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The impact of digital finance on regional economic resilience

PACIFIC-BASIN FINANCE JOURNAL 2024年 85卷

作者： Yang, Yang lin, Zibo Xu, Zhaoyi Liu, shuwen Hunan Normal Univ Business Sch Changsha 410081 Peoples R China Tsinghua Univ Sch Econ & Management Beijing 100062 Peoples R China Hunan Univ Technol & Business Coll Accounting Changsha 410205 Peoples R China

This paper introduces an innovative Urban Economic Resilience Index and uses data from 276 cities in the period from 2011 to 2021 to investigate the impact of digital finance on economic resilience. The empirical results reveal that digital finance significantly improves economic resilience and has a pronounced effect on various dimensions of resilience such as resistance and resilience ability, adaption and adjustment ability, and innovation and transformation ability. Furthermore, these findings demonstrate the robustness including the incorporation of a diverse sample of municipalities, alternative measures for digital finance and economic resilience, the inclusion of supplementary control variables, and the application of the instrumental variable methodology. Furthermore, digital finance improves economic resilience by injecting vitality into innovation and entrepreneurship, accelerating industrial structure upgrading and enhancing financial efficiency. Analyses also show that compared to eastern regions and regions with high levels of network coverage as well as economic development, digital finance is more likely to improve economic resilience. Overall, this study provides new insights into digital finance and economic resilience.

关键词： Digital finance Economic resilience Innovation and entrepreneurship Industrial structure upgrading Financial efficiency

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腹腔镜尾侧入路法和中间入路法治疗右半结肠癌的效果比较

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中国医药科学 2020年第24期10卷 194-197页

作者：洪晓城林树文叶伟杰陈家阳吉成岗广东省东莞市滨海湾中心医院普通外科广东东莞523900

目的探究腹腔镜尾侧入路法和中间入路法在治疗右半结肠癌中的效果差异。方法选取本院2016年7月~2018年7月收治的右半结肠癌患者60例,采用随机数字表法分为侧入组30例和中间组30例。侧入组采用腹腔镜尾侧入路手术法治疗,中间组采用中间... 详细信息

目的探究腹腔镜尾侧入路法和中间入路法在治疗右半结肠癌中的效果差异。方法选取本院2016年7月~2018年7月收治的右半结肠癌患者60例,采用随机数字表法分为侧入组30例和中间组30例。侧入组采用腹腔镜尾侧入路手术法治疗,中间组采用中间入路法手术治疗。所有患者随访1年。比较两组患者围术期指标,手术前后血清癌胚抗原(CEA)和肿瘤特异性生长因子(TSGF)水平和术后1年并发症发生率。结果(1)侧入组患者术中出血量、手术时间、住院时间均显著低于中间组,差异有统计学意义(P<0.05)。(2)两组患者CEA、TSGF水平均显著低于术前,差异有统计学意义(P<0.05);而术后组间比较,差异无统计学意义(P>0.05)。(3)侧入组患者术后1年并发症发生率显著低于中间组,差异有统计学意义(P<0.05)。结论腹腔镜尾侧入路法治疗右半结肠癌,术中出血量少、手术时间和住院时间短,术后并发症的发生率低且不影响肿瘤切除效果,较中间路入疗法更有优势。

关键词：腹腔镜尾侧入路中间路入右半结肠癌

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Tenghuang Jiangu tablet in knee osteoarthritis therapy: A prospective multicenter registry study in China

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JOURNAL OF ETHNOPHARMACOLOGY 2025年 345卷 119525页

作者： Zheng, Zelu Wang, Xiaohan Mi, Baohong Zhou, Jun Yan, Yan Li, shuwen Luo, Yuxin Tang, Kaiqiang Dong, Yawei Quan, Rui lin, Jiaming Zhang, Jiawen Liu, Jiachun Shi, Yuhang Wang, Rongtian Zhang, Yanqiong lin, Na Weng, Xisheng Chen, Weiheng Beijing Univ Chinese Med Affiliated Hosp 3 Dept Mininvas Joint Surg Beijing 100029 Peoples R China Minist Educ Engn Res Ctr Chinese Orthopaed & Sports Rehabil Ar Beijing 100029 Peoples R China China Acad Chinese Med Sci Inst Chinese Mat Med Beijing 100700 Peoples R China Chinese Acad Med Sci & Peking Union Med Coll Peking Union Med Coll Hosp Dept Orthoped Surg State Key Lab Complex Severe & Rare Dis Beijing 100730 Peoples R China

Ethnopharmacological relevance: Chinese patent medicine Tenghuang Jiangu tablet (THJGT) is frequently used to treat knee osteoarthritis (KOA). Aim: This prospective multicenter registry study investigated the effectiveness and safety of THJGT in treating KOA. Materials and methods: Patients with KOA aged 50-75 years were preferentially treated with THJGT, other nonsurgical conventional treatments (CTs), or both. The treatment duration was 8 weeks, with follow-ups at weeks 4 and 8. The visual analog scale (VAS) and Western Ontario and McMaster Universities Osteoarthritis Index (WOMAC) scores were the efficacy assessments. Adverse events and drug reactions were evaluated to assess the safety of THJGT. Propensity score matching (PSM) was used for the subgroup analysis. Results: From September 2019 to January 2021, a total of 2995 participants were included, with 1471 in THJGT group, 490 in CT group and 1034 in THJGT + CT group. After treatment, the VAS and WOMAC scores in the three groups improved significantly (P < 0.001). At week 8, the VAS scores in the THJGT group significantly improved, were lower than those in the CT group (P < 0.01), and were comparable to those in the THJGT + CT group (P = 0.623). The WOMAC scores significantly improved (P < 0.001), with no differences between groups (P > 0.05). The WOMAC pain and stiffness scores were better in the THJGT + CT than in the THJGT and CT groups. PSM revealed that the WOMAC pain and stiffness scores were significantly lower in the THJGT than in the nonsteroidal anti-inflammatory drugs (NSAID) group (P < 0.01 and 0.001). Adverse events were higher in the CT and THJGT + CT groups (P < 0.001). Conclusion: THJGT is effective and safe for treating KOA, particularly in improving stiffness symptoms.

关键词： Chinese patent medicine Knee osteoarthritis Tenghuang Jiangu tablet Clinical effectiveness Propensity score matching Registry study

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The mitochondria-targeted antioxidant MitoQ ameliorates inorganic arsenic-induced DCs/Th1/Th2/Th17/Treg differentiation partially by activating PINK1-mediated mitophagy in murine liver

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ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY 2024年 277卷 116350页

作者： Li, Hui Guo, Yaning Su, Wei Zhang, Huan Wei, Xiaoxi Ma, Xinyu Gong, shuwen Qu, Gaoyang Zhang, lin Xu, Hong Shen, Fuhai Jiang, Shoufang Xu, Dingjie Li, Jinlong North China Univ Sci & Technol Sch Publ Hlth Hebei Key Lab Organ Fibrosis Res Tangshan 063210 Hebei Peoples R China Wannan Med Coll 22 Wenchang West RdHigher Educ Pk Wuhu 241000 Anhui Peoples R China North China Univ Sci & Technol Coll Tradit Chinese Med Tangshan 063210 Hebei Peoples R China

Inorganic arsenic is a well-established environmental toxicant linked to acute liver injury, fibrosis, and cancer. While oxidative stress, pyroptosis, and ferroptosis are known contributors, the role of PTEN-induced kinase 1 (PINK1)-mediated mitophagy in arsenic-induced hepatic immunotoxicity remains underexplored. Our study revealed that acute arsenic exposure prompts differentiation of hepatic dendritic cells (DCs) and T helper (Th) 1, Th2, Th17, and regulatory T (Treg) cells, alongside increased transcription factors and cytokines. Inorganic arsenic triggered liver redox imbalance, leading to elevated alanine transaminase (ALT), hydrogen peroxide (H 2 O 2 ), malondialdehyde (MDA), and activation of nuclear factor erythroid 2-related factor (Nrf2)/heme oxygenase-1 (HO -1) pathway. PINK1-mediated mitophagy was initiated, and its inhibition exacerbates H 2 O 2 accumulation while promoting DCs/Th1/Th2/Treg differentiation in the liver of arsenic-exposed mice. Mitoquinone (MitoQ) pretreatment relieved arsenic-induced acute liver injury and immune imbalance by activating Nrf2/HO-1 and PINK1-mediated mitophagy. To our knowledge, this is the first report identifying PINK1mediated mitophagy as a protective factor against inorganic arsenic-induced hepatic DCs/Th1/Th2 differentiation. This study has provided new insights on the immunotoxicity of inorganic arsenic and established a foundation for exploring preventive and therapeutic strategies targeting PINK1-mediated mitophagy in acute liver injury. Consequently, the application of mitochondrial antioxidant MitoQ may offer a promising treatment for the metalloid-induced acute liver injury.

关键词： Arsenic Immunotoxicity Liver PINK1 Mitophagy Mitoquinone

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A Facile Way To Improve the Bioavailability of Nanomedicine Based on the Threshold Theory

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MOLECULAR PHARMACEUTICS 2022年第5期19卷 1647-1655页

作者： Zhang, Huihui Cui, Pengfei Gao, Zihan Zhou, shuwen Wang, Cheng Jiang, Pengju Ni, Xinye Wang, Jianhao Qiu, lin Chanchou Univ Sch Pharm Changzhou 213164 Peoples R China Nanjing Med Univ Affiliated Changzhou Peoples Hosp 2 Changzhou 213003 Peoples R China

One of the most significant barriers to the clinical trans-formation of nanomedicines is low drug distribution in solid tumors due toquick clearance of nanomedicine after injection. Studies have revealed thatthe distribution of nanomedicine in tumor sites can be considerably improvedwhen the number of nanoparticles supplied in a short period surpasses thethreshold. Most routinely employed nanomaterials have dose-related safetyconcerns. To resolve this problem, we use highly biocompatible albumin toconstruct blank nanoparticles and doxorubicin loading nanoparticles. Underthe guidance of the threshold theory, when the quantity of drug loadingnanoparticles is constant, the drug delivery effectiveness improves with theaddition of blank nanoparticles. This enhanced impact was verified both invitro and in vivo. The area under the curve of the high dose group (19.5x1011) is 2.5 times higher than that of the low dose group(6.5x1011). In addition, the drug distribution of the high dose group at the tumor site was also improved by 1.5 times comparedwith the low dose group. The results of histopathological sections also showed that the administration of excess blank nanoparticleswithin 24 h has no damage to the animals. This study contributes to the clinical transition of nanomedicine by providing fresh ideas for anticancer nanomedicine research

关键词： nanomedicine bioavailability threshold theory

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Progress in Intradermal and Transdermal Gene Therapy with Microneedles

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PHARMACEUTICAL RESEARCH 2022年第10期39卷 2475-2486页

作者： Zhu, Ting Zhang, Wenya Jiang, Pengju Zhou, shuwen Wang, Cheng Qiu, lin Shi, Honglei Cui, Pengfei Wang, Jianhao Changzhou Univ Sch Pharm Changzhou 213164 Peoples R China Jiangsu Univ Wujin Hosp Changzhou 213017 Jiangsu Peoples R China Xuzhou Med Univ Wujin Clin Coll Changzhou 213017 Jiangsu Peoples R China

Gene therapy is one of the most widely studied treatments and has the potential to treat a variety of intractable diseases. The skin's limited permeability, as the body's initial protective barrier, drastically inhibits the delivery effect of gene medicine. Given the potential adverse effects and physicochemical features of the medications, improving generic drug penetration into the skin barrier and achieving an effective level of target tissues remains a challenge. Microneedles have made tremendous improvements in aided gene transfer and medication delivery as a unique method. Microneedles offer the advantage of being minimally invasive and painless, as well as the ability to distribute gene medicines straight through the stratum corneum. Microneedles have been used to penetrate skin tissue with various nucleic acids and medicines in recent years, allowing for a wide range of applications in the treatment of skin ailments. This review focuses on skin-related disorders and immunity, and it primarily discusses the progress of microneedle transdermal gene therapy in recent years. It also complements the current major vectors and related microneedle gene therapy applications.

关键词： gene delivery gene therapy microneedles transdermal delivery

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Identification and validation of key biomarkers and potential therapeutic targets for primary open-angle glaucoma

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Science China(Life Sciences) 2023年第12期66卷 2837-2850页

作者： Jian Wu Caixia lin Chenlong Yang Lijie Pan Hongyi Liu Sirui Zhu shuwen Wei Xu Jia Qi Zhang Ziyu Yu Xiaofang Zhao Weihai Liu Yehong Zhuo Ningli Wang Beijing Institute of Ophthalmology Beijing Tongren Eye CenterBeijing Tongren HospitalCapital Medical UniversityBeijing Key Laboratory of Ophthalmology and Visual SciencesBeijing 100730China Beijing Tongren Eye Center Beijing Tongren HospitalCapital Medical UniversityBeijing 100730China Department of Neurosurgery Peking University Third HospitalCenter for Precision Neurosurgery and Oncology of Peking University Health Science CenterBeijing 100191China North America Medical Education Foundation Union City CA 94539USA State Key Laboratory of Ophthalmology Zhongshan Ophthalmic CenterSun Yat-sen UniversityGuangzhou 510060China Spencer Center for Vision Research Byers Eye InstituteSchool of MedicineStanford UniversityPalo Alto CA 94304USA

Primary open-angle glaucoma(POAG)is a prevalent cause of blindness worldwide,resulting in degeneration of retinal ganglion cells and permanent damage to the optic ***,the underlying pathogenetic mechanisms of POAG are currently indistinct,and there has been no effective nonsurgical treatment *** objective of this study is to identify novel biomarkers and potential therapeutic targets for *** mRNA expression microarray datasets GSE27276 and GSE138125,as well as the single-cell high-throughput RNA sequencing(sc RNA-seq)dataset GSE148371 were utilized to screen POAGrelated differentially expressed genes(DEGs).Functional enrichment analyses,protein-protein interaction(PPI)analysis,and weighted gene co-expression network analysis(WGCNA)of the DEGs were ***,the hub genes were validated at a single-cell level,where trabecular cells were annotated,and the mRNA expression levels of target genes in different cell clusters were *** and quantitative real-time PCR(q PCR)were performed for further *** analysis identified 43 downregulated and 32 upregulated genes in POAG,which were mainly enriched in immune-related pathways,oxidative stress,and endoplasmic reticulum(ER)*** networks showed that FN1 and DUSP1 were the central hub nodes,while GPX3 and VAV3 were screened out as hub genes through WGCNA and subsequently validated by q ***,FN1,GPX3,and VAV3 were determined to be pivotal core genes via single-cell *** relevant biomarkers involved in the pathogenesis of POAG,may serve as potential therapeutic *** studies are necessary to unveil the mechanisms underlying the expression variations of these genes in POAG.

关键词： primary open-angle glaucoma biomarkers immune response oxidative stress endoplasmic reticulum stress

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Gut Microbiota, a Potential Mediated Target for Reducing Geniposide Hepatotoxicity by Interacting with Isoflavones

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ENGINEERING 2025年第4期47卷 222-235页

作者： Yang, Wen Zhang, Wen Huang, Xinhui Geng, shuwen Zhai, Yujia Jiang, Yuetong Tian, Tian Gao, Yuye He, Jing Huang, Taohong Li, Yunxia Zhang, Wenjing Wen, Jun Wu, Jian-lin Wang, Guangji Zhou, Tingting Naval Med Univ Sch Pharm Dept Pharmaceut Anal Shanghai 200433 Peoples R China Naval Med Univ Shanghai Key Lab Pharmaceut Metabolite Res Shanghai 200433 Peoples R China Naval Med Univ Naval Med Ctr Shanghai 200433 Peoples R China Shimadzu China Co Ltd Shanghai 200233 Peoples R China Tongji Univ Tongji Hosp Sch Med Dept Neurol Shanghai 200092 Peoples R China Shanghai Univ Tradit Chinese Med Shanghai Municipal Hosp Tradit Chinese Med Dept Psychiat Shanghai 200071 Peoples R China Macau Univ Sci & Technol State Key Lab Qual Res Chinese Med Macau 999078 Peoples R China China Pharmaceut Univ State Key Lab Nat Med Key Lab Drug Metab & Pharmacokinet Nanjing 210009 Peoples R China

Geniposide, the principal active iridoid glucoside ingredient in Fructus gardeniae used in numerous traditional Chinese clinical prescriptions, has been shown to cause herbal hepatotoxicity because of its glycone metabolite genipin. This study explored the role of gut microbiota in alleviating geniposide hepatotoxicity with isoflavones in soy products. Metabolic profiling using ultra high-performance liquid chromatography-quadrupole time-of-flight mass spectrometry (UHPLC-Q/TOF-MS) revealed two metabolic pathways and six main forms of geniposides in vivo. Enzyme inhibitor experiments have shown that isoflavones alter geniposide metabolism by mediating specific enzymes, including (3-glucosidase ((3-GC) and sulfotransferase (SULT), in an established pseudo-sterile rat model. Isoflavones pretreatment by gavage for three weeks optimized the structure of the gut microbiota was linked to the regulation of key metabolic enzymes. Furthermore, experiments involving fecal microbiota transplantation (FMT) established the direct contribution of the gut microbiota to the regulation of enzyme activities and geniposide metabolism. This study demonstrated that isoflavones in soy products regulated the metabolic enzymes of geniposode dependent on gut microbiota, especially Lactobacillus spp., which was further verified in our clinical trials analyzed using 16S ribosomal RNA (rRNA) and metagenomic sequencing, thus regulating geniposide metabolism. Furthermore, as dominant beneficial bacterium, Lactobacillus spp. were discovered to be promising microbial targets for the better management of geniposide hepatotoxicity. These findings provide valuable insights for the prevention and intervention of drug-induced liver injury. (c) 2025 THE AUTHORS. Published by Elsevier LTD on behalf of Chinese Academy of Engineering and Higher Education Press Limited Company. This is an open access article under the CC BY-NC-ND license (http://***/licenses/by-nc-nd/4.0/).

关键词： Geniposide Isoflavones rich diet Gut microbiota Fecal microbiota transplantation Intestinal metabolic enzymes Drug-induced liver injury

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Tauroursodeoxycholic acid ameliorates renal injury induced by COL4A3 mutation

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KIDNEY INTERNATIONAL 2024年第3期106卷 433-449页

作者： Yu, shuwen Gu, Xiangchen Zheng, Qimin Liu, Yunzi Suhas, Teija Du, Wen Xie, lin Fang, Zhengying Zhao, Yafei Yang, Mingxin Xu, Jing Wang, Yimei lin, Meei-Hua Pan, Xiaoxia Miner, Jeffrey H. Jin, Yuanmeng Xie, Jingyuan Shanghai Jiao Tong Univ Ruijin Hosp Sch Med Dept Nephrol Shanghai Peoples R China Shanghai Jiao Tong Univ Inst Nephrol Sch Med Shanghai Peoples R China Shanghai Univ Tradit Chinese Med Yueyang Hosp Integrated Tradit Chinese & Western M Dept Nephrol Shanghai Peoples R China Washington Univ Sch Med Dept Med Div Nephrol St Louis MO USA Washington Univ Sch Med Dept Cell Biol & Physiol St Louis MO USA

COL4A3/A4/A5 mutations have been identified as critical causes of Alport syndrome and other genetic chronic kidney diseases. However, the underlying pathogenesis remains unclear, and specific treatments are lacking. Here, we constructed a transgenic Alport syndrome mouse model by generating a mutation (Col4a3 p.G799R) identified previously from one large Alport syndrome family into mice. We observed that the mutation caused a pathological decrease in intracellular and secreted collagen IV alpha 3 alpha 4 alpha 5 heterotrimers. The mutant collagen IV alpha 3 chains abnormally accumulated in the endoplasmic reticulum and exhibited defective secretion, leading to persistent endoplasmic reticulum stress in vivo and in vitro. RNA-seq analysis revealed that the MyD88/p38 MAPK pathway plays key roles in mediating subsequent inflammation and apoptosis signaling activation. Treatment with tauroursodeoxycholic acid, a chemical chaperone drug that functions as an endoplasmic reticulum stress inhibitor, effectively suppressed endoplasmic reticulum stress, promoted secretion of the alpha 3 chains, and inhibited the activation of the MyD88/p38 MAPK pathway. Tauroursodeoxycholic acid treatment significantly improved kidney function in vivo. These results partly clarified the pathogenesis of kidney injuries associated with Alport syndrome, especially in glomeruli, and suggested that tauroursodeoxycholic acid might be useful for the early clinical treatment of Alport syndrome.

关键词： Alport syndrome apoptosis endoplasmic reticulum fibrosis inflammation

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A novel rat model for cerebral venous sinus thrombosis: verification of similarity to human disease via clinical analysis and experimental validation

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JOURNAL OF TRANSLATIONAL MEDICINE 2022年第1期20卷 174-174页

作者： Mu, shuwen lin, Yinghong Xu, Yongjun Wei, Xianqing Zeng, Zihuan lin, Kunzhe Zhu, linghua Liu, Qinghong Qi, Xingfeng Wei, Liangfeng Liang, Shengxiang Wang, Shousen Fujian Med Univ Dept Neurosurg Fuzong Clin Med Coll Fuzhou 350025 Peoples R China Fujian Univ Tradit Chinese Med Fuzhou 350122 Peoples R China Fujian Med Univ Hosp 900 Lab Basic Med Fuzong Clin Med Coll Fuzhou 350025 Peoples R China Shenyang Univ Technol Coll Mat Sci & Engn Shenyang 110870 Peoples R China Fujian Med Univ Hosp 900 Dept Neurosurg Fuzong Clin Med Coll Fuzhou 350025 Fujian Peoples R China Fujian Med Univ Dept Neurosurg Affiliated Fuzhou Hosp 1 Fuzhou 350009 Peoples R China Fujian Med Univ Hosp 900 Dept Pathol Fuzong Clin Med Coll Fuzhou 350025 Peoples R China Fujian Univ Tradit Chinese Med Natl Local Joint Engn Res Ctr Rehabil Med Technol Fuzhou 350122 Fujian Peoples R China Fujian Univ Tradit Chinese Med Rehabil Ind Inst Fuzhou 350122 Peoples R China

Background Cerebral venous sinus thrombosis (CVST) is a rare neurovascular disorder with highly variable manifestations and clinical courses. Animal models properly matched to the clinical form of CVST are necessary for elucidating the pathophysiology of the disease. In this study, we aimed to establish a rat model that accurately recapitulates the clinical features of CVST in human patients. Methods This study consisted of a clinical analysis and animal experiments. Clinical data for two centres obtained between January 2016 and May 2021 were collected and analysed retrospectively. In addition, a Sprague-Dawley rat model of CVST was established by inserting a water-swellable rubber device into the superior sagittal sinus, following which imaging, histological, haematological, and behavioural tests were used to investigate pathophysiological changes. Principal component analysis and hierarchical clustering heatmaps were used to evaluate the similarity between the animal models and human patients. Results The imaging results revealed the possibility of vasogenic oedema in animal models. Haematological analysis indicated an inflammatory and hypercoagulable state. These findings were mostly matched with the retrospective clinical data. Pathological and serological tests further revealed brain parenchymal damage related to CVST in animal models. Conclusions We successfully established a stable and reproducible rat model of CVST. The high similarity between clinical patients and animal models was verified via cluster analysis. This model may be useful for the study of CVST pathophysiology and potential therapies.

关键词： Cerebral venous sinus thrombosis Animal model Pathophysiology Vasogenic oedema

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