Background Excessive iron accumulation in the brain with oxidative damage has commonly been observed in hemorrhagic stroke as well as neurodegenerative *** L-type calcium channels are key passages responsible for the ...
Background Excessive iron accumulation in the brain with oxidative damage has commonly been observed in hemorrhagic stroke as well as neurodegenerative *** L-type calcium channels are key passages responsible for the iron entry into neuronal cells during iron overloaded state and therefore we postulated its blocker could potentially protect neural stem cells (NSCs) from iron induced toxicity.
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