforant path, and recording electrode in DG which was accompanied by a cannula. Field potential activity was recorded in freely moving state, one week after the surgery. It started by recording baseline synaptic activity, paired-pulse stimulations, induction of long-term potentiation(LTP), and post-LTP recording for 2h and again at 24 h. About 540 pmol of relaxin-3 was diluted in the vehicle(10% acetonitril in saline) and injected into DG 30 min before induction of LTP. Short-term plasticity was also studied by paired-pulse stimulations which preceded LTP induction. Comparison of relaxin-3 and the control group(vehicle) showed that relaxin-3 not only significantly decreased paired-pulse depression at 20 ms and 240 ms inter-pulse intervals, but even facilitated these responses. LTP on the other hand was induced in both groups, but showed no difference between them during 120 min, or one day later. Based on these observations, it is concluded that relaxin-3 alters short-term synaptic plasticity in DG, but does not affect long-term plasticity. Relaxin-3, co-released with GABA in NI-DG synapses, is not the only way with which NI can affect hippocampus. Further research should consider indirect connections of NI with hippocampus, such as the medial septum, to fully identify potential effects of NI in hippocampal synaptic plasticity.

关键词： relaxin-3 dentate gyrus short-term plasticity

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Identification of novel genes involved in learning and memory by mouse forward genetic

Identification of novel genes involved in learning and memor...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Xuebo Cheng Yichang Jia School of Medicine Tsinghua University

Known from classical genetics studies in model organisms, such as ***, D. melanogaster, and so on, point mutation could be more informative to human disease than knockout mutation. As the complete loss of the function of the gene may contribute to lethal or a compensatory mechanisms will be initiated and take over the function of the deleted gene. Mouse, as a mammal with complicated brain and behavior output, is a valuable tool for identifying and studying the pathways responsible for certain neurological disease. Cognitive deficiency is common in many neurological diseases, including Alzheimer's disease and autism spectrum disorders. To point out the mechanism of cognition will facilitate us to know the pathogenesis of the diseases and supply potential treatment. Combined above, a chemical mutagen, N-ethyl N-nitrosourea, was injected into C57BL/6J mouse for forward genetic screening. As the mutation sites are caused without any bias and could lead us to discover novel genes for the related phenotypes. In our study, two well established behavior paradigms, contextual fear conditioning and novel object recognition, were adopted for evaluating the hippocampus dependent long term memory ability. After G1 dominant screening, 3 mutant strains carrying inheritable learning and memory deficits were identified. With the combination of exon enrichment and next-generation sequencing, candidates were narrowed down to one gene in two mutant families. One of the potential causative gene is Mark2(MAP/Microtubule affinity regulating kinase2), which was reported to be involved in Alzheimer's disease. Meanwhile, the Mark2 mutant mice also exhibit a compulsive behavior, with repetitive glooming and leading to neck and head lesions. Therefore, our approach is feasible to identify novel genes in cognitive process. Further study will provide us new insights into the molecular basis of the cognitive process and etiology of neurological diseases.

关键词： Forward genetics,Learning and memory,Mouse,Hippocampus

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Neuritin reverses deficits in murine novel object associative recognition memory caused by exposure to extremely low-frequency (50Hz) electromagnetic fields

Neuritin reverses deficits in murine novel object associativ...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Qian-Ru Zhao Jun-Mei Lu Jin-Jing Yao Zheng-Yu Zhang Chen Ling Yan-Ai Mei Institutes of Brain Science School of Life Sciences and State Key Laboratory of Medical NeurobiologyFudan University Division of Cellular and Molecular Therapy Department of PediatricsUniversity of Florida College of Medicine

Animal studies have shown that electromagnetic field exposure may interfere with the activity of brain cells, thereby generating behavioral and cognitive disturbances. However, the underlying mechanisms and possible preventions are still unknown. In this study, we used a mouse model to examine the effects of exposure to extremely low-frequency(50 Hz) electromagnetic fields(ELF MFs) on a recognition memory task and morphological changes of hippocampal neurons. The data showed that ELF MFs exposure(1 m T, 12 h/day) induced a time-dependent deficit in novel object associative recognition memory and also decreased hippocampal dendritic spine density. This effect was observed without corresponding changes in spontaneous locomotor activity and was transient, which has only been seen after exposing mice to ELF MFs for 7-10 days. The over-expression of hippocampal neuritin, an activity-dependent neurotrophic factor, using an adeno-associated virus(AAV) vector significantly increased the neuritin level and dendritic spines density. This increase was paralleled with ELF MFs exposure-induced deficits in recognition memory and reductions of dendritic spine density. Collectively, our study provides evidence for the association between ELF MFs exposure, impairment of recognition memory, and resulting changes in hippocampal dendritic spine density. Neuritin prevented this ELF MFs-exposure-induced effect by increasing the hippocampal spine density.

关键词： extremely low-frequency electromagnetic fields neuritin dendrite spines novel object recognition test hippocampus

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The insect analog of norepinephrine mediates starvation-induced foraging behavior in adult Drosophila

The insect analog of norepinephrine mediates starvation-indu...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Zhe Yang Yue Yu Vivian Zhang Yinjun Tian Wei Qi Liming Wang Life Sciences Institute Zhejiang University Department of Molecular and Cell Biology University of California

The central nervous system plays a critical role in energy homeostasis by monitoring changes in metabolic state and modulating energy intake and expenditure in response, including sensory physiology, foraging and feeding behaviors, and memory formation. Starved animals often exhibit elevated locomotion, which has been speculated to partly resemble foraging behavior and facilitate food acquisition and energy intake. Despite its importance, the neural mechanism underlying starvation-induced hyperactivity remains unknown in any species. In this study we confirm and extend previous findings that starvation induces locomotor activity in adult fruit flies Drosophila melanogaster. Starvation-induced hyperactivity can be suppressed by food cues via both central nutrient-sensing and peripheral sweet-sensing mechanisms, suggesting that this behavior is directed towards food localization and acquisition, and therefore partly resembles foraging behavior. We further find that octopamine, the insect counterpart of vertebrate norepinephrine, as well as the neurons expressing octopamine, are both necessary andsufficient for starvation-induced hyperactivity. Despite its importance in starvation-induced hyperactivity, octopamine is not required for starvation-induced changes in feeding behavior including elevated preference towards nutritious food over non-nutritious food, higher tendency to initiate feeding, and increased food consumption, suggesting independent regulations of energy intake behaviors by starvation. Taken together, our results establish a quantitative behavioral paradigm to investigate the regulation of energy homeostasis by the CNS, and identified a conserved neural substrate that links organismal metabolic state to a specific behavioral output.

关键词： The insect analog of norepinephrine mediates starvation-induced foraging behavior in adult Drosophila

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Contribution of somatostatin- and parvalbumin-expressing interneurons to feature specific surround suppression in mouse V1

Contribution of somatostatin- and parvalbumin-expressing int...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Fengling Li Haishan Yao Institute of Neuroscience and State Key Laboratory of Neuroscience Shanghai Institutes for Biological SciencesChinese Academy of Sciences University of Chinese Academy of Sciences

The responses of V1 neurons to stimuli in the receptive field(RF) can be modulated by stimuli outside the RF. The surround modulation often manifests as surround suppression, and the degree of suppression depends on the similarity between stimuli in the RF and the surround. Such feature specific surround suppression may underlie the perceptual phenomenon of figure-ground segregation. Recent studies demonstrated that specific types of inhibitory neurons in the visual cortex contribute to surround suppression of V1 neurons. However, the functional roles of specific interneuron types in the feature specific surround suppression remains unclear. In this study, by presenting preferred orientation in the RF and varying the orientation or phase of the surround grating, we examined how surround suppression depends on the difference in orientation or phase between RF and surround for V1 neurons in mice. More importantly, we used optogenetics technique to manipulate the activities of somatostatin- or parvalbumin-expressing interneurons and examined the effect on orientation tuning and phase tuning of surround suppression. We found that surround suppression in mouse V1 is both orientation-dependent and phase-dependent, with stronger suppression for iso-orientation surround or same-phase surround. The degree of orientation-specific surround suppression(OSSI) and phase-specific surround suppression(PSSI) are correlated. Suppressing the activity of somatostatin-expressing interneurons significantly reduced the surround suppression, consistent with previous reports. Our preliminary data also showed that SOM suppression did not significantly change PSSI or OSSI in anesthetized mice. We are now performing experiments to examine how activating specific interneuron types influences the orientation- or phase-dependent surround suppression.

关键词： feature specific surround suppression primary visual cortex(V1) inhibitory neurons mouse

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Neuromorphic implementation of attractor dynamics in a decision circuit with NMDA receptors:a simulation study

Neuromorphic implementation of attractor dynamics in a decis...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Hongzhi You Key Laboratory for Neuro Information of Ministry of Education School of Life Science and TechnologyUniversity of Electronic Science and Technology of China

Decision making is an important cognitive function of selecting an option or an action from among a set of alternatives, and its time integration relies on the dynamics of NMDA receptors(NMDAR) and the saddle-node structure of the decision network. Recently, Federico and his collaborators(2015) achieved decision making and perceptual bistability in spike-based neuromorphic VLSI systems. However, corresponding decision performance can be improved further because they didn't take the dynamics of NMDARs into account. Here we present a novel method for the neuromorphic implementation of a decision circuit with NMDARs aimed at computing the attractor dynamics underlying the slow time integration of sensory evidences. Using the dynamic voltage-current circuit and multipliers, we applied the dynamical system approach of circuit synthesis to implement the NMDAR gating variable circuit, which is described by the first order kinetic equation of a reversible chemical reaction. Meanwhile, we developed a decision circuit with NMDARs according to the two-variable version of a biophysically realistic decision model proposed by Wong and Wang(2006). Circuit simulations in HSpice demonstrated that the NMDAR gating variable circuit can capture the nonlinear dynamical characteristics of corresponding gating variable, and the decision circuit with NMDARs can reproduce ramping neural activities observed in the electrophysiological recording in monkey's experiments. The build-up rate of these activities increases with the coherence of sensory inputs. Interestingly, the decision circuit can show working memory when the strong recurrent synaptic connections are ***, the theoretical analysis suggested that the dynamics of the decision in our neuromorphic circuit during the presentation of sensory evidences is determined by the saddle node structure. There is only one stable state for our neuromorphic circuit with weak recurrent connections after removing sensory evidences, but

关键词： Neuromorphic engineering decision making NMDA receptors attractor dynamics dynamical system approach

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MiR-181-regulates retinal cell fate by targeting Prox1 in Xenopus eye development

MiR-181-regulates retinal cell fate by targeting Prox1 in Xe...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Kaili Liu Zilong Li Ying Liu Xiumei Wang Rongqiao He The State Key Laboratory of Brain and Cognitive Science Institute of BiophysicsChinese Academy of Sciences

Retinal cell fate determination is modulated by both signaling molecules and transcriptional factors, however, the roles of micro RNAs(mi RNAs) in this network are far from clear. By gain- and loss-of-functional analysis, we show that mi R-181 plays important roles in regulating retinal cell fate during Xenopus development. Mi R-181 a and mi R-181 b, belong to the retina-specific mi R-181 cluster/family, we show that both members are initially expressed in developing retinoblast cells and then remain at a high level in the inner nuclear cell layer of mature retina. Loss of miR-181 a or mi R-181 b promotes the production of horizontal cells at the expense of bipolar cells. By screening the bipolar/horizontal cells-related genes predicted to be the targets of mi R-181, Prox1 expression was found to be negatively regulated by mi R-181 at both the transcripts and protein levels. In situ hybridization analysis shows that mi R-181 and Prox1 are expressed in an apparently complementary pattern within the retina, and the in vitro assay with Luciferase reporter system proves that mi R-181 can repress gene expression by directly targeting a conserved element in the 3'UTR of Prox1. Furthermore, the effect of mi R-181 downregulation on retinal cell fates can be compromised by coinjection of Prox1 si RNA. These results indicate that the Prox1-mi R-181 interaction plays important roles in retinal cell fate determination.

关键词： MiR-181,Prox1,bipolar cells,horizontal cells,Xenopus laevis

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PGE2 modulates GABAA receptors via an EP1 receptor-mediated signaling pathway

PGE2 modulates GABAA receptors via an EP1 receptor-mediated ...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Wen-Hao Dong Guang Yang Chang-Long Hu Yan-Ai Mei School of Life Sciences Institute of Brain Science and State Key Laboratory of Medical NeurobiologyFudan University

Objective PGE2 is one of the most abundant prostanoids in mammalian tissues, but its effect on neuronal receptors has not been well investigated. This study examines the effect of PGE2 on GABAA receptor currents in rat cerebellar granule neurons. Methods GABAA currents were recorded using a patch-clamp technique. Cell surface and total protein of GABAA ?1/2/3 subunits was carried out by Western blot analysis. Results Upon incubation of neurons with PGE2(1 ?M) for 60 minutes, GABAA currents were significantly potentiated. This PGE2-driven effect could be blocked by PKC or Ca MKII inhibitors as well as EP1 receptor antagonist, and mimicked by PMA or EP1 receptor agonist. Furthermore, Western blot data showed that PGE2 did not increase the total expression level of GABAA receptors, but significantly increased surface levels of GABAA ?1/2/3 subunits after 1 h of treatment. Consistently, both PKC and Ca MKII inhibitors were able to reduce PGE2-induced increases in cell surface expression of GABAA receptors. Conclusion Activation of either the PKC or Ca MKII pathways by EP1 receptors mediates the PGE2-induced increase in GABAA currents. This suggests that upregulation of postsynaptic GABAA receptors by PGE2 may have profound effects on cerebellar functioning under physiological and pathological conditions.

关键词： PGE2 GABAA receptors cerebellar granule cells EP receptor PKC

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The impact of cathepsin C on lipopolyccharide-induced neuroinflammation in mouse brain

The impact of cathepsin C on lipopolyccharide-induced neuroi...

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第六届亚太神经科学联合会学术会议暨中国神经科学学会第十一届全国学术会议

作者： Yan-Li Zhang Kai Fan Jian-Mei Ma Department of Anatomy Dalian Medical University

Cathepsin C(Cat C) is a lysosomal cystein protease which belongs to the papain super family. It has been found involved in inflammation process in peripheral system. Our previous study found that the expression and enzymatic activity of Cat C was upregulated in neuroinflammation which was induced by lipopolyccharide(LPS) intraperitoneal injection, but the impact of Cat C on neuroinflammation remains unclear. In this study, LPS(1 mg/kg) was used to inject intracerebroventricularly(i.c.v.) to induce neuroinflammation on wild type(WT), Cat C overexpression(OE), and knockdown(KD) mice, respectively. We found that Cat C and TNF-α,IL-1β expression was induced in the activated microglia following LPS injection at levels of protein and m RNA expression by immunohistochemisty, in situ hybridization, ELISA and realtime-PCR. Among three genotypes, the expression of TNF-αand IL-1βwas the highest in Cat C OE mice, and lowest in Cat C KD mice brain. We also stimulated primary microglia of different genotypes(WT, Cat C OE, Cat C KD) with LPS in vitro. It showed consistent results in primary microglia of different genotypes. In order to study the functional role of microglial Cat C in neuroinflammation, Cat C active monomer was used to stimulate BV2 microglia. We found that BV2 cells were characterized by large somata and TNF-αexpression was increased following Cat C treatment in a dose-dependent manner. The data indicate that Cat C overexpression could promote LPS-induced neuroinflammation by increasing release of proinflammatory cytokines, which might be associated with microglial polarization to M1 phenotype.

关键词： Cathepsin C microglia genotype LPS cytokine

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