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检索条件"主题词=Bnip3"
514 条 记 录,以下是161-170 订阅
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New relationship of E2F1 and bnip3 with caveolin-1 in lung cancer-associated fibroblasts
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THORACIC CANCER 2020年 第6期11卷 1369-1371页
作者: Shen, Cheng Chen, Xuanming Xiao, Kai Che, Guowei Sichuan Univ West China Hosp Dept Thorac Surg Chengdu 610041 Peoples R China Sichuan Univ West China Hosp Sch Med Chengdu Peoples R China Sichuan Kangcheng Biotechnol Co Ltd Chengdu Peoples R China
In recent years, studies have found that E2F1, a downstream effector of caveolin-1 (Cav-1), participates in tumor cell metabolic reprogramming. E2F1 modulates mitochondrial fusion and mitophagy. Bioinformatic analysis... 详细信息
来源: 评论
Placental localization and expression of the cell death factors bnip3 and Nix in preeclampsia, intrauterine growth retardation and HELLP syndrome
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EUROPEAN JOURNAL OF OBSTETRICS & GYNECOLOGY AND REPRODUCTIVE BIOLOGY 2005年 第2期122卷 172-176页
作者: Stepan, H Leo, C Purz, S Höckel, M Horn, LC Univ Leipzig Dept Obstet & Gynecol D-04103 Leipzig Germany Univ Leipzig Inst Pathol Div Gynecol & Perinatal Pathol D-04103 Leipzig Germany
Objective: bnip3 and its homologue Nix are pro-apoptotic factors of the Bcl-2-family and are expressed in malignant tumors. In vitro, this expression was shown to be mediated by hypoxia. Recently, it has been shown th... 详细信息
来源: 评论
Regulation of bnip3 in normal and cancer cells
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MOLECULES AND CELLS 2006年 第1期21卷 1-6页
作者: Lee, H Paik, SG Chungnam Natl Univ Sch Biosci & Biotechnol Dept Biol Taejon 305764 South Korea Chungnam Natl Univ Inst Biotechnol Taejon 305764 South Korea
Bcl-2/adenovirus E1B 19 kDa-interacting protein 3 (bnip3) is a mitochondrial pro-apoptotic protein that has a single Bcl-2 homology 3 (BH3) domain and a COOH-terminal transmembrane (TM) domain. Although it belongs to ... 详细信息
来源: 评论
Epigenetic Regulation of E2F-1-Dependent bnip3 Transcription and Cell Death by Nuclear Factor-κB and Histone Deacetylase-1
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PEDIATRIC CARDIOLOGY 2011年 第3期32卷 263-266页
作者: Gang, Hongying Dhingra, Rimpy Wang, Yan Mughal, Wajihah Gordon, Joseph W. Kirshenbaum, Lorrie A. St Boniface Gen Hosp Res Ctr Dept Pharmacol & Therapeut Fac MedInst Cardiovasc Sci Winnipeg MB R2H 2A6 Canada St Boniface Gen Hosp Res Ctr Dept Physiol Fac MedInst Cardiovasc Sci Winnipeg MB R2H 2A6 Canada
A delicate balance exists between cell growth and cell death. In the context of the adult myocardium, inappropriate or inordinate cell loss through an apoptotic process may profoundly influence cardiac structure, func... 详细信息
来源: 评论
An unexpected journey for bnip3
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AUTOPHAGY 2024年 第6期20卷 1447-1448页
作者: Delgado, Jose M. Shoemaker, Christopher J. Dartmouth Coll Geisel Sch Med Dept Biochem & Cell Biol Hanover NH USA Dartmouth Canc Ctr Lebanon NH USA Dartmouth Coll Geisel Sch Med Dept Biochem & Cell Biol Hanover NH 03755 USA
Mitophagy is a cellular process that enables the selective degradation of damaged, dysfunctional, or superfluous mitochondria. During mitophagy, specific proteins recognize and tag mitochondria for degradation. These ... 详细信息
来源: 评论
Regulation of mitochondrial integrity, autophagy and cell survival by bnip3
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AUTOPHAGY 2007年 第6期3卷 616-619页
作者: Tracy, Kristin Macleod, K. F. Univ Chicago Ben May Dept Canc Res Chicago IL 60637 USA
Understanding the role of bnip3 in the systemic response to hypoxia has been complicated by conflicting results that indicate on the one hand that bnip3 promotes cell death, and other data, including our own that bnip... 详细信息
来源: 评论
Post-Translational Control of bnip3 and Mitophagy by ULK1 Kinase
Post-Translational Control of BNIP3 and Mitophagy by ULK1 Ki...
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作者: Poole, Logan Patrick The University of Chicago
学位级别:Ph.D., Doctor of Philosophy
bnip3 is a mitochondrial cargo receptor that specifically targets mitochondria for degradation at the autolysosome through a specialized form of autophagy called mitophagy. bnip3 is transcriptionally upregulated in re... 详细信息
来源: 评论
Isolation of Rab5-positive endosomes reveals a new mitochondrial degradation pathway utilized by bnip3 and Parkin
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Small GTPases 2020年 第1期11卷 69-76页
作者: Hammerling, Babette C. Shires, Sarah E. Leon, Leonardo J. Cortez, Melissa Q. Gustafsson, Åsa B. Skaggs School of Pharmacy and Pharmaceutical Sciences University of California San Diego La Jolla CA United States
Degradation of mitochondria is an important cellular quality control mechanism mediated by two distinct pathways: one involving Parkin-mediated ubiquitination and the other dependent on mitophagy receptors. It is know... 详细信息
来源: 评论
bnip3 Regulates at101 [(-)-Gossypol] Induced Death in Malignant Peripheral Nerve Sheath Tumor Cells
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2016年
作者: niroop kaza
bnip3 Regulates at101 [(-)-Gossypol] Induced Death in Malignant Peripheral Nerve Sheath Tumor Cells. by Kaza, Niroop; Kohli, Latika; Graham, Christopher D.; Klocke, Barbara J.; Carroll, Steven L.; Roth, Kevin A.; publ... 详细信息
来源: 评论
bnip3 is essential for mediating 6-thioguanine-and 5-fluorouracil-induced autophagy following DNA mismatch repair processing
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细胞研究(英文版) 2010年 第6期20卷 665-675页
作者: Xuehuo Zeng Timothy J Kinsella Department of Radiation Oncology The Case Integrative Cancer Biology Program Case Western Reserve University ClevelandOH44106 USA
DNA mismatch repair (MMR) processes the chemically induced mispairs following treatment with clinically important nucleoside analogs such as 6-thioguanine (6-TG) and 5-fluorouracil (5-FU). MMR processing of these drug... 详细信息
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