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CD38gene-modified dendritic cells inhibit murine asthma development through increasing IL-12production and promoting Th1cell differentiation

CD38gene-modified dendritic cells inhibit murine asthma deve...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： jiaoliwang Department of Respiratory Medicine Nanjing Medical UniversityAffiliated Hangzhou Hospital(Hangzhou First People's Hospital)HangzhouZhejiang 310006

目的 Predominant Th2 cell and impaired Th1 cell polarization plays a critical role in the development of ***38 is related to the increased IL-12 release from dendritic cells(DCs)and Th1 cell polarization induced by *** CD38 expression will affect DC function in asthma development is still *** will construct CD38 adenovirus and transfect it into DC from asthmatic *** we will evaluate the Th1 polar ability of ***,we will also transfect this adenovirus into DC indeuced from mice bone marrow and confirm its therapeutic effect to *** changes of related signal molecules will also be explored.方法 In this study we overexpressed CD38 in DCs through infection of DCs with CD38 adenovirus(Ad-CD38)结果 We found DCs with CD38 overexpression alone showed no phenotype change but increased their maturation and secreted more IL-12 when synergized with *** stimulated CD38-overexpressing DCs were more effective on inducing Th1 cell *** received transfer of CD38-overexpressing DCs showed milder symptom of ***,we detected decreased IL-4,IL-5 and IL-13 in bronchoalveolar lavage fluid(BALF),IgE in sera and increased IFN-γ in BALF from recipients of CD38-overexpressing *** addition,we found more phosphorylated p38 in LPS stimulated CD38-overexpressing DCs and pretreatment with p38 specific inhibitor could abolish the priority of LPS stimulated CD38-overexpressing DCs to release IL-12 and induce Th1 cell differentiation.结论 Our results suggest that CD38 is involved in the DC function of alleviating asthma through rebuilding Th1/Th2 balance,thus providing a new idea for asthma therapy.

关键词： CD38 dendritic cells bronchial asthma Th1polarization adenovirus

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PIM2can induce asthma pathogenesis through IL-10

PIM2can induce asthma pathogenesis through IL-10

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： WeiDu GuochaoShi Ruijin Hospital Shanghai Jiao Tong University School of Medicine

目的 T cell associated inflammation especially Th2 type inflammation plays important role in asthma pathogenesis and PIM2,a member of serine/threonine kinase family,can promote growth and survival of T cells,and its mRNA level is significantly increased in asthma patients,but its role in asthma pathogenesis is still unclear,and our study is aimed to exlpore the effect of PIM2 on asthma pathogenesis.方法 BALB/cfemale mice sensitized and challenged by OVA were used as in vivo asthma model,and PIM2 inhibitor SMI-4a was injected into asthma mice during challenge period to observe the possible effect of *** asthma associated symptoms were documented during challenge,airway responsiveness、HE staining of lung tissue and serum level of cytokines including OVA specific IgE、IL-4、IL-5、IL-10、TGF-β1、IL-2 were collected 24 hours after the last challenge.结果 We've found that in asthma mice model inhibition of PIM2 can attenuate asthma symptom,airway responsiveness and airway inflammation compared with asthma mice model without inhibition of PIM2,and compared with normal mice,serum levels of OVA specific IgE、IL-4、IL-10 were significantly increased in asthma mice(OVA-IgE：1.26±0.14 and 1.56±0.2ng/ml,P＜0.01,IL-4：7.53±0.57 and 12.92±1.35pg/ml,P＜0.01,IL-10：8.91±1.62 and 41.47±5.89pg/ml,P＜0.001,respectively in normal and asthma mice),but only serum level of IL-10 was decreased after PIM2 inhibition compared with asthma mice(41.47±5.89 and 24.21±5.69pg/ml respectively in asthma mice without and with PIM2 inhibition,P＜0.001).结论 PIM2 plays important role in airway hyper-responsiveness,airway inflammation and can aggravate asthma symptoms in asthma mice *** role in asthma pathogenesis may be through increased expression level of IL-10.

关键词： PIM2 Asthma pathogenesis Inflammation IL-10

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Mitochondrial OGG1protects against PM2.5-induced oxidative DNA damage in BEAS-2B cells

Mitochondrial OGG1protects against PM2.5-induced oxidative D...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： GangLiu ChunyanLi LaweiYang ZiyingLin WenyaXu Affiliated Hospital of Guangdong Medical College

目的 PM2.5 is associated with higher mortality and increased hospital admissions in individuals with asthma and other respiratory *** enzyme 8-oxoguanine glycosylase 1(OGG1)has been shown to be involved in the repair of oxidative DNA ***,the effect of OGG1 on oxidative DNA damage caused by particulate matter 2.5(PM2.5)is unknown.方法 Human bronchial epithelial cell line(BEAS-2B)transfected with OGG1 overexpression vector or siRNA were exposed to the PM2.5(100μg/ml)for 24 *** DNA strand breaks were assessed by comet assay and Real-time quantitative PCR(RT-qPCR).Flow cytometry was used to test tetraethylbenzi-midazolylcarbocyanine iodide(JC-1)and The level of intracellular reactive oxygen species.结果 Herein,we demonstrated that OGG1 could inhibit the generation of ROS and alleviate mitochondrial dysfunction and increased the expression of IL-1β caused by *** dichlorodihydrofluorescein diacetate(DCFH-DA)staining and 5,5',6,6'-tetrachloro-1,1',3,3'-.tetraethylbenzi-midazolylcarbocyanine iodide(JC-1)staining using flow cytometry showed that PM2.5 induces the generation of ROS and leads to a reduction in mitochondrial membrane potential(MMP)in BEAS-2B *** of OGG1 inhibited the generation of ROS and the decline in *** of OGG1 by RNA interference(RNAi)increased the generation of ROS and reduced the ***-time quantitative PCR(RT-qPCR)for the mitochondrial DNA copy number(mtDNAcn)and flow cytometry for apoptosis revealed that OGG1 inhibits the apoptosis and decreases mtDNAcn induced by ***,the results of the comet assay showed that OGG1 had a significant repair effect on DNA strand breaks caused by *** of OGG1 also significantly suppressed the expression of IL-1β caused by ***,these data suggest that PM2.5 leads to mitochondrial dysfunction and the up-regulation of IL-1β could be reversed by overexpression of *** mitochondrial dysfunction caused by PM2.5 could be relieved by

关键词： OGG1 PM2.5 MMP ROS mtDNAcn

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The effect of a novel mask on efficacy and compliance during CPAP in OSAHS patients

The effect of a novel mask on efficacy and compliance during...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： YanZhang Respiratory Medicine of Xiangya Hospital Central South University

目的 The objective of this study was to determine the efficacy and compliance between a novel mask and nasal mask during automated continuous positive airway pressure (CPAP) in patients with obstructive sleep apnea hypopnea syndrome.方法 Twenty four subjects diagnosed by over-night polysomn- ography (PSG) in our outpatient (male= twenty-three) were randomized to undergo an in-laboratory automated CPAP titration with PSG via either a standard nasal mask or a novel mask at the first night, followed by using the alternative mask style at the second *** the auto-CPAP titration of 2 nights every participant chose their preferred mask style and voluntarily use their preferred mask or not to undergo auto-CPAP for 1 month at home.结果 These measurements included polysomnographic variables, downloaded data from CPAP machine, side-effect and mask preference, CPAP *** therapy through nasal mask or the novel mask can improve AHI, mean saturation, minimum saturation and sleep *** were no significant differences between nasal mask and the novel mask for polysomno- graphic variables of sleep architecture, including NREM1 (11.56±5.61％ VS 11.70±6.73％, p＞0.05), NREM2 (66.18±13.39％ VS 64.56±11.80％,p＞0.05), NREM3 (9.09±6.42％ VS 10.28±6.11％, p＞0.05), REM (13.14±7.10％ VS 13.50±7.41％, p＞0.05).There was no significant difference for the mean oxygen saturation (96.77±0.97％ VS 96.75±1.11％, p＞0.05), minimum oxygen saturation (90.50±2.99％ VS 89.58±3.48％, p＞0.05) and residual AHI(3.47±1.88/h, 3.43±1.76/h, p＞0.05) on Auto-CPAP between these 2 mask *** 95th CPAP pressure level was higher (14.75±2.68 cmH2O, 13.29±2.48 cmH2O, p＜0.05) but the 95th air leakage was lower(2.60±3.43 L/min, 6.84±6.40 L/min, p＜0.05) when using the novel mask than a nasal *** novel mask had less mouth dry but more ***, there is a good CPAP compliance in 1 month through nasal mask or novel mask and there was no significant difference between nasal mask and novel mas

关键词： OSAHS CPAP novel mask nasal mask efficacy and compliance

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Baicalin attenuates bleomycin-induced pulmonary fibrosis via adenosine A2a receptor related TGF-β1-induced ERK1/2 signaling pathway

Baicalin attenuates bleomycin-induced pulmonary fibrosis via...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： XiaoyingHUANG Yicheng He YanfanChen PeiliangWu MayunChen LiangxingWang Division of Pulmonary Medicine the First Affiliated Hospital of Wenzhou Medical University

目的 Baicalin has been reported to have anti-fibrosis effect;however, its mechanism still remains to be *** A2a receptor (A2aR) is a novel inflammation regulator, and transforming growth factor-β1 (TGF-β1)-induced extracellular signal regulated kinase1/2 (ERK1/2) signaling pathway plays an important role in idiopathic pulmonary fibrosis (IPF).This study was to explore the relationship of A2aR and TGF-β1-induced ERK1/2 in bleomycin (BLM)-induced pulmonary fibrosis in mice, and to investigate whether A2aR mediate the anti-fibrosis effect of Baicalin on BLM-induced pulmonary fibrosis.方法 The A2aR-/- and A2aR+/+ mice were respectively divided into three groups： control group, model group, baicalin *** fibrosis was induced in mice of model groups by intratracheal instillation of bleomycin,and baicalin was administered in mice of baicalin groups daily for 28 *** and ultrastructural changes of lung tissues were *** coefficient and the levels of hydroxyproline (HYP) in lung tissues were measured at the same *** levels of serum TGF-β1 were measured by *** expression of TGF-β1, ERK1/2,p-ERK1/2 and A2aR were detected by western blot and immunohistochemical staining techniques.结果 Severe lung fibrosis was observed in the bleomycin-treated mice on day *** histopathological findings and collagen content of lung tissues were much severer/higher in A2aR-/- mice than in A2aR+/+ *** also showed that TGF-β1 and p-ERK1/2 were upregulated in bleomycin-treated mice and expressed higher in A2aR-/- mice compared to A2aR+/+ ***, bleomycin-treated A2aR+/+ mice had increased A2aR level in ***, longterm treatment with baicalin in A2aR-/- and A2aR+/+ mice significantly ameliorated the histopathological changes in ***, Increased TGF-β1 and p-ERK1/2 expressions in bleomycin-treated A2aR-/- and A2aR+/+ mice were obviously diminished by *** baicalin-treated A2aR-/- mice had severer lung f

关键词： pulmonary fibrosis baicalin adenosine A2a receptor TGF-β1 extracellular signal regulated kinase1

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House dust mite allergens increases the expression of VEGF in human bronchiolar epithelium cell via PI3K pathway

House dust mite allergens increases the expression of VEGF i...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： xiangboShen Department of Respiratory CHANGSHA CENTRAL HOSPITAL

目的 To investigate Effect of Standardization of dust mite preparations(Alutard(R) SQ)on the Expression of Vascular Endothelial Growth Factor in Human bronchiolar epithelial cell(HBE).方法 Methyltetrazolium(MTT)assay was used to assess the HBE Cell viability under different concentration of dust *** used Real time-PCR to detected VEGF(165)/β-actin mRNA ratio under different concentrations of dust ***-linked immunosorbent assay(ELISA)was applied to detect the cell culture supernatant of VEGF protein expression.结果 The dust mite did not affect the Cell viability within the concentration of 1000U/*** 200U/L group,400U/L group,600U/L group,800U/L group,1000U/L group increased VEGF(165)gene expression compared to the control group;compared with other groups,the 400U/L group VEGF(165)gene expression was the highest;compared with 400U/L group the LY294002+400U/L group decreased VEGF(165)expression,and increased VEGF(165)gene expression compared with the control group;the LY294002 group showed no significant difference compared with the control *** ELISA result：The 200U/L group,400U/L group,600U/L group,800U/L group,1000U/L group,LY294002+400U/L group increased VEGF expression of cell culture supernatant compared with the control group;the VEGF protein in the supernatant of 400U/L group was most compared with other groups;the VEGF protein of LY294002+400U/L group decreased compared with 400U/L group,and compared with the control group the VEGF protein increased expression(P=0.024),the VEGF protein of LY294002 group showed no significant difference compared with the control group.结论 The Standardization of dust mite preparations(Alutard(R) SQ)increased the gene and protein expression of VEGF(165)in human bronchiolar epithelium cell(16-HBE)under certain concentrations,and can be inhibited by PI3K-specific inhibitor *** dust mite allergens increase 16-HBE VEGF expression via PI3K pathway.

关键词： asthma House dust mite VEGF PI3K

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Utility of A Type Ⅲ Portable Monitor for Diagnosis of OSA

Utility of A Type Ⅲ Portable Monitor for Diagnosis of OSA

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： LiyueXu BrendanKeenan AllanPack SamuelKuna ElizabethKneeland YuanChang XiaosongDong JingLi LongZhao PeiAn Xueli Zhang FangHan 北京大学国际医院 Penn Center for Sleep University of PennsylvaniaPhiladelphiaAmerica Department of Pulmonary Medicine Peking University People's Hos

目的 This study aims to compare NOX T3 study at-home to in-lab polysomnogram and simultaneous NOX T3 ***,we aim to compare the diagnostic accuracy of the NOX T3 portable sleep monitor to that of simultaneously recorded in-lab polysomnogram(PSG)in adults being evaluated for obstructive sleep apnea(OSA).方法 A total of 81 participants were recruited following face-to-face evaluation at PKU People's hospital's sleep *** participants initially performed an overnight HST using the Nox-T3 portable *** 2 weeks of the HST,participants performed an in-laboratory PSG with a simultaneous Nox-T3 portable monitor *** records were manually scored using the American Academy of Sleep Medicine(AASM)criteria,and PM records were double-scored using the device's autoscore algorithm as well as manual *** separate manually edited scorings were performed using different definitions for hypopnea in both HST and PSG,1)events with ≥ 30％reduction in airflow from baseline for ≥ 10 seconds accompanied by ≥ 4％oxygen desaturation,and 2)events with ≥ 30％reduction in airflow from baseline for ≥ 10 seconds associated with ≥ 3％reduction in oxygen saturation and/(or an arousal in PSG).The ability of subjects to perform HST was assessed as the percentage of successful HST on first and second recording attempts and HST quality.结果 The final sample consisted of 81 participants(63 males,18 females)with a mean±SD age,ESS,BMI,of 47.65±13.98,10.28±5.11,27.55±5.45Kg/*** rate for 1st HST and 2nd NOX study in lab was 93.75％(75/80),95％(76/80).No one failed in second time's home study when the first time of HST *** average apnea- hypopnea index ±SD(AHI 3％oxygen desaturation for hypopnea)in 1st NOX,2nd NOX and PSG was 28.54±20.35/h,32.54±22.39/h,33.51±23.2/*** score-derived T3 AHI(3％)and PSG-derived AHI(3％)were strongly related(r =.92).The T3(manual scored AHI)demonstrated a high degree of sensitivity for the presence of obstructive sleep apnea syndro

关键词： type Ⅲ Portable monitor Validation NOX T3 Home sleep test

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CB1receptors in the rat model of chronic intermittent hypoxia abnormal bone metabolism mechanism of action

CB1receptors in the rat model of chronic intermittent hypoxi...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： xiaolinggao The Second Hospital of Medical University

目的 To observe the endocannabinoid system(ECs)in cannabinoid receptor 1(CB1R)situation and trend after the intervention CB1R expressed in bone tissue,and by measuring the levels of bone-derived enzyme *** After using CB1R antagonist rimonabant intervention can alleviate bone metabolism and risk of abnormal bone structure in rats CIH.方法 Select 48 male SD rats,by establish the OSAS typical pathological model-chronic intermittent hypoxia(CIH)experimental animal models,according to the different methods of exposure and cycle times were randomly divided into six groups,namely 4,6W hypoxic group(IH),4,6W the control group(ON),4,6W hypoxic intervention group(HI),where the HI group were given intraperitoneal injection CB1R before modeling antagonist 1.5mg/kg/d were hypoxia *** the experiment of 4W,6W randomly selected half of the rats when the experiment was terminated,taken femoral vein 5ml,1h standing later,centrifuged the supernatant to determine the serum TRAP content by using ELASA method;then intercept the bone tissue of rats in each group with decalcification solution soak until softened tissue,after paraffin embedding machine slicer cut in a uniform thickness of bone tissue placed on a glass slide,HE staining in light microscopy whether the change in bone microarchitecture;observe the expression of bone tissue CB1R using immunohistochemistry and determination.结果 *** determination：After modeling,4W and 6W-IH group were higher than TRAP modeling before 4,6W-ON group,there was difference(t =-7.930,P ＜0.001),and Comparison between ON TRAP activity level group,the difference was not statistically significant(t =-1.273,P = 0.224);after the intervention,4W and 6W-HI group TRAP levels were lower than before the intervention 4,6W-IH group,there are statistical difference(t =-7.930,P ＜0.001),these results prompted increased after hypoxia time TRAP levels can be elevated above normal,but after the intervention,TRAP levels can interfere with the extension

关键词： obstructive sleep apnea syndrome(OSAS) cannabinoid receptor system1(CB1R)

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The protective role of overexpressing MUC5AC in MHV-68 induced combined pulmonary fibrosis and emphysema mouse

The protective role of overexpressing MUC5AC in MHV-68 induc...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： LiHe Jingzhou Central Hospital

目的 Mucins have long been regarded as exerting a role as a barrier to prevent mucosal infection but some data supporting overexpressed mucins induced airways obstruction and *** wonder if increased MUC5AC secretion provided benefit against murine gammaherpesvirus 68 (MHV-68) and MHV-68 induced pulmonary ***, we develop a combined pulmonary fibrosis and emphysema (CPFE) murine model by tracheal instillation of MHV-68 in long time cigarette smokeinduced old male emphysema ***, an MUC5AC-overexpressing mouse model will be generated to understand whether increased MUC5AC secretion add protection against MHV-68 in CPFE murine model or induce airways obstruction and inflammation.方法 Firstly, Experimental mice were randomly divided into three groups： group A (normal control group, n=6), group B (emphysema group, passive cigarette smoking for 1 hour twice everyday for 13 months, 0.1 ml PBS by tracheal instillation on day 361, n=6), group C (emphysema + MHV-68 group, 1×105 plaque forming units of MHV-68 in 0.1 ml PBS by tracheal instillation on day 361, n=24), and the group C were further divided into four groups： group C1 (sacrificed on day 367, on day 7 after MHV-68 tracheal instillation, n=6);group C2 (sacrificed on day 374, on day 14 after MHV-68 tracheal instillation, n=6);group C3 (sacrificed on day 381, on day 21 after MHV-68 tracheal instillation, n=6);group C4 (sacrificed on day 388, on day 28 after MHV-68 tracheal instillation, n=6).Group A and B were sacrificed on day *** bronchoalveolar lavage fluid (BALF) and lung specimens were collected. Secondly, another group of experimental mice were randomly divided into five groups： normal control group;pcDNA3.1 control group;pcDNA3.1-MUC5AC group;CPFE group;pcDNA3.1-MUC5AC+CPFE *** analysis of each group was investigated by hematoxylin and eosin staining, and masson trichrome ***-68 gp150 mRNA was detected by real-time ***5AC levels in lung tissues were analyze

关键词： MUC5AC MHV-68 combined pulmonary fibrosis and emphysema

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NoninvasivePositivePressureVentilationtoRelieveDyspneaof COPDpatientsduringExercisetraining:Aprospectivestudy

NoninvasivePositivePressureVentilationtoRelieveDyspnea...

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2016中华医学会呼吸病学年会暨第十七次全国呼吸病学学术会议

作者： Boxue Han Suiyang Zhang Thesecondartillerygeneralhospital

目的 TodeterminewhethernoninvasiveventilationsupportduringexerciseofaprograminthestableCOPDpatientscouldenabletheindividualstoreleasedyspneaandincreaseactivitytolerance.方法 Arandomized,self-controlledandprospectiveobservationalstudy,20participantswiththeseverebutstableCOPD(FEV1/pre：45.6±2.6％)were***subjectcompletedfivekindsoftests,onthewaytodoingtreadmillwalkingexerciseasquicklyaspossible,foraperiodof16days.5kindsoftestswereincluded：Test0：baselinewithoutventilator***Test1andTest2wererequiredwiththeventilatorsupport(NIPPVvianozzle)inSmode(spontaneousmode)whilehavingarest：twodifferentpressurelevels(IPAP/EPAP：18/8cmH2Oand14/4cmH2O)wereinTest1andTest2***3andTest4：bothexerciseandrestwereaidedwithventilator(NIPPVviatotalfacemask),SmodewasappliedinTest3,besides,AVAPS-AE(averagevolumeassuredpressuresupportauto-EPAP,AVAPS-AE)modewasadoptedinTest4(parameters：maximumEPAP=8cmH2O,minimumEPAP=4cmH2O,Rate=3cmH2O/min,tidalvolumedependingonindividualBMI).Durationofthewalk,distancewalkedandrecoverytimewererecordedin***theexercisesessions,heartrate,respiratoryrate,pulseoxygensaturationandBorgscoreatdifferenttimewhenT1,T2,T3(the1st,3rdand5thminuteduringwalking)andT4,T5,T6(the1st,2ndand3rdminuteduringresting)weremonitored***measuresabovewerestatisticallyanalyzed.结果 ***withthetest0,thedurationofrecoveryinTest1,Test3andTest4weresignificantlyshorter(p＜0.05).Nosignificantchangesofdistanceanddutationofthewalkwereobservedintest1,2,3,4(p＞0.05).***,SpO2(％),HRandBorgscoreatT5inTest1,Test3andTest4werelowerthantest0(p＜0.05)andthesemeasuresatT2inTest4weremorelower(p＜0.05).ComparedwithTest0,SpO2,RR,HRandBorgscoreinotherfourtestsdidnotchangegreatly(p＞0.05).结论 Noni

关键词： NoninvasiveVentilation COPD PulmonaryRehabilitation

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