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Author Correction: Single-nucleus chromatin accessibility profiling highlights regulatory mechanisms of coronary artery disease risk

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Nature genetics 2022年第8期54卷 1259页

作者： Adam W Turner Shengen Shawn Hu Jose Verdezoto Mosquera Wei Feng Ma Chani J Hodonsky Doris Wong Gaëlle Auguste Yipei Song Katia Sol-Church Emily Farber Soumya Kundu Anshul Kundaje Nicolas G Lopez Lijiang Ma Saikat Kumar B Ghosh Suna Onengut-Gumuscu Euan A Ashley Thomas Quertermous Aloke V Finn Nicholas J Leeper Jason C Kovacic Johan L M Björkegren Chongzhi Zang Clint L Miller Center for Public Health Genomics University of Virginia Charlottesville VA USA. Department of Biochemistry and Molecular Genetics University of Virginia Charlottesville VA USA. Medical Scientist Training Program University of Virginia Charlottesville VA USA. Department of Pathology University of Virginia Charlottesville VA USA. Robert M. Berne Cardiovascular Research Center University of Virginia Charlottesville VA USA. Genome Analysis & Technology Core University of Virginia Charlottesville VA USA. Genome Sciences Laboratory University of Virginia Charlottesville VA USA. Department of Genetics Stanford University School of Medicine Stanford CA USA. Department of Computer Science Stanford University Stanford CA USA. Division of Vascular Surgery Department of Surgery Stanford University Stanford CA USA. Department of Genetics and Genomic Sciences Icahn Institute for Genomics and Multiscale Biology Icahn School of Medicine at Mount Sinai New York NY USA. CVPath Institute Gaithersburg MD USA. Department of Public Health Sciences University of Virginia Charlottesville VA USA. Division of Cardiovascular Medicine Department of Medicine Stanford University Stanford CA USA. Cardiovascular Research Institute Icahn School of Medicine at Mount Sinai New York NY USA. Victor Chang Cardiac Research Institute Darlinghurst New South Wales Australia. St. Vincent's Clinical School University of New South Wales Sydney New South Wales Australia. Integrated Cardio Metabolic Centre Department of Medicine Karolinska Institutet Huddinge Sweden. Center for Public Health Genomics University of Virginia Charlottesville VA USA. zang@virginia.edu. Department of Biochemistry and Molecular Genetics University of Virginia Charlottesville VA USA. zang@virginia.edu. Department of Public Health Sciences University of Virginia Charlottesville VA USA. zang@virginia.edu. Center for Public Health Genomics University of Virginia Charlottesville VA USA. clintm@virg

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Prostaglandin E2-EP4 signaling persistently amplifies CD40-mediated induction of IL-23 p19 expression through canonical and non-canonical NF-κB pathways

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Cellular & Molecular Immunology 2016年第2期13卷 240-250页

作者： Xiaojun Ma romohiro Aoki Shuh Narumiya Core Research for Evolutional Science and Technology (CREST) Medical Innovation Center Kyoto University Graduate School of Medicine Kyoto 606-8507 Japan Center for Innovation in Immunoregulation Technology and Therapeutics Kyoto University Graduate School of Medicine Kyoto 606-8501 Japan

While there is mounting evidence that interleukin （IL）-23-IL-17 axis plays a critical role in the pathogenesis of various autoimmune diseases, much remains to be elucidated on how IL-23 is induced in the pathological processes. IL-23 is a heterodimer composed of p19 and p40, the latter being shared with IL-12. We previously reported that prostaglandin （PG） E2 promotes CD40-mediated induction of 1123a （p19） expression through its E receptor subtype 4 （EP4） receptor in splenic dendritic cells （DCs）. Here, we have analyzed signaling pathways regulating 1123a induction in the cross talk between EP4 and CD40 in bone marrow-derived DCs. We found that PGE2 synergistically induced 1123a transcription with CD40 signaling. An EP4 agonist, but not agonists of EP1, EP2, or EP3, reproduced this action. Stimulation of CD40 with an agonist antibody evoked biphasic induction of 1123a expression, with the early phase peaking at 1 h and the late phase peaking at 12 h and lasting up to 36 h after stimulation, whereas induction by lipopolysaccharide or tumor necrosis factor-α was transient. The early phase induction by CD40 stimulation was absent in DCs derived from Nfkbl-deficient mice, and the late phase induction was eliminated by RNA interference of nuclear factor-kappa B （NF-κB） p100 subunit. Further, cAMP response element-binding protein （CREB） depletion completely eliminated the induction of 1123a by CD40 stimulation. The addition of the EP4 agonist amplified the induction in both phases through the cAMP-protein kinase A （PKA） pathway. These results suggest that 1123a expression in DCs is synergistically triggered by the PG E2-EP4-cAMP-PKA pathway and canonical/non-canonical NF-KB pathways and CREB activated by CD40 stimulation.

关键词： dendritic cell IL-23 p19 prostaglandin E2 EP4

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Decomposition of powerful axisymmetrically polarized laser pulses in underdense plasma

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Physical Review E 2016年第6期94卷 063205-063205页

作者： Nobuhiko Nakanii Tomonao Hosokai Naveen C. Pathak Shinichi Masuda Alexei G. Zhidkov Hiroki Nakahara Kenta Iwasa Yoshio Mizuta Naoki Takeguchi Takamitsu P. Otsuka Keiichi Sueda Jumpei Ogino Hirotaka Nakamura Michiaki Mori Masaki Kando Ryosuke Kodama Photon Pioneers Center Osaka University Core Research of Evolutional Science and Technology (CREST) Japan Science and Technology Agency Kansai Photon Science Institute National Institutes for Quantum and Radiological Science and Technology Graduate School of Engineering Osaka University Institute of Laser Engineering Osaka University

Interaction of relativistically intense axisymmetrically polarized (radially or azimuthally polarized) laser pulses (RIAPLP) with underdense plasma is shown experimentally and theoretically to be essentially different from the interaction of conventional Gaussian pulses. The difference is clearly observed in distinct spectra of the side-scattered laser light for the RIAPLP and Gaussian pulses, as well as in the appearance of a spatially localized strong side emission of second harmonic of the laser pulse in the case of RIAPLP. According to our analysis based on three-dimensional particle-in-cell simulations, this is a result of instability in the propagation of RIAPLP in uniform underdense plasma.

关键词： Laser driven ion acceleration

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Quantitative imaging of fibrotic and morphological changes in liver of non-alcoholic steatohepatitis (NASH) model mice by second harmonic generation (SHG) and auto-fluorescence (AF) imaging using two-photon excitation microscopy (TPEM)

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Biochemistry and Biophysics Reports 2016年 8卷 277-283页

作者： Yamamoto, Shin Oshima, Yusuke Saitou, Takashi Watanabe, Takao Miyake, Teruki Yoshida, Osamu Tokumoto, Yoshio Abe, Masanori Matsuura, Bunzo Hiasa, Yoichi Imamura, Takeshi Department of Molecular Medicine for Pathogenesis Ehime University Graduate School of Medicine Japan Department of Gastroenterology and Metabiology Ehime University Graduate School of Medicine Japan Division of Bio-imaging Proteo-Science Center Ehime University Japan Translational Research Center Ehime University Hospital Japan Core Research for Evolutional Science and Technology (CREST) Japan Science and Technology Agency (JST) Japan Department of Lifestyle-related Medicine and Endocrinology Ehime University Graduate School of Medicine Japan

Non-alcoholic steatohepatitis (NASH) is a common liver disorder caused by fatty liver. Because NASH is associated with fibrotic and morphological changes in liver tissue, a direct imaging technique is required for accurate staging of liver tissue. For this purpose, in this study we took advantage of two label-free optical imaging techniques, second harmonic generation (SHG) and auto-fluorescence (AF), using two-photon excitation microscopy (TPEM). Three-dimensional ex vivo imaging of tissues from NASH model mice, followed by image processing, revealed that SHG and AF are sufficient to quantitatively characterize the hepatic capsule at an early stage and parenchymal morphologies associated with liver disease progression, respectively. © 2016 The Authors

关键词： Collagen Fibrosis Fluorescence imaging NASH SHG Two-photon excitation microscopy

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Corrigendum to "Derivation of pluripotent stem cells from nascent undifferentiated teratoma" [Dev. Biol. 446 (2019) 43-55]

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Developmental biology 2019年第1期453卷 110页

作者： Yuri An Tamotsu Sekinaka Yukiko Tando Daiji Okamura Keiko Tanaka Yumi Ito-Matsuoka Asuka Takehara Nobuo Yaegashi Yasuhisa Matsui Cell Resource Center for Biomedical Research Institute of Development Aging and Cancer (IDAC) Tohoku University Sendai Miyagi Japan Graduate School of Life Sciences Tohoku University Sendai Miyagi Japan. Cell Resource Center for Biomedical Research Institute of Development Aging and Cancer (IDAC) Tohoku University Sendai Miyagi Japan. Department of Advanced Bioscience Graduate School of Agriculture Kindai University Nakamachi Nara Japan. Department of Obstetrics and Gynecology Tohoku University Graduate School of Medicine Sendai Miyagi Japan. Graduate School of Life Sciences Tohoku University Sendai Miyagi Japan The Japan Agency for Medical Research and Development-Core Research for Evolutional Science and Technology (AMED-CREST) Chuo-ku Tokyo Japan Center for Regulatory Epigenome and Diseases Tohoku University School of Medicine Sendai Miyagi Japan. Electronic address: yasuhisa.matsui.d3@tohoku.ac.jp.

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Autism spectrum disorder model mice: Focus on copy number variation and epigenetics

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science China(Life sciences) 2015年第10期58卷 976-984页

作者： Nobuhiro NAKAI Susumu OTSUKA Jihwan MYUNG Toru TAKUMI Graduate School of Biomedical Sciences Hiroshima University Graduate School of Biostudies Kyoto University RIKEN Brain Science Institute Core Research for Evolutional Science and Technology Japan Science and Technology Agency

Autism spectrum disorder(ASD) is gathering concerns in socially developed countries. ASD is a neuropsychiatric disorder of genetic origin with high prevalence of 1%–2%. The patients with ASD characteristically show impaired social skills. Today, many genetic studies identify numerous susceptible genes and genetic loci associated with ASD. Although some genetic factors can lead to abnormal brain function linked to ASD phenotypes, the pathogenic mechanism of ASD is still unclear. Here, we discuss a new mouse model for ASD as an advanced tool to understand the mechanism of ASD.

关键词： copy number variations DNA methylation histone modification ASD model mouse

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Correction: White matter diffusion estimates in obsessive-compulsive disorder across 1653 individuals: machine learning findings from the ENIGMA OCD Working Group

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Molecular psychiatry 2024年第4期29卷 1216页

作者： Bo-Gyeom Kim Gakyung Kim Yoshinari Abe Pino Alonso Stephanie Ameis Alan Anticevic Paul D Arnold Srinivas Balachander Nerisa Banaj Nuria Bargalló Marcelo C Batistuzzo Francesco Benedetti Sara Bertolín Jan Carl Beucke Irene Bollettini Silvia Brem Brian P Brennan Jan K Buitelaar Rosa Calvo Miguel Castelo-Branco Yuqi Cheng Ritu Bhusal Chhatkuli Valentina Ciullo Ana Coelho Beatriz Couto Sara Dallaspezia Benjamin A Ely Sónia Ferreira Martine Fontaine Jean-Paul Fouche Rachael Grazioplene Patricia Gruner Kristen Hagen Bjarne Hansen Gregory L Hanna Yoshiyuki Hirano Marcelo Q Höxter Morgan Hough Hao Hu Chaim Huyser Toshikazu Ikuta Neda Jahanshad Anthony James Fern Jaspers-Fayer Selina Kasprzak Norbert Kathmann Christian Kaufmann Minah Kim Kathrin Koch Gerd Kvale Jun Soo Kwon Luisa Lazaro Junhee Lee Christine Lochner Jin Lu Daniela Rodriguez Manrique Ignacio Martínez-Zalacaín Yoshitada Masuda Koji Matsumoto Maria Paula Maziero Jose M Menchón Luciano Minuzzi Pedro Silva Moreira Pedro Morgado Janardhanan C Narayanaswamy Jin Narumoto Ana E Ortiz Junko Ota Jose C Pariente Chris Perriello Maria Picó-Pérez Christopher Pittenger Sara Poletti Eva Real Y C Janardhan Reddy Daan van Rooij Yuki Sakai João Ricardo Sato Cinto Segalas Roseli G Shavitt Zonglin Shen Eiji Shimizu Venkataram Shivakumar Noam Soreni Carles Soriano-Mas Nuno Sousa Mafalda Machado Sousa Gianfranco Spalletta Emily R Stern S Evelyn Stewart Philip R Szeszko Rajat Thomas Sophia I Thomopoulos Daniela Vecchio Ganesan Venkatasubramanian Chris Vriend Susanne Walitza Zhen Wang Anri Watanabe Lidewij Wolters Jian Xu Kei Yamada Je-Yeon Yun Mojtaba Zarei Qing Zhao Xi Zhu Paul M Thompson Willem B Bruin Guido A van Wingen Federica Piras Fabrizio Piras Dan J Stein Odile A van den Heuvel Helen Blair Simpson Rachel Marsh Jiook Cha Department of Psychology College of Social Sciences Seoul National University Seoul Republic of Korea. Department of Brain and Cognitive Sciences College of Natural Sciences Seoul National University Seoul Republic of Korea. Graduate School of Medical Science Kyoto Prefectural University of Medicine Department of Psychiatry Kyoto City Japan. Bellvitge Biomedical Research Insitute-IDIBELL Bellvitge University Hospital Barcelona Spain. CIBER of Mental Health (CIBERSAM) Carlos III Health Institute Madrid Spain. Department of Clinical Sciences University of Barcelona Barcelona Spain. The Margaret and Wallace McCain Centre for Child Youth & Family Mental Health and Campbell Family Mental Health Research Institute Centre for Addiction and Mental Health Toronto ON Canada. Department of Psychiatry University of Toronto Toronto ON Canada. Program in Neurosciences and Mental Health The Hospital for Sick Children Toronto ON Canada. Department of Psychiatry Yale University School of Medicine New Haven CT 06510 USA. The Mathison Centre for Mental Health Research & Education Hotchkiss Brain Institute Cumming School of Medicine University of Calgary Calgary AB Canada. Departments of Psychiatry and Medical Genetics Cumming School of Medicine University of Calgary Calgary AB Canada. OCD clinic Department of Psychiatry National Institute of Mental Health And Neurosciences (NIMHANS) Bangalore India. Laboratory of Neuropsychiatry Department of Clinical and Neuroscience and Neurorehabilitation IRCCS Santa Lucia Foundation Rome Italy. Center of Image Diagnostic Hospital Clínic de Barcelona Barcelona Spain. Magnetic Resonance Image Core Facility Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS) Barcelona Spain. Departamento e Instituto de Psiquiatria do Hospital das Clinicas IPQ HCFMUSP Faculdade de Medicina Universidade de Sao Paulo Sao Paulo SP Brazil. Department of Methods and Techniques in Psychology Pontifical Catholic University São Paulo SP

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Genome-wide analyses identify 30 loci associated with obsessive–compulsive disorder

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Nature Genetics 2025年 1-13页

作者： Strom, Nora I. Gerring, Zachary F. Galimberti, Marco Yu, Dongmei Halvorsen, Matthew W. Abdellaoui, Abdel Rodriguez-Fontenla, Cristina Sealock, Julia M. Bigdeli, Tim Coleman, Jonathan R. Mahjani, Behrang Thorp, Jackson G. Bey, Katharina Burton, Christie L. Luykx, Jurjen J. Zai, Gwyneth Alemany, Silvia Andre, Christine Askland, Kathleen D. Bäckman, Julia Banaj, Nerisa Barlassina, Cristina Nissen, Judith Becker Bienvenu, O. Joseph Black, Donald Bloch, Michael H. Børte, Sigrid Bosch, Rosa Breen, Michael Brennan, Brian P. Brentani, Helena Buxbaum, Joseph D. Bybjerg-Grauholm, Jonas Byrne, Enda M. Cabana-Dominguez, Judit Camarena, Beatriz Camarena, Adrian Cappi, Carolina Carracedo, Angel Casas, Miguel Cavallini, Maria Cristina Ciullo, Valentina Cook, Edwin H. Crosby, Jesse Cullen, Bernadette A. De Schipper, Elles J. Delorme, Richard Djurovic, Srdjan Elias, Jason A. Estivill, Xavier Falkenstein, Martha J. Fundin, Bengt T. Garner, Lauryn Gironda, Christina Goes, Fernando S. Grados, Marco A. Grove, Jakob Guo, Wei Haavik, Jan Hagen, Kristen Harrington, Kelly Havdahl, Alexandra Höffler, Kira D. Hounie, Ana G. Hucks, Donald Hultman, Christina Janecka, Magdalena Jenike, Eric Karlsson, Elinor K. Kelley, Kara Klawohn, Julia Krasnow, Janice E. Krebs, Kristi Lange, Christoph Lanzagorta, Nuria Levey, Daniel Lindblad-Toh, Kerstin Macciardi, Fabio Maher, Brion Mathes, Brittany McArthur, Evonne McGregor, Nathaniel McLaughlin, Nicole C. Meier, Sandra Miguel, Euripedes C. Mulhern, Maureen Nestadt, Paul S. Nurmi, Erika L. O’Connell, Kevin S. Osiecki, Lisa Ousdal, Olga Therese Palviainen, Teemu Pedersen, Nancy L. Piras, Federica Potluri, Sriramya Rabionet, Raquel Ramirez, Alfredo Rauch, Scott Reichenberg, Abraham Riddle, Mark A. Ripke, Stephan Rosário, Maria C. Sampaio, Aline S. Schiele, Miriam A. Skogholt, Anne Heidi Sloofman, Laura G. Smit, Jan Artigas, María Soler Thomas, Laurent F. Tifft, Eric Vallada, Homero van Kirk, Nathanial Veenstra-VanderWeele, Jeremy Vulink, Nienke N. Walker, Christopher P. Wang, Ying Wendland, Jens R. Winsvold Department of Psychology Humboldt-Universität zu Berlin Berlin Germany Department of Psychiatric Phenomics and Genomics (IPPG) Ludwig-Maximilians University Munich Munich Germany Centre for Psychiatry Research Department of Clinical Neuroscience Karolinska Institutet and Stockholm Health Services Region Stockholm Stockholm Sweden Department of Biomedicine Aarhus University Aarhus Denmark University Hospital of Psychiatry and Psychotherapy University of Bern Bern Switzerland Department of Mental Health and Neuroscience Translational Neurogenomics QIMR Berghofer Medical Research Institute Brisbane QLD Australia Department of Population Health and Immunity Healthy Development and Ageing Walter and Eliza Hall Institute of Medical Research Melbourne VIC Australia Department of Psychiatry Human Genetics Yale University New Haven CT United States VA Connecticut Healthcare System West Haven CT United States Department of Center for Genomic Medicine Psychiatric and Neurodevelopmental Genetics Unit Massachusetts General Hospital Boston MA United States Stanley Center for Psychiatric Research Broad Institute of MIT and Harvard Cambridge MA United States Department of Genetics University of North Carolina at Chapel Hill Chapel Hill NC United States Department of Psychiatry Amsterdam University Medical Centers University of Amsterdam Amsterdam Netherlands Center for Research in Molecular Medicine and Chronic Diseases (CiMUS) Genomics and Bioinformatics University of Santiago de Compostela Santiago de Compostela Spain Grupo de Medicina Xenómica Genetics Instituto de Investigación Sanitaria de Santiago de Compostela (FIDIS) Santiago de Compostela Spain Vanderbilt Genetics Institute Vanderbilt University Nashville TN United States Department of Psychiatry and Behavioral Sciences SUNY Downstate Health Sciences University Brooklyn NY United States VA NY Harbor Healthcare System Brooklyn NY United States Social Genetic and Developmental Psychiatry Centre Insti

Obsessive–compulsive disorder (OCD) affects ~1% of children and adults and is partly caused by genetic factors. We conducted a genome-wide association study (GWAS) meta-analysis combining 53,660 OCD cases and 2,044,417 controls and identified 30 independent genome-wide significant loci. Gene-based approaches identified 249 potential effector genes for OCD, with 25 of these classified as the most likely causal candidates, including WDR6, DALRD3 and CTNND1 and multiple genes in the major histocompatibility complex (MHC) region. We estimated that ~11,500 genetic variants explained 90% of OCD genetic heritability. OCD genetic risk was associated with excitatory neurons in the hippocampus and the cortex, along with D₁ and D₂ type dopamine receptor-containing medium spiny neurons. OCD genetic risk was shared with 65 of 112 additional phenotypes, including all the psychiatric disorders we examined. In particular, OCD shared genetic risk with anxiety, depression, anorexia nervosa and Tourette syndrome and was negatively associated with inflammatory bowel diseases, educational attainment and body mass index. © The Author(s) 2025.

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Helicity-dependent terahertz emission spectroscopy of topological insulator Sb2Te3 thin films

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Physical Review B 2017年第19期96卷 195407-195407页

作者： Chien-Ming Tu Yi-Cheng Chen Ping Huang Pei-Yu Chuang Ming-Yu Lin Cheng-Maw Cheng Jiunn-Yuan Lin Jenh-Yih Juang Kaung-Hsiung Wu Jung-Chun A. Huang Way-Faung Pong Takayoshi Kobayashi Chih-Wei Luo Department of Electrophysics National Chiao Tung University Hsinchu Taiwan 300 Republic of China Department of Physics Tamkang University New Taipei City 25137 Taiwan Republic of China Department of Physics Lund University P.O. Box 118 SE-221 00 Lund Sweden Department of Physics and Advanced Optoelectonic Technology Center National Cheng Kung University Tainan 701 Taiwan Republic of China National Synchrotron Radiation Research Center Hsinchu Taiwan 300 Republic of China Institute of Physics National Chiao Tung University Hsinchu Taiwan 300 Republic of China Taiwan Consortium of Emergent Crystalline Materials Ministry of Science and Technology Taipei 10601 Taiwan Republic of China Advanced Ultrafast Laser Research Center and Department of Engineering Science The University of Electro-Communications Chofugaoka 1-5-1 Chofu Tokyo 182-8585 Japan Japan Science and Technology Agency Core Research for Evolutional Science and Technology (CREST) Japan Science and Technology Agency K's Gobancho 7 Gobancho Chiyoda-ku Tokyo 102-0076 Japan

We report on helicity-dependent terahertz emissions that originate from the helicity-dependent photocurrents in topological insulator Sb2Te3 thin films due to ultrafast optical excitation. The polarity of the emitted terahertz radiation is controlled by both the incident angle and the helicity of optical pulses. Using an unprecedented decomposition-recombination procedure in the time domain, the signals of the Dirac fermions are fully separated from bulk contributions. These results provide insights into the optical coupling of topological surface states and open up opportunities for applying helicity-dependent terahertz emission spectroscopy in spintronics.

关键词： Optical generation of spin carriers Spin-orbit coupling Surface states Topological materials Topological insulators Ultrafast pump-probe spectroscopy

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Formation of parallel aq/org two-phase flows in extended-nanochannel by partial hydrophobic modification 19

Formation of parallel aq/org two-phase flows in extended-nan...

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19th International Conference on Miniaturized Systems for Chemistry and Life sciences, MicroTAS 2015

作者： Kazoe, Y. Ugajin, T. Ohta, R. Mawatari, K. Kitamori, T. University of Tokyo Japan Japan Science and Technology Agency Core Research for Evolutional Science and Technology Japan

ISBN: (纸本)9780979806483

We report establishment of a nm-partial hydrophobic modification method by focused ion beam (FIB) and formation of parallel aqueous/organic two-phase flows in extended-nanochannels by control of liquid-liquid interface. Using the parallel two-phase flow, we demonstrated solvent extraction by integrating phase confluence, extraction and phase separation into 25 fL volume. Due to 103 times/sec interaction between solute molecules and surface by diffusion in 100 nm space, extraction of lipid into the organic phase was completed within 4.5 ms. The ultra high performance compared to microfluidics will greatly contribute to novel applications such as single cell analysis. © 15CBMS-0001.

关键词： Two phase flow

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