Reconfigurable computing has shown significant promise in many fields. The lack of high-level design tools hampers the widespread adoption of reconfigurable computing systems. The designer needs both in-depth software...
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Reconfigurable computing has shown significant promise in many fields. The lack of high-level design tools hampers the widespread adoption of reconfigurable computing systems. The designer needs both in-depth software and hardware design knowledge to develop applications for GPP/RAU hybrid system. This paper presents an initial ASCRA (Application-Specific Compiler for Reconfigurable Architecture) compilation framework for automatic mapping of C to VHDL that brings the gap in automatic compilation tools for reconfigurable computing. In ASCRA, the loop pipelining is mainly concerned. The loop pipelining framework is presented in this paper. Most compilers use a modulo scheduling with a constant initiation interval to schedule the start of each iteration and use a directed pipelining division method to generate pipeline. In order to increase throughput and reduce the pipeline depth, a modulo scheduling with the variable initiation interval and an improved pipelining division method are presented in ASCRA. In modulo scheduling, the variable initiation interval is generated by the two-dimensional vector algorithm. The improved pipelining division method mergers the shorter delay pipeline stage without changing the maximum frequency to generate pipeline. A verification platform of ASCRA is also built on a ML509 development board. The experiment on selected kernels that shows the methods give significant performance improvement.
This paper reviews the work of Wu and Lin on the Union Jack lattice Ising model. This model is of interest as it one of the few to display re-entrant phase transitions. Specifically, we re-examine their result for the...
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This paper reviews the work of Wu and Lin on the Union Jack lattice Ising model. This model is of interest as it one of the few to display re-entrant phase transitions. Specifically, we re-examine their result for the general anisotropic sublattice magnetizations, comparing these with the works of Vaks, Larkin and Ovchinnikov, and our own numerical simulations. We discuss the disagreements found in both sublattice predictions including non-zero antiferromagnetic results and a rotational variance. We will then suggest additional conditions and modified formulae that will allow valid results to be produced.
The in vitro effects of insulin on different phosphodiesterase activities present in rat epididymal fat cells from normal and hypothyroid rats were studied. Insulin increases the Vmax of a particulate, low Km, cyclic[...
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The in vitro effects of insulin on different phosphodiesterase activities present in rat epididymal fat cells from normal and hypothyroid rats were studied. Insulin increases the Vmax of a particulate, low Km, cyclic[c]AMP phosphodiesterase in both types of cells, this effect being more clearly evident with the fat cells from hypothryoid animals;combination of insulin and thyroidectomy resulted in a 400% stimulation with 10-10 to 10-9 M insulin. A clear and significant effect was apparent at 10-11 M insulin. The dose-response curve was biphasic, since stimulation by insulin was suppressed for doses of hormone higher than 10-8 to 10-7 M. Insulin effects were very fast, since clear stimulation was observed after only 2 min of incubation;the maximal increase was obtained after 10 min. Insulin did not significantly affect the soluble cAMP phosphodiesterase aactivity in normal cells, confirming results obtained by others. The soluble cAMP phosphodiesterase activity was clearly stimulated by insulin when the fat cells were prepared from hypothyroid rats. Maximal stimulation was obtained with 10-9 M insulin;the response was again very fast. Soluble cGMP phosphodiesterase activity was also increased additively by hypothyroidism and insulin, maximal stimulation being obtained with 10-9 M insulin. With this dose of insulin the additive effects of thyroidectomy and insulin produced a 5-fold stimulation. The effect of insulin on the soluble cGMP phosphodiesterase was very fast (2-5 min). With both soluble cyclic nucleotide phosphodiesterase activities, insulin increased the maximal velocity but not the apparent Km of the enzyme. Hypothyroidism and insulin produced additive effects suggesting a different mechanism of action of these 2 hormonal situations on the degradation of the intracellular pools of cAMP and cGMP.
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