检索结果-内蒙古大学图书馆

Transcriptomic implications of low herbicide concentrations in hepatic cells: Insights into the individual and combined effects of 2,4-D, glyphosate, and AMPA

引用

Toxicology Letters 2025年 409卷 1-11页

作者： Valente, Leticia Cardoso Riechelmann-Casarin, Luana Otton, Rosemari Delella, Flávia Karina Barbisan, Luís Fernando Romualdo, Guilherme Ribeiro São Paulo State University (UNESP) Experimental Research Unit (UNIPEX) Botucatu Medical School Brazil Interdisciplinary Post-graduate Program in Health Sciences Cruzeiro do Sul University São Paulo Brazil São Paulo State University (UNESP) Department of Structural and Functional Biology Botucatu Biosciences Institute Brazil

Glyphosate and 2,4-D are among the most widely used herbicides globally, leading to environmental presence, food contamination, and human contact. Investigations based on current toxicological limits or populational-based herbicide exposures are warranted, and in vitro bioassays provide useful tools for toxicological screening. Thus, this study evaluated the transcriptomic implications of non-cytotoxic exposures to glyphosate, its metabolite aminomethylphosphonic acid (AMPA), or 2,4-D - or to their mixes - on hepatic cells. The half maximal effective concentration (IC50) of each herbicide was calculated (cell viability) in human hepatic C3A cells and 1000-fold lower concentrations were used for transcriptomic analysis (RNA-Seq) after 48 h exposure, resembling current toxicological limits and considering herbicide water levels (glyphosate: 0.95 µg/mL;AMPA: 3.7 µg/mL;2,4-D: 0.56 µg/mL). Glyphosate exposure enriched MAPK-related biological processes (upregulated TNF, FOS, IGF1, and PDGFB), and downregulated genes associated with lipid metabolism (CD36 and PPARA). Many AMPA exposure-related differentially expressed genes (DEGs, such as PFKFB3, HK2, and ALDOA) were associated with glucose metabolic pathways. Glyphosate and its metabolite yielded a common molecular signature, as illustrated by principal component analysis and the function of 212 shared DEGs. The exposure to 2,4-D was associated with the JNK cascade and the solute carrier family annotations. The herbicide mixtures had a discrete effect on enhancing the impact of individual herbicides, although important epithelial-mesenchymal transition genes were exclusively modified by the mixes (COL11A2, LOXL3, SNAI1). Altogether, our data reveals new perspectives on the short-term molecular effects of herbicide exposure in liver cells, emphasizing potential avenues for further exploration. © 2025 Elsevier B.V.

关键词： 2,4-dichlorophenoxyacetic acid Aminomethylphosphonic acid C3A cells Glyphosate Transcriptomic analysis

来源：评论

学校读者我要写书评

暂无评论

Comparison of the systemic action of electroacupuncture and laserpuncture in experimental injuries on the back skin of Wistar rats(Rattus novergicus)

引用

Journal of Traditional Chinese Medicine 2021年第5期41卷 779-788页

作者： Ivens Takechi Nakagawa Hamilton Ricardo Alonso Fabrício Campos Kuroda JoséRoberto Passarini Junior Juan Guzman Quispe Cabanillas Fernanda Aparecida Sampaio Mendonça Gláucia Maria Tech dos Santos Andrea Aparecida de Aro Edson Rosa Pimentel Maria Esméria Corezola do Amaral Marcelo Augusto Marretto Esquisatto Graduate Program in Biomedical Sciences University Center of Herminio Ometto Foundation-FHOAvenida Dr.Maximiliano Baruto500-Jardim UniversitarioArarasSao Paulo 13607-339Brazil Department of Structural and Functional Biology Institute of BiologyState University of Campinas-UNICAMPAvenida Bertrand Russels/n.Caixa Postal 6109CampinasSao Paulo 13083-865Brazil

OBJECTIVE:To evaluate the systemic action of electroacupuncture and laserpuncture in the repair of excisional cutaneous injuries on the back of adult female Wistar ***:Ninety animals were divided into three experimental groups:C-control;E-treated with electroacupuncture[acupoints:Feishu(BL13),Geshu(BL17),Zusanli(ST36)];L-treated with laserpuncture(same acupoints)and euthanized on the 7 th,14 th and 21 st days for tissue removal and preparation for histomorphometric and biochemical(dosages,zymography and Western blotting)***:The number of fibroblasts in the E and L groups presented higher values than the C on the14 th and 21 st *** number of granulocytes was lower than C on the 21 st day in groups E and *** total number of newly formed vessels increased on the 21 st day and was higher in both treatments when compared to *** birefringent collagen fibers detected on the 21 st day was lower in groups E and L than group *** glycosaminoglycans and hydroxyproline amount was similar among groups throughout the experimental *** active isoform of matrixmetallo proteinase(MMP)-2 and the latent isoform of MMP-9 did not show any differences among all groups and experimental *** amount of collagenⅠpresented higher values in the L group on the 14 th day and in the E and L groups on the 21 st *** typeⅢcollagen,groups E and L presented values lower than C in all experimental *** amount of transforming growth factor-β1 in the E and L groups showed higher amounts than C on the 7 th day and lower on the 14 th and 21 st *** vascular endothelial growth factor,E and L groups presented similar and lower values than C on the 7 th and 14 th days,respectively,and similar on the 21 st ***:The therapies evaluated in this article stimulated fibroblast proliferation,neoangiogenesis and altered the reorganization of collagen fibers in the repair of excisional injuries in female adult rats.

关键词： wound healing electroacupuncture inflammation morphological and morphometric findings

来源：评论

学校读者我要写书评

暂无评论

Methylmercury exposure during prenatal and postnatal neurodevelopment promotes oxidative stress associated with motor and cognitive damages in rats: an environmental-experimental toxicology study

引用

Toxicology Reports 2022年 9卷 563-574页

作者： Fagundes, Beatriz Helena Fernandes Nascimento, Priscila Cunha Aragão, Walessa Alana Bragança Chemelo, Victória Santos Bittencourt, Leonardo Oliveira Eiró-Quirino, Luciana Silva, Marcia Cristina Freitas Freire, Marco Aurelio M. Fernandes, Luanna Melo Pereira Maia, Cristiane do Socorro Ferraz Crespo-Lopez, Maria Elena Lima, Rafael Rodrigues Laboratory of Functional and Structural Biology Institute of Biological Sciences Federal University of Para PA Belém Brazil Graduate Program in Health and Society University of the State of Rio Grande do Norte RN Mossoró Brazil Laboratory Pharmacology of Inflammation and Behavior Institute of Health Sciences Federal University of Para PA Belém Brazil Laboratory of Molecular Pharmacology Institute of Biological Sciences Federal University of Para PA Belém Brazil

The environmental contamination by methylmercury (MeHg) is a major concern for public health. The effects of MeHg in the central nervous system (CNS) of adult animals have been extensively investigated;however, little is known about the effects of MeHg exposure during intrauterine and lactation periods on motor and cognitive functions of adolescent rats. Therefore, this study aimed to investigate the effect of MeHg exposure during intrauterine life and lactation on both motor and cognitive functions of offspring rats. Ten female Wistar rats were exposed to 40 μg/kg/day of MeHg through cookie treats from the first day of pregnancy until the last day of breastfeeding. Both motor and cognitive functions of offspring male rats were assessed by open field, rotarod, and step-down inhibitory avoidance tests. Forty-one days after birth, the hippocampus and cerebellum were collected to determine total Hg content, antioxidant capacity against peroxyl radicals (ACAP), reduced glutathione (GSH) levels, lipid peroxidation (LPO), and nitrite levels. MeHg exposure during CNS development increased Hg levels in both hippocampal and cerebellar parenchymas, triggered oxidative stress throughout ACAP and GSH decrease, increased LPO and nitrite levels. These alterations resulted in reduced spontaneous and stimulated locomotion and short- and long-term memory deficits. Therefore, damages triggered by MeHg exposure during intrauterine life and lactation had detrimental effects on oxidative biochemistry and motor and cognitive functions of offspring rats. © 2022 The Authors

关键词： Central nervous system Cerebellum Hippocampus Methylmercury Neurotoxicology

来源：评论

学校读者我要写书评

暂无评论

functional role of Annexin A1 and S100A11 in psoriasis pathogenesis: Insights from IMQ-induced models and keratinocyte modulation

引用

International Immunopharmacology 2025年 159卷 114915页

作者： Correia-Silva, Rebeca D. Corrêa, Mab P. Rodrigues-Silva, Nathália Santos, Diego D. Greco, Karin V. Gil, Cristiane D. Structural and Functional Biology Graduate Program Department of Morphology and Genetics Escola Paulista de Medicina Universidade Federal de São Paulo (UNIFESP) SP São Paulo Brazil Department of Pathology and Forensic Medicine Faculdade de Medicina de São José do Rio Preto (FAMERP) São José do Rio Preto SP Brazil Division of Surgery and Interventional Science University College London (UCL) London United Kingdom

Psoriasis (Pso) is a chronic inflammatory skin disease involving immune dysregulation and epidermal hyperplasia. Annexin A1 (ANXA1), a glucocorticoid-regulated protein, and S100A11 are implicated in keratinocyte function, but their roles in Pso remain unclear. This study translationally elucidates the functional role of ANXA1 and S100A11 in Pso by integrating analyses in human and murine skin biopsies and in vitro assays using human keratinocytes. The expression patterns of the ANXA1 and S100A11 in Pso were investigated using an imiquimod (IMQ)- induced murine model and human skin biopsies. Furthermore, the in vitro effect of the ANXA1-derived peptide Ac₂_–₂₆ (5, 25, and 50 ng/mL) on IL-17-stimulated human keratinocytes (HaCaT cell line) was evaluated. IMQ-treated mice exhibited hallmark psoriatic features, including epidermal thickening, mast cell degranulation, and elevated IL-23 levels. Immunohistochemical analyses revealed significant upregulation of ANXA1 and S100A11 in psoriatic skin, with coexpression observed in keratinocytes, particularly in superficial epidermal layers. Transcriptomic analysis corroborated these findings, showing elevated ANXA1 and S100A11 expression in psoriatic lesions compared to controls. In HaCaT cells, IL-17 stimulation reduced ANXA1 and S100A11 levels. Treatment with Ac₂_–₂₆ at the highest concentrations (25 and 50 ng/mL) significantly reduced viability/metabolism of IL-17-stimulated keratinocytes, while the lowest concentration of Ac₂_–₂₆ (5 ng/mL) effectively increased ROS production. In conclusion, ANXA1 and S100A11 are key players in Pso, with their expression and coexpression closely linked to disease pathogenesis. Further studies are warranted to explore the clinical implications of targeting the ANXA1/S100A11 axis in inflammatory skin diseases. © 2025

关键词： Annexin A1 IL-17 Imiquimod-induced model Keratinocyte S100A11 Skin

来源：评论

学校读者我要写书评

暂无评论

Advances in the crosstalk between maternal separation and voluntary ethanol consumption and effects on reproduction

引用

Life Sciences 2022年第PartB期311卷 000-000页

作者： Teixeira, Giovana Rampazzo Martins, Otávio Augusto Kremer, Rafael Veras, Allice Santos Cruz Pinheiro, Patricia Fernanda Felipe Mello-Junior, Wilson Martinez, Francisco Eduardo Department of Physical Education School of Technology and Sciences UNESP Campus of Presidente Prudente SP São Paulo Brazil Department of Cellular Biology Embryology Genetics - Federal University of Santa Catarina Santa Catarina Florianópolis Brazil Department of Medicine - Federal University of Fronteira Sul Rio Grande do Sul Passo Fundo Brazil Multicenter Graduate Program in Physiological Sciences SBFIS São Paulo State University (UNESP) Campus of Araçatuba SP São Paulo Brazil Department of Animal Production and Preventive Veterinary Medicine Faculty of Veterinary Medicine and Animal Science São Paulo State University (UNESP) - SP Botucatu Brazil Department of Structural and Functional Biology Institute of Biosciences São Paulo State University (UNESP) SP Botucatu Brazil

Maternal separation (SM) is an event caused by early stress and may be associated with behavioral changes and vulnerabilities, enhancing ethanol consumption in adulthood. The aim of the study was to evaluate whether MS potentiates the effects of ethanol ingestion on physiological hormone regulation and its interference in testicular and epididymal morphofunctional aspects in voluntary ethanol-consuming rats. Therefore, for the first time, we investigated the effect of maternal separation and ethanol consumption in adulthood and for this we used free choice ethanol-consuming strains. Responses of metabolic and hormonal parameters were also addressed, as well as their effects on reproductive function. In summary, MS promoted an increase in voluntary ethanol consumption in UChA and UChB animals. There was an influence of MS on the increase of circulating corticosterone and testosterone in UChB animals (high-ethanol-preferring 10 % v/v). MS performed in the hyporesponsive period to stress promoted an increase in glucose and circulating lipids, as well as a reduction in lactate dehydrogenase levels. Daily sperm production and transit time through the epididymis in UChB animals were increased by MS. Together, these findings show that MS potentiates the effects of ethanol ingestion and promotes an imbalance in plasma hormone concentrations, interfering with the reproductive functional imbalance of ethanol-consuming rats. © 2022 Elsevier Inc.

关键词： Alcohol-induced disorders Corticosterone Early-life stress Pituitary-adrenal system

来源：评论

学校读者我要写书评

暂无评论

Electromagnetic Stimulation Combined with Aloe vera Increases Collagen Reorganization in Burn Repair

引用

Journal of Pharmacy and Pharmacology 2018年第7期6卷 633-646页

作者： Sofia Poletti Leticia D. Lucke Fernanda O Bortolazzo Renata M. Acunha Marcos G. Mattos Femanda OG. Gaspi Thiago A. M. Andrade Maria Esmeria C. Amaral Andrea A .Aro Edson R Pimentel Marcelo AM. Esquisatto Fernanda A. S. Mendonga Glaucia M. T. Santos Graduate Program in Biomedical Sciences of the Herminio Ometto University Center UIVIARARAS Araras Sao Paulo13607-339 Brazil Department of Structural and Functional Biology Institute of Biology University of Campinas-UNICAMP Campinas Sao Paulo13083-970 Brazil

Objectives： Burns are shown as a clinical problem for their severity and multiple complications due to the time required to heal. Therapies that improve their healing are of great importance, especially for being minimally invasive, of low cost and best performance, all related to the speed and quality of healing. This study investigated the effects of the magnetic electro stimulator Haihuá CD9 isolated or in association with Aloe vera in rats skin burns. Methods： Experimental groups （n = 30/group） were：（C） Carbopol gel; （F） A. vera/Carbopol gel; （H） Haihuá＋Carbopol gel; （H＋F） Haihuá＋A. vera/Carbopol gel. Samples were collected on the 7th, 14th, and 21st experimental days for structural and morphometric analysis, hydroxyproline and glycosaminoglycans quantification, zymography for MMP-2 and MMP-9 and Western Blotting for TGF-β1, VEGF, Collagen I and III. Key findings： The expression of TGF-β1 in H＋F was increased on the 7th day and of MMP-9 on the 7th and 14th days. The expression of VEGF increased in the first experimental periods and decreased in the last for the treated groups. There was an increase in the fibroblasts and birefringent collagen fibers in groups treated with Haihuá isolated or in association with A. vera in all periods. The quantification of collagen I increased, while collagen III decreased in H＋F. The higher amount of GAGs and MMP-2 active isoform was detected in H and H＋F during all periods. Conclusions： Considering the results of the present study, electromagnetic stimulation in association with the A. vera extract promoted an increase in the number of fibroblasts, GAGs content, MMP-2 activity, the deposition and organization of collagen fibers, favoring the repair of injuries to second degree burns, and may also present therapeutic potential in this injury type.

关键词： Burns repair herbal medicine electromagnetic stimulation rats

来源：评论

学校读者我要写书评

暂无评论

Patterns of somatic structural variation in human cancer genomes (vol 578, pg 112, 2020)

引用

NATURE 2023年第7948期614卷 E38-E38页

作者： Li, Yilong Roberts, Nicola D. Wala, Jeremiah A. Shapira, Ofer Schumacher, Steven E. Kumar, Kiran Khurana, Ekta Waszak, Sebastian Korbel, Jan O. Haber, James E. Imielinski, Marcin Weischenfeldt, Joachim Beroukhim, Rameen Campbell, Peter J. Cancer Genome Project Wellcome Trust Sanger Institute Hinxton UK Totient Inc Cambridge MA USA Wellcome Sanger Institute Wellcome Genome Campus Hinxton UK Department of Haematology University of Cambridge Cambridge UK Cambridge University Hospitals NHS Foundation Trust Cambridge UK Korea Advanced Institute of Science and Technology Daejeon South Korea Department of Zoology Genetics and Physical Anthropology University of Santiago de Compostela Santiago de Compostela Spain Centre for Research in Molecular Medicine and Chronic Diseases (CIMUS) University of Santiago de Compostela Santiago de Compostela Spain The Biomedical Research Centre (CINBIO) University of Vigo Vigo Spain Centre for Research in Molecular Medicine and Chronic Diseases (CiMUS) Universidade de Santiago de Compostela Santiago de Compostela Spain Department of Zoology Genetics and Physical Anthropology (CiMUS) Universidade de Santiago de Compostela Santiago de Compostela Spain The Biomedical Research Centre (CINBIO) Universidade de Vigo Vigo Spain The Broad Institute of Harvard and MIT Cambridge MA USA Bioinformatics and Integrative Genomics Harvard University Cambridge MA USA Department of Cancer Biology Dana-Farber Cancer Institute Boston MA USA Broad Institute of MIT and Harvard Cambridge MA USA Department of Medical Oncology Dana-Farber Cancer Institute Boston MA USA Harvard Medical School Boston MA USA Massachusetts General Hospital Center for Cancer Research Charlestown MA USA Dana-Farber Cancer Institute Boston MA USA Department of Biostatistics and Computational Biology Dana-Farber Cancer Institute and Harvard Medical School Boston MA USA Weill Cornell Medical College New York NY USA Department of Physiology and Biophysics Weill Cornell Medicine New York NY USA Institute for Computational Biomedicine Weill Cornell Medicine New York NY USA Controlled Department and Institution New York NY USA Englander Institute for Precision Medicine Weill Cornell Medicine Ne

来源：评论

学校读者我要写书评

暂无评论

Retraction notice to "Toxicity of spike fragments SARS-CoV-2 S protein for zebrafish: A tool to study its hazardous for human health?" [Sci. Total Environ. 813 (2022) / 152345]

引用

The Science of the total environment 2024年 957卷 177653页

作者： Bianca H Ventura Fernandes Natália Martins Feitosa Ana Paula Barbosa Camila Gasque Bomfim Anali M B Garnique Ivana F Rosa Maira S Rodrigues Lucas B Doretto Daniel F Costa Bruno Camargo-Dos-Santos Gabrielli A Franco João Favero Neto Juliana Sartori Lunardi Marina Sanson Bellot Nina Pacheco Capelini Alves Camila C Costa Mayumi F Aracati Letícia F Rodrigues Rafaela Hemily Cirilo Raul Marcelino Colagrande Francisco I F Gomes Rafael T Nakajima Marco A A Belo Percília Cardoso Giaquinto Susana Luporini de Oliveira Silas Fernandes Eto Dayanne Carla Fernandes Wilson G Manrique Gabriel Conde Roberta R C Rosales Iris Todeschini Ilo Rivero Edgar Llontop Germán G Sgro Gabriel Umaji Oka Natalia Fernanda Bueno Fausto K Ferraris Mariana T Q de Magalhães Renata J Medeiros Juliana M Mendonça-Gomes Mara Souza Junqueira Kátia Conceição Leticia Gomes de Pontes Antonio Condino-Neto Andrea C Perez Leonardo J G Barcellos José Dias Correa Júnior Erick Gustavo Dorlass Niels O S Camara Edison Luiz Durigon Fernando Q Cunha Rafael H Nóbrega Glaucia M Machado-Santelli Chuck S Farah Flavio P Veras Jorge Galindo-Villegas Letícia V Costa-Lotufo Thiago M Cunha Roger Chammas Luciani R Carvalho Cristiane R Guzzo Guilherme Malafaia Ives Charlie-Silva Laboratório de Controle Genético e Sanitário Diretoria Técnica de Apoio ao Ensino e Pesquisa Faculdade de Medicina da Universidade de São Paulo Brazil. Laboratório Integrado de Biociências Translacionais (LIBT) Instituto de Biodiversidade e Sustentabilidade (NUPEM) Universidade Federal do Rio de Janeiro (UFRJ) Macaé RJ Brazil. Department of Microbiology Institute of Biomedical Sciences University of Sao Paulo Sao Paulo Brazil. Department of Cell Biology Institute of Biomedical Sciences University of São Paulo Brazil. Reproductive and Molecular Biology Group Department of Morphology Institute of Biosciences Sao Paulo State University Botucatu São Paulo Brazil. Department of Structural and Functional Biology Institute of Biosciences of Botucatu São Paulo State University SP Brazil. Department of Preventive Veterinary Medicine São Paulo State University (UNESP) Jaboticabal Brazil. Department of Pharmacology Center of Research in Inflammatory Diseases Ribeirao Preto Medical School University of São Paulo Brazil. Brasil University Descalvado São Paulo Brazil. Universidade Estadual Paulista Júlio de Mesquita Filho Instituto de Biociências - Departamento de Fisiologia São Paulo Brazil. Universidade Estadual Paulista Júlio de Mesquita Filho São Paulo Brazil. Postgraduate Program in Health Sciences PROCISA Federal University of Roraima Brazil. Immunochemistry Laboratory Butantan Institute São Paulo Brazil. Aquaculture Health Research and Extension Group GRUPESA Aquaculture Health Laboratory LABSA Department of Veterinary Medicine Federal University of Rondônia Rolim de Moura Campus Rondônia Brazil. Department of Preventive Veterinary Medicine São Paulo State University Jaboticabal Brazil. Department of Cell and Molecular Biology Ribeirao Preto Medical School University of São Paulo Ribeirão Preto Brazil. Departamento de Bioquímica Instituto de Química Universidade de São Paulo Brazil. Pontifícia Universidade Católica de Minas Gerais Brazil. Departamento de B

来源：评论

学校读者我要写书评

暂无评论

The repertoire of mutational signatures in human cancer (vol 578, pg 94, 2020)

引用

NATURE 2023年第7948期614卷 E41-E41页

作者： Alexandrov, Ludmil B. Kim, Jaegil Haradhvala, Nicholas J. Huang, Mi Ni Ng, Alvin Wei Tian Wu, Yang Boot, Arnoud Covington, Kyle R. Gordenin, Dmitry A. Bergstrom, Erik N. Islam, S. M. Ashiqul Lopez-Bigas, Nuria Klimczak, Leszek J. McPherson, John R. Morganella, Sandro Sabarinathan, Radhakrishnan Wheeler, David A. Mustonen, Ville Getz, Gad Rozen, Steven G. Stratton, Michael R. Department of Cellular and Molecular Medicine Department of Bioengineering Moores Cancer Center University of California San Diego CA USA Wellcome Sanger Institute Wellcome Genome Campus Hinxton UK Department of Cellular and Molecular Medicine and Department of Bioengineering University of California San Diego La Jolla CA USA UC San Diego Moores Cancer Center San Diego CA USA University of California San Diego San Diego CA USA Broad Institute of MIT and Harvard Cambridge MA USA Center for Cancer Research Massachusetts General Hospital Boston MA USA Massachusetts General Hospital Boston MA USA Department of Pathology Massachusetts General Hospital Boston MA USA Harvard Medical School Boston MA USA Laboratory for Medical Science Mathematics RIKEN Center for Integrative Medical Sciences Yokohama Japan Computational Biology Program Ontario Institute for Cancer Research Toronto ON Canada Programme in Cancer & Stem Cell Biology Duke-NUS Medical School Singapore Singapore Centre for Computational Biology Duke-NUS Medical School Singapore Singapore Programme in Cancer and Stem Cell Biology Duke-NUS Medical School Singapore Singapore Programme in Cancer and Stem Cell Biology Centre for Computational Biology Duke-NUS Medical School Singapore Singapore SingHealth Duke-NUS Institute of Precision Medicine National Heart Centre Singapore Singapore Singapore Cancer Science Institute of Singapore National University of Singapore Singapore Singapore Institute of Molecular and Cell Biology Singapore Singapore Laboratory of Cancer Epigenome Division of Medical Science National Cancer Centre Singapore Singapore Singapore Duke-NUS Medical School Singapore Singapore Human Genome Sequencing Center Baylor College of Medicine Houston TX USA Dan L. Duncan Cancer Center Baylor College of Medicine Houston TX USA Department of Molecular and Human Genetics Baylor College of Medicine Houston TX USA Genome Integrity and Structural Biology Laboratory National Ins

来源：评论

学校读者我要写书评

暂无评论

Effect of climate on traits of dominant and rare tree species in the world’s forests

引用

Nature Communications 2025年第1期16卷 1-15页

作者： Hordijk, Iris Poorter, Lourens Liang, Jingjing Reich, Peter B. de-Miguel, Sergio Nabuurs, Gert-Jan Gamarra, Javier G. P. Chen, Han Y. H. Zhou, Mo Wiser, Susan K. Pretzsch, Hans Paquette, Alain Picard, Nicolas Hérault, Bruno Bastin, Jean-Francois Alberti, Giorgio Abegg, Meinrad Adou Yao, Yves C. Almeyda Zambrano, Angelica M. Alvarado, Braulio V. Alvarez-Davila, Esteban Alvarez-Loayza, Patricia Alves, Luciana F. Amaral, Iêda Ammer, Christian Antón-Fernández, Clara Araujo-Murakami, Alejandro Arroyo, Luzmila Avitabile, Valerio Aymard C, Gerardo A. Baker, Timothy Banki, Olaf Barroso, Jorcely Bastian, Meredith L. Birigazzi, Luca Birnbaum, Philippe Bitariho, Robert Boeckx, Pascal Bongers, Frans Bouriaud, Olivier Brancalion, Pedro H. S. Brandl, Susanne Brearley, Francis Q. Brienen, Roel Broadbent, Eben N. Bruelheide, Helge Cazzolla Gatti, Roberto Cesar, Ricardo G. Cesljar, Goran Chazdon, Robin L. Chisholm, Chelsea Cienciala, Emil Clark, Connie J. Clark, David B. Colletta, Gabriel Coomes, David Cornejo Valverde, Fernando Corral-Rivas, Jose J. Crim, Philip Cumming, Jonathan Dayanandan, Selvadurai de Gasper, André L. Decuyper, Mathieu Derroire, Géraldine DeVries, Ben Djordjevic, Ilija Dourdain, Aurélie Dolezal, Jiri Engone Obiang, Nestor Laurier Enquist, Brian Eyre, Teresa Fandohan, Adandé Belarmain Fayle, Tom M. Ferreira, Leandro V. Feldpausch, Ted R. Finér, Leena Fischer, Markus Fletcher, Christine Frizzera, Lorenzo Gianelle, Damiano Glick, Henry B. Harris, David Hector, Andrew Hemp, Andreas Herbohn, John Hillers, Annika Honorio Coronado, Eurídice N. Hui, Cang Cho, Hyunkook Ibanez, Thomas Jung, Ilbin Imai, Nobuo Jagodzinski, Andrzej M. Jaroszewicz, Bogdan Johannsen, Vivian Joly, Carlos A. Jucker, Tommaso Karminov, Viktor Kartawinata, Kuswata Kearsley, Elizabeth Kenfack, David Kennard, Deborah Kepfer-Rojas, Sebastian Keppel, Gunnar Khan, Mohammed Latif Killeen, Timothy Kim, Hyun Seok Kitayama, Kanehiro Köhl, Michael Korjus, Henn Kraxner, Florian Laarmann, Diana Lang, Mait Lewis, Simon Lu, Huicui Lukina, Natalia Maitner, Br Institute of Integrative Biology ETH Zurich (Swiss Federal Institute of Technology) Zurich Switzerland Wageningen University and Research Wageningen Netherlands Department of Forestry and Natural Resources Purdue University West Lafayette IN United States Department of Forest Resources University of Minnesota St Paul MN United States Hawkesbury Institute for the Environment Western Sydney University Penrith NSW Australia Department of Agricultural and Forest Sciences and Engineering University of Lleida Lleida Spain Forest Science and Technology Centre of Catalonia (CTFC) Solsona Spain Forestry Division Food and Agriculture Organization of the United Nations Rome Italy Faculty of Natural Resources Management Lakehead University Thunder Bay ON Canada Manaaki Whenua–Landcare Research Lincoln New Zealand Chair for Forest Growth and Yield Science TUM School for Life Sciences Technical University of Munich Munich Germany Centre for Forest Research Université du Québec à Montréal Montréal QC Canada GIP ECOFOR Paris France CIRAD Forêts et Sociétés Montpellier France Forêts et Sociétés Univ Montpellier CIRAD Montpellier France Gembloux Agro Bio-Tech University of Liege Liege Belgium Faculty of Science and Technology Free University of Bolzano Bolzano Italy Department of Agricultural Food Environmental and Animal Sciences University of Udine Udine Italy Swiss Federal Institute for Forest Snow and Landscape Research WSL Birmensdorf Switzerland UFR Biosciences University Félix Houphouët-Boigny Abidjan Cote d'Ivoire Spatial Ecology and Conservation Laboratory Center for Latin American Studies University of Florida Gainesville FL United States Forestry School Tecnológico de Costa Rica TEC Cartago Costa Rica Fundacion ConVida Universidad Nacional Abierta y a Distancia UNAD Medellin Colombia Field Museum of Natural History Chicago IL United States Center for Tropical Research Institute of the Environment and Sustainability UCLA Los Angeles CA

Species’ traits and environmental conditions determine the abundance of tree species across the globe. The extent to which traits of dominant and rare tree species differ remains untested across a broad environmental range, limiting our understanding of how species traits and the environment shape forest functional composition. We use a global dataset of tree composition of >22,000 forest plots and 11 traits of 1663 tree species to ask how locally dominant and rare species differ in their trait values, and how these differences are driven by climatic gradients in temperature and water availability in forest biomes across the globe. We find three consistent trait differences between locally dominant and rare species across all biomes;dominant species are taller, have softer wood and higher loading on the multivariate stem strategy axis (related to narrow tracheids and thick bark). The difference between traits of dominant and rare species is more strongly driven by temperature compared to water availability, as temperature might affect a larger number of traits. Therefore, climate change driven global temperature rise may have a strong effect on trait differences between dominant and rare tree species and may lead to changes in species abundances and therefore strong community reassembly. © The Author(s) 2025.

关键词：

来源：评论

学校读者我要写书评

暂无评论

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

请选择保存的检索档案：

请选择收藏分类：

通借通还

建议与咨询 留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

时间限定

文献类型

馆藏选择

核心期刊

语言

文献类型

帮助

文字说明：

检索规则说明：

检索范例：

分类表

所选分类

限定检索结果

文献类型

馆藏范围

日期分布

学科分类号

主题

机构

作者

语言

请选择保存的检索档案： 新增检索档案 确定 取消

请选择收藏分类： 新增自定义分类 确定 取消

通借通还

建议与咨询留下您的常用邮箱和电话号码，以便我们向您反馈解决方案和替代方法

请选择保存的检索档案：

请选择收藏分类：