检索结果-内蒙古大学图书馆

Characteristics of global cerebral ischemia models constructed by modified four-vessel occlusion in rats

Neural Regeneration Research 2006年第5期1卷 436-440页

作者： Jinbao Li Lai Jiang Hua Xu Yuanchang Xiong Xiaoming Deng Department of Anesthesiology Changhai Hospital Second Military Medical University of ChinesePLA Shanghai 200433 China

BACKGROUND： Pulsinelli et al developed a kind of rat models with four-vessel occlusion-induced global cerebral ischemia, Because the histo-pathological changes and severe cerebral ischemia reproducibility of this model are good and the stability of this model in circulation respiration is superior to that of other models, so four-vessel occlusion method has become a classic modeling method for global cerebral ischemia model, This model has been improved in some laboratories to meet different requirements in different studies. OBJECTIVE： To establish a highly reproducible rat model of reversible forebrain ischemia by modifying four-vessel occlusion model introduced by Pulsinelli et al, and to investigate its neurophysiological and pathological changes and the characteristics of modified operation. DESIGN ： Completely randomized grouping, controlled tria. SETTING ： Department of Anesthesiology,Changhai Hospital, Second Military Medical University of Chinese PLA. MATERIALS ： A total of 65 male healthy SD rats, weighing 250-300 g, were provided by the Experimental Animal Center of Second Military Medical University of Chinese PLA. VSM hemodynamic monitor and temperature monitor （Thermal ert TH-5, U.S.A） were used. METHODS ： The trial was conducted in the Department of Anesthesiology, Changhai Hospital, Second Military Medical University of Chinese PLA from January 2005 to March 2006. ① Experimental grouping： Sixty-five rats were randomly divided into the following 7 groups： sham-operation group （n =9）： given the same operation, without occlusion of vessels; 5 minutes ischemia group （n =9）： ischemia 5 minutes and reperfusion 72 hours; 10 minutes ischemia group （n =8）： ischemia 10 minutes and reperfusion 72 hours; 15 minutes ischemia group （n =9）： ischemia 15 minutes and reperfusion 72 hours; 20 minutes ischemia group （n =8）： ischemia 20 minutes and reperfusion 72 hours; 30 minutes ischemia group （n =7）; ischemia 30 minutes and reperfusion 72 hours; ischemia con

关键词：疾病模型动物脑缺血/病理生理学脑缺血/病理学血压再灌注损伤/病理生理学大鼠动物男(雄)性

来源：评论

学校读者我要写书评

暂无评论

氯沙坦、卡托普利联合对兔心肌缺血再灌注诱导内皮细胞损伤的影响

引用

陕西医学杂志 2008年第4期37卷 407-410页

作者：郭潇王莉高雁甘肃省兰州市兰西医院心肾内科兰州730050

目的:研究氯沙坦联合卡托普利对兔心肌缺血再灌注损伤内皮细胞的保护作用及其机制。方法:结扎兔左冠状动脉前降支制作心肌缺血再灌注损伤模型,随机分为空白对照组、缺血再灌注组和联合组。联合组在心肌缺血前10min给予氯沙坦和卡托普利... 详细信息

目的:研究氯沙坦联合卡托普利对兔心肌缺血再灌注损伤内皮细胞的保护作用及其机制。方法:结扎兔左冠状动脉前降支制作心肌缺血再灌注损伤模型,随机分为空白对照组、缺血再灌注组和联合组。联合组在心肌缺血前10min给予氯沙坦和卡托普利。观察各组缺血再灌注过程中心电图的动态改变,以及氯沙坦和卡托普利联合对血管性血友病因子及丙二醛和血清一氧化氮含量、损伤兔血浆及心肌组织血管紧张素的水平的影响。结果:缺血再灌注组和联合组均造成明显的心电图动态改变,与空白对照组比较,联合组心电图ST段出现有效改变。缺血再灌注组和联合组血浆和心肌组织血管紧张素水平呈时间依赖性升高,血管性血友病因子活性和丙二醛水平较缺血再灌注组显著降低,而血清一氧化氮水平明显提高。结论:氯沙坦和卡托普利的联合使用对心肌缺血再灌注损伤内皮细胞有明显的保护作用,其作用与多层面的清除氧自由基的膜脂质过氧化及拮抗血管紧张素受体AT1有关。

关键词：心肌缺血/并发症再灌注损伤/病理生理学细胞/损伤 @氯沙坦卡托普利

来源：评论

学校读者我要写书评

暂无评论

高血压对大鼠脑缺血再灌注后细胞凋亡的影响

引用

陕西医学杂志 2008年第5期37卷 522-524,F0003页

作者：詹剑杨丽余昌胤范瑞明遵义医学院附属医院神经内科遵义563003

目的:通过观察高血压对大鼠脑缺血再灌注后神经功能和细胞凋亡的影响,探讨高血压加重缺血性脑损伤的机制。方法:将Wistar雄性大鼠随机分为肾性高血压缺血再灌注组(HIR),正常血压缺血再灌注组(IR)和假手术组(N),采用大脑中动脉线栓法造... 详细信息

目的:通过观察高血压对大鼠脑缺血再灌注后神经功能和细胞凋亡的影响,探讨高血压加重缺血性脑损伤的机制。方法:将Wistar雄性大鼠随机分为肾性高血压缺血再灌注组(HIR),正常血压缺血再灌注组(IR)和假手术组(N),采用大脑中动脉线栓法造成大脑缺血再灌注模型,缺血时间为2h,再灌注24h。进行神经功能评分后取脑,采用免疫组织化学技术检测脑组织bcl-2、Bax的表达,TUNEL法检测细胞凋亡。结果:正常血压缺血再灌注组神经功能评分低于肾性高血压缺血再灌注组(P<0.05)。肾性高血压缺血再灌注组与正常血压缺血再灌注组比较,bcl-2表达明显减少(P<0.05),Bax表达明显增多(P<0.05),神经细胞凋亡明显增多(P<0.05)。结论:高血压通过增加Bax表达、抑制bcl-2表达,促进脑细胞凋亡,加重缺血再灌注后脑损伤。

关键词：高血压,肾血管性/并发症脑缺血/病因学再灌注损伤/病理生理学细胞凋亡

来源：评论

学校读者我要写书评

暂无评论

The effects of various organ preservation solutions on hepatocyte membrane potentials, intracellular calcium concentrations, and outcome following liver transplantation

引用

AMERICAN JOURNAL OF SURGERY 2000年第2期179卷 154-160页

作者： Cohen, AJ Burczynski, FJ Rosser, BG Lipschitz, J Minuk, GY Univ Manitoba Hlth Sci Ctr Liver Dis Unit Winnipeg MB R3A 1R9 Canada Univ Manitoba Hlth Sci Ctr Dept Surg Winnipeg MB Canada Univ Manitoba Hlth Sci Ctr Dept Pharmacol Winnipeg MB Canada Univ Manitoba Hlth Sci Ctr Dept Med Winnipeg MB Canada Univ Manitoba Hlth Sci Ctr Fac Med Winnipeg MB Canada Univ Manitoba Hlth Sci Ctr Fac Pharm Winnipeg MB Canada

BACKGROUND: Hepatocyte membrane potential differences (PDs) may be altered by the preservation solutions used in liver transplantation. Such alterations could impact on the survival of the donor liver, extent of biochemical injury, and flux of important ionic compounds, The purpose of the present study was to document these outcomes in the presence of four different presentation solutions. METHODS: Livers of adult male Sprague-Dawley rats (N = 3 to 4 per group) were impaled with intracellular microelectrodes prior to and at various time periods for 6 hours following complete hepatic resection, Just prior to resection, each liver was perfused with preservation solutions associated with high (normal saline [NS]), moderate (Euro-Collins [EC]), and low (University of Wisconsin solution [UW]) risks of reperfusion injury. RESULTS: Baseline (in situ) PDs were similar in all groups (-37 +/- 4 mV, mean +/- SD). Ten minutes postresection, hepatic PDs were as follows: NS, -23.8 +/- 3.5 mV;EC, -11.4 +/- 0.4 mV;and UW, -8.7 +/- 0.3 mV (P <0.01 for all groups). Maximum depolarization occurred at 6 hours postresection (NS, -8.1 +/- 1.1 mV;EC, -7.7 +/- 1.3 mV;and UW, -8.6 +/- 1.0 mV), To determine whether these changes are of pathophysiologic importance, the NS solution was modified (addition of 0.1% ethanol) to achieve similar PD changes as those observed with UW. Liver transplants were then performed where the donor livers had been perfused and preserved for 6 hours with either NS or the modified NS (MNS) solution. Posttransplant (10 day) survival was 1 of 6 (17%) in the NS group and 4 of 6 (67%) in the MNS group (P <0.05), Regarding the effects of PD changes on ionic flux, intracellular calcium levels were documented for up to 4 hours by fluorescence video microscopy using Fura-a in isolated hepatocytes exposed to NS, UW, and MNS solutions. Intracellular calcium levels were similar in all solutions at each time point studied. CONCLUSIONS: The results of this study indicate that

关键词：腺苷/治疗应用别嘌呤醇/治疗应用钙/分析钙信号/药物作用乙醇/治疗应用荧光染料/诊断应用 Fura-2/诊断应用谷胱甘肽/治疗应用移植物存活/药物作用肝切除术高渗溶液/治疗应用胰岛素/治疗应用肝/药物作用肝/病理学肝/病理生理学肝移植/病理学肝移植/生理学膜电位/药物作用显微镜检查荧光显微镜检查电视器官保存器官保存液/治疗应用棉子糖/治疗应用大鼠 Sprague-Dawley 再灌注损伤/病理生理学再灌注损伤/预防和控制氯化钠统计学非参数治疗结果动物男(雄)性大鼠

来源：评论

学校读者我要写书评

暂无评论

Regulation of liver inflammatory injury by signal transducer and activator of transcription-6

引用

AMERICAN JOURNAL OF PATHOLOGY 2000年第1期157卷 297-302页

作者： Kato, A Yoshidome, H Edwards, MJ Lentsch, AB Univ Louisville Sch Med JG Brown Canc Ctr Dept Surg Louisville KY 40202 USA

Liver Injury induced by hepatic ischemia/reperfusion is characterized by activation of the transcription factor NF-KB, increased production of tumor necrosis factor-alpha (TNF alpha), liver neutrophil accumulation, and hepatocellular damage. Exogenous administration of interleukin-4 (IL-4) or IL-13 was recently shown to regulate this inflammatory Injury in association with activation of signal transducer and activator of transcription-6 (STAT6), The objective of the present study was to determine whether STAT6 was required for the regulation of liver inflammation by IL-4 and IL-13. Wild-type and STAT6 knockout mice underwent 90 minutes of hepatic ischemia followed by 8 hours of reperfusion, Hepatic ischemia/reperfusion in wildtype and STAT6 knockout mice significantly Increased (P < 0.05) NF-kappa B activation, serum levels of TNF alpha, liver accumulation of neutrophils [measured by myeloperoxidase (MPO) content], and hepatocellular damage [measured by serum alanine aminotransferase (ALT)] compared to sham controls. In wild-type mice, activation of STAT6 was not observed after ischemia/reperfusion, Administration of 1 mu g of IL-4 or IL-13 at reperfusion reduced serum TNF alpha, liver neutrophil accumulation, and hepatocellular injury in wild-type mice, Treatment with IL-4 or IL-13 had no effect on liver NF-kappa B activation but significantly Increased activation of STAT6, In STAT6 knockout mice, neither IL-4 nor IL-13 had any effect on TNF alpha, MPO, or ALT values, the regulatory effects of these cytokines being completely abolished. The data suggest that activation of STAT6 may regulate liver inflammatory injury.

关键词：基因表达调控/药物作用炎症白细胞介素13/药理学白细胞介素4/药理学缺血/病理生理学缺血/预防和控制肝/药物作用肝/代谢肝/病理学小鼠近交BALBC 小鼠基因敲除 NF-κB/代谢 RNA 信使/药物作用 RNA 信使/遗传学 RNA 信使/代谢再灌注损伤/病理生理学再灌注损伤/预防和控制 STAT6转录因子信号传导反式激活因子类/遗传学反式激活因子类/代谢反式激活因子类/生理学肿瘤坏死因子α/遗传学肿瘤坏死因子α/代谢动物男(雄)性小鼠

来源：评论

学校读者我要写书评

暂无评论

Reperfusion injury pathophysiology in sickle transgenic mice

引用

BLOOD 2000年第1期96卷 314-320页

作者： Osarogiagbon, UR Choong, S Belcher, JD Vercellotti, GM Paller, MS Hebbel, RP Univ Minnesota Sch Med Dept Med Minneapolis MN 55455 USA

Reperfusion of tissues after interruption of their vascular supply causes free-radical generation that leads to tissue damage, a scenario referred to as "reperfusion Injury." Because sickle disease involves repeated transient ischemic episodes, we sought evidence for excessive free-radical generation in sickle transgenic mice. Compared with normal mice, sickle mice at ambient air had a higher ethane excretion (marker of lipid peroxidation) and greater conversion of salicylic acid to 2,3-dihydroxybenzoic acid (marker of hydroxyl radical generation). During hypoxia (11% O-2), only sickle mice converted tissue xanthine dehydrogenase to oxidase. Only the sickle mice exhibited a further increase in ethane excretion during restitution of normal oxygen tension after 2 hours of hypoxia. Only the sickle mice showed abnormal activation of nuclear factor-kappa B after exposure to hypoxia-reoxygenation. Allopurinol, a potential therapeutic agent, decreased ethane excretion in the sickle mice. Thus, sickle transgenic mice exhibit biochemical foot-prints consistent with excessive free-radical generation even at ambient air and following a transient induction of enhanced sickling. We suggest that reperfusion injury physiology may contribute to the evolution of the chronic organ damage characteristic of sickle cell disease. If so, novel therapeutic approaches might be of value. (Blood. 2000;96:314-320) (C) 2000 by The American Society of Hematology.

关键词：别嘌呤醇/治疗应用贫血镰状细胞性/药物疗法贫血镰状细胞性/遗传学贫血镰状细胞性/病理生理学酶抑制剂/治疗应用乙烷/分析血红蛋白镰状/遗传学羟自由基/代谢肾/代谢脂质过氧化作用肝/代谢小鼠近交C57BL 小鼠转基因 NF-κB/代谢参考值再灌注损伤/病理生理学水杨酸/药代动力学黄嘌呤脱氢酶/代谢黄嘌呤氧化酶/代谢动物人类小鼠

来源：评论

学校读者我要写书评

暂无评论

Partial liquid ventilation for acute allograft dysfunction after canine lung transplantation

引用

ANNALS OF THORACIC SURGERY 1999年第2期67卷 332-339页

作者： Itano, H Aoe, M Ichiba, S Yamashita, M Date, H Andou, A Shimizu, N Okayama Univ Sch Med Dept Surg 2 Okayama 7008558 Japan

Background. This study was designed to investigate the efficacy of partial liquid ventilation (PLV) on acute allograft dysfunction after lung transplantation. Methods. The canine left lung allotransplantation model was used, with the graft preserved in 4 degrees C lowpotassium dextran glucose solution for 18 hours. The control group (n = 6) had conventional mechanical ventilation, and the PLV group (n = 6) had perfluorooctylbromide instilled into the airway 30 minutes after reperfusion. For 360 minutes, allograft function and hemodynamics were evaluated. After the evaluation, myeloperoxidase activity of the graft tissue was assayed. Results. All dogs survived for 360 minutes. In the PT,V group, Pao, shunt fraction, and alveolar to arterial gradient for O-2, were si,significantly better than those in the control group after 120, 180, and 120 minutes, respectively (p < 0.05). After 240 minutes, peak airway pressure became significantly lower than that in the control group (p < 0.05). The Pao, at 360 minutes was 102 +/- 55 mm Hg in the control group and 420 +/- 78 mm Hg in the PLV group (p < 0.0001), and the peak airway pressure was 21.4 +/- 4.1 mm Hg in the control group and 14.7 +/- 5.0 mm Hg in the PLV group (p < 0.05). Myeloperoxidase activity in the PLV group was lower than that in the control group. Conclusions. The study shows that PLV alleviated acute allograft dysfunction after lung transplantation. (C) 1999 by The Society of Thoracic Surgeons.

关键词：氟碳化合物/投药和剂量肺/血液供给肺/病理学肺移植/病理学肺移植/生理学氧/血液肺换气/生理学再灌注损伤/病理学再灌注损伤/病理生理学再灌注损伤/治疗呼吸人工移植同种动物狗

来源：评论

学校读者我要写书评

暂无评论

Reperfusion pattern of the immature femoral head after critical ischemia -: A microsphere study in pigs

引用

ACTA ORTHOPAEDICA SCANDINAVICA 1999年第5期70卷 439-445页

作者： Drescher, W Schneider, T Becker, C Hobolth, L Rüther, W Bünger, C Hansen, ES Univ Aarhus Inst Expt Clin Res Aarhus Denmark Univ Dusseldorf Dept Orthoped D-4000 Dusseldorf Germany Univ Hamburg Dept Orthoped Hamburg Germany

The topographic reperfusion pattern of the femoral head after critical ischemia has not yet been investigated. We determined the blood flow of the porcine hip regions with the femoral head epiphysis divided into 24 subregions by the tracer microsphere technique. Blood flow was measured under steady-state conditions, at the end of a 6-hour increase in intracapsular hip joint pressure to 250 mm Hg, and 4 hours after release of the joint tamponade. Total femoral head epiphyseal blood flow decreased with ischemia and regained steady-state perfusion after tamponade. The reperfusion pattern of the femoral head epiphysis appeared identical with that of the steady state before ischemia. However, 2 of the II experimental epiphyses remained ischemic in the reperfusion phase. We conclude that hip joint tamponade above the arterial pressure level for 6 hours causes global ischemia in the femoral head epiphysis in the immature pig, without regional differences in reperfusion, and that reperfusion occurs at a level like that of the steady state before ischemia.

关键词：年龄因素活组织检查血流速度血压疾病模型动物股动脉/病理生理学股骨头/血液供给股骨头/生长和发育微球体放射性示踪剂随机分配再灌注损伤/病因学再灌注损伤/病理学再灌注损伤/病理生理学猪时间因素动物女(雌)性男(雄)性

来源：评论

学校读者我要写书评

暂无评论

Lowering reperfusion pressure reduces the injury after pulmonary ischemia

引用

ANNALS OF THORACIC SURGERY 2000年第1期69卷 198-203页

作者： Halldorsson, AO Kronon, MT Allen, BS Rahman, S Wang, TR Hope Childrens Hosp Heart Inst Children Div Cardiothorac Surg Oak Lawn IL 60453 USA Univ Chicago Chicago IL 60637 USA

Background. Controlled reperfusion with a modified solution limits pulmonary injury following ischemia. Our initial studies infused this modified reperfusate at a pressure of 40 to 50 mm Hg to insure distribution. However, perhaps a lower pressure, which is closer to the normal physiologic pressure in the lung, would improve results by decreasing sheer stress. Methods. Fifteen adult pigs underwent 2 hours of lung ischemia by clamping the left bronchus and pulmonary artery. Five (group 1) then underwent uncontrolled reperfusion by removing the vascular clamps and allowing unmodified blood to reperfuse the lung at a pulmonary artery pressure of 20 to 30 mm Hg. The other 10 pigs underwent controlled reperfusion by mixing blood from the femoral artery with a crystalloid solution, and infusing this modified reperfusate into the ischemic lung through the pulmonary artery for 10 minutes before removing the arterial clamp. In 5 (group 2), the modified solution was infused at a pressure of 40 to 50 mm Hg, and in 5 (group 3) 20 to 30 mm Hg. Lung function was assessed 60 minutes after reperfusion and expressed as percentage of control. Results. Compared to uncontrolled reperfusion (group 1), controlled reperfusion at a pressure of 40 to 50 mm Hg (group 2) significantly improved postreperfusion pulmonary compliance (77% versus 86%;p < 0.001 versus group 1), and arterial/alveolar ratio (a/A) ratio (27% versus 52%;p < 0.001 versus group 1);as well as decreased pulmonary vascular resistance (PVR) (198% versus 154%;p < 0.001 versus group 1), lung water (84.3% versus 83.5%;p < 0.001 versus group 1), and myeloperoxidase (0.35 versus 0.23 optical density/min/mg protein). Reducing the pressure of the modified reperfusate to 20 to 30 mm Hg further improved postreperfusion compliance (92% +/- 1%;p < 0.001 versus groups 1 and 2) and a/A ratio (76% +/- 1%;p < 0.001 versus groups 1 and 2);and lowered PVR (133% +/- 2%;p < 0.001 versus groups 1 and 2), lung water (82.7% +/- 0.1%;p < 0.001 ver

关键词：血液血压/生理学身体水份/化学支气管收缩缺血/治疗肺/血液供给肺/代谢肺/病理生理学肺顺应性/生理学器官保存液/投药和剂量器官保存液/治疗应用过氧化物酶/分析压力肺动脉肺水肿/病因学肺水肿/代谢肺换气/生理学再灌注/方法再灌注损伤/病理生理学再灌注损伤/预防和控制流变学猪血管阻力/生理学动物

来源：评论

学校读者我要写书评

暂无评论

The role of glutamine in skeletal muscle ischemia/reperfusion injury in the rat hind limb model

引用

AMERICAN JOURNAL OF SURGERY 1999年第2期178卷 147-150页

作者： Prem, JT Eppinger, M Lemmon, G Miller, S Nolan, D Peoples, J Wright State Univ Sch Med Dept Surg Dayton OH 45409 USA

OBJECTIVES: Ischemia/reperfusion injury is a commonly occurring event with severe pathologic consequences. Reperfusion initiates both the local and systematic damage in part through rapid oxygen generation. The glutathione system is a major mechanism of reducing this oxidative stress. If this system can be maintained or augmented during this stress then less damage may occur. Glutamine provides the source of glutamate to this system and has been shown to preserve total glutathione levels after injury/ischemia to both hepatic and gut models. To test this effect, we looked at glutamine and its role in ischemia/reperfusion injury in a rat hind limb model. METHODS: Fifty male HSD/Holtzman rats weighing 350-400 g were randomized to receive glutamine (3% sol) or normal saline via intraperitoneal injections. The groups were then subjected to 2 hours of ischemia to their hind limbs using the Tourni-Cot method. Animals were then randomized to reperfusion groups of 30 minutes, 2 hours, and 4 hours. Muscle tissue assays were performed for lipid peroxidation (LPO), total glutathione (GSH), and myeloperoxidase (MPO). Peripheral blood was analyzed for creatinephosphokinase levels (CPK). RESULTS: Animals that received glutamine showed a general trend of less lipid peroxidation products than the normal saline groups. In animals that received glutamine and underwent 2 hours of ischemia and reperfusion times of 0 minutes, 30 minutes, and 2 hours, there were significantly less percent changes in lipid peroxidation products from controls (4.6% vs 48.2%, P <0.05), (18.9% vs 123%, P <0.05), (12.6% vs 115%, P <0.05). A general trend upward was noted in CPK levels in both groups. In animals receiving 2 hours of ischemia and 30 minutes of reperfusion, there was a significantly greater level of creatinephosphokinase (CPK) calculated as percent change from control in the normal saline group as compared with the glutamine group (209.2% vs 92.7%). Myeloperoxidase assay of muscle tissue revealed

关键词：肌酸激酶/血液疾病模型动物谷氨酸/代谢谷氨酰胺/投药和剂量谷氨酰胺/治疗应用谷胱甘肽/生物合成后肢/血液供给注射腹腔内缺血/代谢缺血/病理生理学脂质过氧化作用/药物作用肌骨骼/血液供给肌骨骼/药物作用肌骨骼/代谢肌骨骼/病理学中性白细胞/药物作用中性白细胞/病理学氧化性应激/药物作用氧化性应激/生理学过氧化物酶/代谢预防和防护用药/投药和剂量预防和防护用药/治疗应用随机分配大鼠近交系再灌注损伤/代谢再灌注损伤/病理生理学再灌注损伤/预防和控制单盲法时间因素动物男(雄)性大鼠

来源：评论

学校读者我要写书评

暂无评论

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

请选择保存的检索档案：

请选择收藏分类：

通借通还

建议与咨询 留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

请选择保存的检索档案： 新增检索档案 确定 取消

请选择收藏分类： 新增自定义分类 确定 取消

通借通还

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

请选择保存的检索档案：

请选择收藏分类：